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2. The interplay between dietary fatty acids and gut microbiota influences host metabolism and hepatic steatosis

3. Dual regulation of TxNIP by ChREBP and FoxO1 in liver

5. Membrane estrogen receptor-α contributes to female protection against high-fat diet-induced metabolic disorders

7. Author Correction: Dimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptor

8. Dimorphic metabolic and endocrine disorders in mice lacking the constitutive androstane receptor

9. Ephrin-B1 regulates the adult diastolic function through a late postnatal maturation of cardiomyocyte surface crests

11. Membrane estrogen receptor-α contributes to female protection against high-fat diet-induced metabolic disorders.

12. ATGL-dependent white adipose tissue lipolysis controls hepatocyte PPARα activity

13. The hepatocyte insulin receptor is required to program the liver clock and rhythmic gene expression

14. Contributions of peroxisome proliferator-activated receptor β/δ to skin health and disease

16. Nuclear HMGB1 protects from nonalcoholic fatty liver disease through negative regulation of liver X receptor

17. Endoplasmic reticulum stress controls iron metabolism through TMPRSS6 repression and hepcidin mRNA stabilization by RNA-binding protein HuR

19. Src is activated by the nuclear receptor peroxisome proliferator-activated receptor β/δ in ultraviolet radiation-induced skin cancer

22. Hepatocyte-specific deletion of Pparα promotes NAFLD in the context of obesity

25. Selective Liver Estrogen Receptor Modulation Prevents Steatosis, Diabetes, and Obesity Through the Anorectic Growth Differentiation Factor 15 Hepatokine in Mice

26. GW501516-activated PPARβ/δ promotes liver fibrosis via p38-JNK MAPK-induced hepatic stellate cell proliferation

27. Selective Liver Estrogen Receptor α Modulation Prevents Steatosis, Diabetes, and Obesity Through the Anorectic Growth Differentiation Factor 15 Hepatokine in Mice

28. An essential role for the bmp-smad pathway in hepcidin induction during ER stress and NAFLD/NASH

29. Metabolic Effects of a Chronic Dietary Exposure to a Low-Dose Pesticide Cocktail in Mice: Sexual Dimorphism and Role of the Constitutive Androstane Receptor

31. Dual extraction of mRNA and lipids from a single biological sample

32. A Specific ChREBP and PPARα Cross-Talk Is Required for the Glucose-Mediated FGF21 Response

33. Dietary oleic acid regulates hepatic lipogenesis through a liver X receptor-dependent signaling

34. Selective Activation of Estrogen Receptor α Activation Function-1 Is Sufficient to Prevent Obesity, Steatosis, and Insulin Resistance in Mouse

35. Perilipin 5 fine-tunes lipid oxidation to metabolic demand and protects against lipotoxicity in skeletal muscle

38. Identification of a novel PPAR β/δ/miR‐21‐3p axis in UV ‐induced skin inflammation

39. Erratum: Hepatic circadian clock oscillators and nuclear receptors integrate microbiome-derived signals

40. Hepatic circadian clock oscillators and nuclear receptors integrate microbiome-derived signals

41. Noonan syndrome-causing SHP2 mutants inhibit insulin-like growth factor 1 release via growth hormone-induced ERK hyperactivation, which contributes to short stature

42. Combined QTL and Selective Sweep Mappings with Coding SNP Annotation andcis-eQTL Analysis RevealedPARK2andJAG2as New Candidate Genes for Adiposity Regulation

43. Src is activated by the nuclear receptor peroxisome proliferator‐activated receptor β/δ in ultraviolet radiation‐induced skin cancer

44. Identification of a novel PPARβ/δ/miR-21-3p axis in UV-induced skin inflammation.

45. Morbillivirus Glycoprotein Expression Induces ER Stress, Alters Ca2+ Homeostasis and Results in the Release of Vasostatin

46. Noonan syndrome-causing SHP2 mutants inhibit insulin-like growth factor 1 release via growth hormone-induced ERK hyperactivation, which contributes to short stature

47. Signal Strength Dictates Phosphoinositide 3-Kinase Contribution to Ras/Extracellular Signal-Regulated Kinase 1 and 2 Activation via Differential Gab1/Shp2 Recruitment: Consequences for Resistance to Epidermal Growth Factor Receptor Inhibition

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