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1. Mimicking hypomethylation of FUS requires liquid–liquid phase separation to induce synaptic dysfunctions

2. Spatially non-uniform condensates emerge from dynamically arrested phase separation

3. Biomolecular condensate phase diagrams with a combinatorial microdroplet platform

4. Reentrant liquid condensate phase of proteins is stabilized by hydrophobic and non-ionic interactions

5. Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodies

6. Does Soluble TREM2 Protect Against Alzheimer's Disease?

7. Tie-Line Analysis Reveals Interactions Driving Heteromolecular Condensate Formation

8. Heritability and genetic variance of dementia with Lewy bodies

9. C-terminal calcium binding of α-synuclein modulates synaptic vesicle interaction

10. Lipid Rafts Act as a Common Platform for Amyloid-β Oligomer-Induced Alzheimer’s Disease Pathology

11. Genetic variants in the SHISA6 gene are associated with delayed cognitive impairment in two family datasets

12. TET2 Regulates the Neuroinflammatory Response in Microglia

13. ANXA11 biomolecular condensates facilitate protein-lipid phase coupling on lysosomal membranes

14. <scp>MicroRNA</scp> ‐128 suppresses tau phosphorylation and reduces amyloid‐beta accumulation by inhibiting the expression of <scp>GSK3β</scp> , <scp>APPBP2</scp> , and <scp>mTOR</scp> in Alzheimer's disease

15. Tie-line analysis reveals interactions driving heteromolecular condensate formation

16. New insights into the genetic etiology of Alzheimer's disease and related dementias

17. Identification of a sex-specific genetic signature in dementia with Lewy bodies: a meta-analysis of genome-wide association studies

18. Amyloid-β toxicity modulates tau phosphorylation through the PAX6 signalling pathway

19. A multiplex platform to identify mechanisms and modulators of proteotoxicity in neurodegeneration

20. Solid/liquid coexistence during aging of FUS condensates

21. Reactive or transgenic increase in microglial TYROBP reveals a TREM2‐independent TYROBP–APOE link in wild‐type and Alzheimer's‐related mice

22. Z-α1-antitrypsin polymers impose molecular filtration in the endoplasmic reticulum after undergoing phase transition to a solid state

23. Z-α

24. Tie-lines reveal interactions driving heteromolecular condensate formation

25. Drug Repositioning for Alzheimer's Disease Based on Systematic 'omics' Data Mining.

26. Lipid rafts act as a common platform for Aß oligomer-induced Alzheimer’s pathology

27. SLITRK2, an X-linked modifier of the age at onset in C9orf72 frontotemporal lobar degeneration

28. Excess of nicastrin in brain results in heterozygosity having no effect on endogenous APP processing and amyloid peptide levels in vivo

29. Elevated plasma triglyceride levels precede amyloid deposition in Alzheimer’s disease mouse models with abundant Aβ in plasma

30. APOEε4, white matter hyperintensities, and cognition in Alzheimer and Lewy body dementia

31. Oral Immunization with Soybean Storage Protein Containing Amyloid-β 4–10 Prevents Spatial Learning Decline

32. Gene-wide analysis detects two new susceptibility genes for Alzheimer's disease.

33. Reentrant liquid condensate phase of proteins is stabilized by hydrophobic and non-ionic interactions

34. Differential interaction with TREM2 modulates microglial uptake of modified Aβ species

35. Gene Expression Imputation Across Multiple Tissue Types Provides Insight Into the Genetic Architecture of Frontotemporal Dementia and Its Clinical Subtypes

36. A Modest Increase in 11C-PK11195-Positron Emission Tomography TSPO Binding in Depression Is Not Associated With Serum C-Reactive Protein or Body Mass Index

37. Wild-type sTREM2 blocks Aβ aggregation and neurotoxicity, while the Alzheimer’s R47H mutant does the opposite

38. Additive Effects of PS1 and APP Mutations on Secretion of the 42-Residue Amyloid β-Protein

39. Presenilin Proteins Undergo Heterogeneous Endoproteolysis between Thr291and Ala299and Occur as Stable N- and C-Terminal Fragments in Normal and Alzheimer Brain Tissue

40. Correction: Is Genetically Associated with Late-Onset Alzheimer’s Disease in Japanese, Koreans and Caucasians.

41. SORL1 is genetically associated with late-onset Alzheimer's disease in Japanese, Koreans and Caucasians.

43. Mendelian randomization implies no direct causal association between leukocyte telomere length and amyotrophic lateral sclerosis

44. Genome sequencing analysis identifies new loci associated with Lewy body dementia and provides insights into the complex genetic architecture

45. High Resolution Biomolecular Condensate Phase Diagrams with a Combinatorial Microdroplet Platform

46. Reentrant liquid condensate phase of proteins is stabilized by hydrophobic and non-ionic interactions

47. Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodies

48. Biomolecular condensates undergo a generic shear-mediated liquid-to-solid transition

49. Beta amyloid aggregates induce sensitised TLR4 signalling causing long-term potentiation deficit and rat neuronal cell death

50. δ-Catenin is genetically and biologically associated with cortical cataract and future Alzheimer-related structural and functional brain changes.

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