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2. Cleavage of Transaldolase by Granzyme B Causes the Loss of Enzymatic Activity with Retention of Antigenicity for Multiple Sclerosis Patients

3. Human Major Histocompatibility Complex (MHC) Class I Molecules with Disulfide Traps Secure Disease-related Antigenic Peptides and Exclude Competitor Peptides

4. Unraveling a Hotspot for TCR Recognition on HLA-A2: Evidence Against the Existence of Peptide-independent TCR Binding Determinants

5. Strategic Mutations in the Class I Major Histocompatibility Complex HLA-A2 Independently Affect Both Peptide Binding and T Cell Receptor Recognition

6. A Correlation between TCR Vα Docking on MHC and CD8 Dependence

7. Tax and M1 Peptide/HLA-A2-Specific Fabs and T Cell Receptors Recognize Nonidentical Structural Features on Peptide/HLA-A2 Complexes

8. TCR ligand discrimination is enforced by competing ERK positive and SHP-1 negative feedback pathways

9. Combinatorial Peptide Libraries and Biometric Score Matrices Permit the Quantitative Analysis of Specific and Degenerate Interactions Between Clonotypic TCR and MHC Peptide Ligands

10. Conversion of a T Cell Antagonist into an Agonist by Repairing a Defect in the TCR/Peptide/MHC Interface

11. Peptide Recognition by Two HLA-A2/Tax11–19-Specific T Cell Clones in Relationship to Their MHC/Peptide/TCR Crystal Structures

12. Shapes of MHC Restriction

13. Clustering of non-major histocompatibility complex susceptibility candidate loci in human autoimmune diseases

14. CD8+ Myelin Peptide-Specific T Cells Can Chemoattract CD4+ Myelin Peptide-Specific T Cells: Importance of IFN-Inducible Protein 10

15. Accumulation of human immunodeficiency virus-specific cytotoxic T lymphocytes away from the predominant site of virus replication during primary infection

16. Identification of an epitope derived from human proteolipid protein that can induce autoreactive CD8+ cytotoxic T lymphocytes restricted by HLA-A3: evidence for cross-reactivity with an environmental microorganism

17. Short Communication: C2H2-546: A zinc finger protein differentially expressed in HTLV-1 infected T cells

18. Structure of the complex between human T-cell receptor, viral peptide and HLA-A2

19. Interferon regulatory factor-2 physically interacts with NF-κB in vitro and inhibits NF-κB induction of major histocompatibility class I and β2-microglobulin gene expression in transfected human neuroblastoma cells

20. T-cell Receptor Use in Multiple Sclerosis

21. The HLA-B14 peptide binding site can accommodate peptides with different combinations of anchor residues

22. Autoreactive CD8+ T-cell responses to human myelin protein-derived peptides

23. T cell recognition of an HLA-A2-restricted epitope derived from a cleaved signal sequence

24. Heterogeneity of T-cell receptor alpha-chain complementarity-determining region 3 in myelin basic protein-specific T cells increases with severity of multiple sclerosis

25. CARE-LASS (calcein-release-assay), an improved fluorescence-based test system to measure cytotoxic T lymphocyte activity

26. Recognition by HLA-A2-restricted cytotoxic T lymphocytes of endogenously generated and exogenously provided synthetic peptide analogues of the influenza a virus matrix protein

27. Skewed T-cell receptor repertoire in genetically identical twins correlates with multiple sclerosis

28. Endogenous peptides bound to HLA-A3 possess a specific combination of anchor residues that permit identification of potential antigenic peptides

30. The peptide binding specificity of HLA class I molecules is largely allele-specific and non-overlapping

31. Peptide binding to HLA-A2 and HLA-B27 isolated from Escherichia coli. Reconstitution of HLA-A2 and HLA-B27 heavy chain/beta 2-microglobulin complexes requires specific peptides

32. Preparation and culture of human lymphocytes

33. HLA-B37 and HLA-A2.1 molecules bind largely nonoverlapping sets of peptides

34. The kinetics of peptide binding to HLA-A2 and the conformation of the peptide-A2 complex can be determined by amino acid side chains on the floor of the peptide binding groove

35. Molecular Cloning and Mapping of a Novel Human KRAB Domain-Containing C2H2-Type Zinc Finger to Chromosome 7q36.1

36. Molecular cloning and mapping of a novel developmentally regulated human C2H2-type zinc finger

37. Extensive T cell receptor cross-reactivity on structurally diverse haptenated peptides presented by HLA-A2

38. A correlation between TCR Valpha docking on MHC and CD8 dependence: implications for T cell selection

39. MHC recognition by hapten-specific HLA-A2-restricted CD8+ CTL

40. Thermodynamic and kinetic analysis of a peptide-class I MHC interaction highlights the noncovalent nature and conformational dynamics of the class I heterotrimer

41. Detection of virus-specific T cells and CD8+ T-cell epitopes by acquisition of peptide-HLA-GFP complexes: analysis of T-cell phenotype and function in chronic viral infections

42. MHC allele-specific molecular features determine peptide/HLA-A2 conformations that are recognized by HLA-A2-restricted T cell receptors

43. Microarray analysis of gene expression in multiple sclerosis and EAE identifies 5-lipoxygenase as a component of inflammatory lesions

44. Identification of a crucial energetic footprint on the alpha1 helix of human histocompatibility leukocyte antigen (HLA)-A2 that provides functional interactions for recognition by tax peptide/HLA-A2-specific T cell receptors

45. Structural, biochemical, and biophysical studies of HLA-A2/altered peptide ligands binding to viral-peptide-specific human T-cell receptors

46. Processing pathways for presentation of cytosolic antigen to MHC class II-restricted T cells

47. The structure and stability of an HLA-A*0201/octameric tax peptide complex with an empty conserved peptide-N-terminal binding site

48. Generation of Continuously Growing<scp>B</scp>Cell Lines by Epstein‐Barr Virus Transformation

49. C2H2-171 : A novel human cDNA representing a developmentally regulated poz domain/zinc finger protein preferentially expressed in brain

50. Evidence for rapid disappearance of initially expanded HIV-specific CD8+ T cell clones during primary HIV infection

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