23 results on '"Krebs, Luke T."'
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2. Notch signaling is essential for vascular morphogenesis in mice
3. Angiotensin IV AT4-receptor system in the rat kidney
4. Notch signal reception is required in vascular smooth muscle cells for ductus arteriosus closure
5. Notch3 is required for arterial identity and maturation of vascular smooth muscle cells
6. Notch signaling regulates left-right asymmetry determination by inducing Nodal expression
7. Notch signal reception is required in vascular smooth muscle cells for ductus arteriosus closure
8. The Snail Family Gene Snai3 Is Not Essential for Embryogenesis in Mice
9. Absence of a Major Role for the Snai1 and Snai3 Genes in Regulating Skeletal Muscle Regeneration in Mice
10. The Notch‐regulated ankyrin repeat protein is required for proper anterior–posterior somite patterning in mice
11. Patent ductus arteriosus in mice with smooth muscle-specific Jag1 deletion
12. Generation of mice with a conditional null allele of the Jagged2 gene
13. Notch1activation in mice causes arteriovenous malformations phenocopied by ephrinB2 and EphB4 mutants
14. Characterization of Notch3‐deficient mice: Normal embryonic development and absence of genetic interactions with a Notch1 mutation
15. The Nrarp gene encodes an ankyrin-repeat protein that is transcriptionally regulated by the notch signaling pathway
16. Characterization of the binding properties and physiological action of divalinal-angiotensin IV, a putative AT4 receptor antagonist
17. Autoradiographic identification of brain angiotensin IV binding sites and differential c-Fos expression following intracerebroventricular injection of angiotensin II and IV in rats
18. AT4 receptors: Specificity and distribution
19. The Snail Family Gene Snai3 Is Not Essential for Embryogenesis in Mice.
20. Patent ductus arteriosus in mice with smooth muscle-specific Jag1 deletion.
21. The NrarpGene Encodes an Ankyrin-Repeat Protein That Is Transcriptionally Regulated by the Notch Signaling Pathway
22. The Angiotensin IV System: Functional Implications
23. Notch1 activation in mice causes arteriovenous malformations phenocopied by ephrinB2 and EphB4 mutants.
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