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Background: Kampo medicine was introduced into the core curriculum model of medical education in Japan in 2007, and has been taught at all 80 medical universities across the country. However, as there are differences in terms of content and between universities, Kampo medicine education needs to be standardized. Therefore, this study aimed to examine the effects of combining multiple strategies, including lectures, problem‐based learning (PBL), and simulator training for the development of a standardized educational curriculum for Kampo medicine. Methods: We offered a lectures‐only class (Kampo strategy‐lectures [Ks‐L]), a class with lectures and decoction (Ks‐LD), a class with lectures, decoction, and PBL (Ks‐LDP), and a class with lectures, decoction, PBL, and simulator training (Ks‐LDPS). We analyzed the data from a 12‐item questionnaire conducted by the Faculty Development Committee of Kagoshima University Faculty of Medicine to evaluate the curriculum. Results: Ks‐LDPS was rated significantly higher than the other modules in active learning, and overall satisfaction. Conclusions: The results of this study suggest that combining multiple strategies rather than offering classes with lectures promoted a positive attitude to Kampo medicine and deep self‐learning among students, thereby improving the educational effects of Kampo medicine education. [ABSTRACT FROM AUTHOR]

The MPN driver mutation, JAK2 exon 12, is rare in MPN and has been exclusively found in polycythaemia vera (PV) (Scott [6]), with no reports of this mutation in MDS/MPN-RS-T to date. We would like to emphasize the importance of analysing JAK2 exon 12 mutation in patients with suspected MDS/MPN-RS-T, but who are negative for JAK2 V617F, CALR exon 9 and MPL W515 mutations. In summary, we identified a patient with MDS/MPN-RS-T harboring SF3B1 and JAK2 exon 12 mutations. Bone marrow morphologies of the patient harboring JAK2 exon 12 mutation, representing features associated with MDS/MPN-RS-T.Figure S3. [Extracted from the article]

The article offers information on the erythroid hypoplasia or aplasia which is a haematological condition observed in acquired pure red cell aplasia (PRCA) and aplastic anaemia. It discusses the pathogenic mechanisms of erythroid hypoplasia have not been fully elucidated, although T-lymphocyte-mediated inhibition of erythropoiesis which is considered as the most accurate mechanism of pathogenesis.

The article offers information about pure red cell aplasia (PRCA). It mentions that PRCA is represented by normochronic normocytic anemia, reticulocytopenia, and severe erythroid hypoplasia of the bone marrow along with clonal T-cell expansion in the bone marrow or peripheral blood. It further present case studies of several patients including some with PRCA and with thymoma, some with PRCA and myelodysplastic syndrome and some with PRCA and follicular lymphoma.

The article describes the case of a 74-year-old man diagnosed with degenerative hepatic hemangioma. Topics covered include how the disease mimics a typical biliary cystadenocarcinoma of the liver, the rarity of a hepatic cavernous hemangioma with cyst formation, and extended right hepatectomy that the patient underwent. Also mentioned are diagnostic imaging techniques such as contrast-enhanced computed tomography used to discover the tumor's features.