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1. The Cross-Racial Mentoring Experiences of Faculty Members at Four-Year Public Universities: A Basic Qualitative Study

Author

Washington, Toya M. Burke

Abstract

This qualitative study examined the experiences of faculty members who have participated in cross-racial mentoring experiences at four-year public universities. This study explored faculty members' experiences and the impact of race on the relationship. Mentoring was an avenue that positively impacted the trajectory of the mentees' careers. This study explored the experiences of mentors and mentees in cross-racial mentoring relations and the effects of the relationship on the personal and professional lives of the participants. The study participants included eight full-time faculty members employed at four-year public universities in a cross-racial mentoring relationship. Data were recorded via Zoom audio-recorded interviews. The data analysis revealed six themes: mentor support, communication, availability, career advancement opportunities, organic relationships, and factors affecting the relationship. The study revealed two key findings: common interests, culture, and gender were more significant factors than race in the mentoring relationship, and mentoring positively affected the personal and professional lives of the participants. Further research is recommended to expand the participant criteria to include faculty of community colleges, private universities, and part-time or retired faculty members. Further research is also recommended to explore the relationship between cross-racial mentoring success and the participants' area of study and to explore the mentoring experiences of different races in cross-racial mentoring relationships. [The dissertation citations contained here are published with the permission of ProQuest LLC. Further reproduction is prohibited without permission. Copies of dissertations may be obtained by Telephone (800) 1-800-521-0600. Web page: http://www.proquest.com/en-US/products/dissertations/individuals.shtml.]

Published
2023

3. P-089 effect of testis tissue retrieval on “Micro Mapping Testicular Extraction” (MMTE): comparison with microscopic testicular sperm extraction (Micro TESE) in non-obstructive azoospermia (NOA)

Author

Tai, T, primary, Shibasaki, S, additional, Takahashi, M, additional, Okuyama, N, additional, Hattori, H, additional, Nagaura, S, additional, Yoshinaga, K, additional, Koizumi, M, additional, Toya, M, additional, Hashimoto, T, additional, Igarashi, H, additional, and Kyono, K, additional

Published
2023
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6. Constitutively Active Akt1 Cooperates with KRasG12D to Accelerate In Vivo Pancreatic Tumor Onset and Progression

Author

Toya M. Albury, Veethika Pandey, Sarah B. Gitto, Lisette Dominguez, Lina P. Spinel, Jacqueline Talarchek, Andres J. Klein-Szanto, Joseph R. Testa, and Deborah A. Altomare

Subjects
Neoplasms. Tumors. Oncology. Including cancer and carcinogens, RC254-282
Abstract

BACKGROUND AND AIMS: Pancreatic adenocarcinoma is a deadly disease characterized by metastatic progression and resistance to conventional therapeutics. Mutation of KRAS is the most frequent early event in pancreatic tumor progression. AKT isoforms are frequently activated in pancreatic cancer, and reports have implicated hyperactivation of AKT1, as well as AKT2, in pancreatic tumor formation. The objective here is to delineate the role of AKT in facilitating in vivo pancreatic tumor progression in the context of KRAS mutation and predisposition to pancreatic cancer. METHODS: Mice with Akt1 and KRas mutant alleles expressed using the pancreas Pdx promoter were mated to characterize the incidence and frequency of histologic and genetic alterations known to occur commonly in human pancreatic ductal adenocarcinoma. RESULTS: Active Akt1 (Akt1Myr, containing a myristoylation sequence) cooperated with active mutant KRasG12D to accelerate pancreatic carcinoma onset and progression and increase phosphorylation of downstream effectors in the Akt pathway. Mucin and smooth muscle actin expression was found in and around pancreatic intraepithelial neoplasms (PanINs), and accelerated time to metastasis was found in Akt1Myr/KRasG12D mice. CONCLUSIONS: In contrast to prior reports of pancreatic KRas mutant mice mated with mice deficient for various tumor suppressor genes, which resulted in aggressive disease within a few months of age, Akt1Myr/KRasG12D mice enabled the study of PanINs and spontaneous pancreatic transformation more characteristic of human pancreatic progression in elderly individuals. The Akt1Myr/KRasG12D model holds promise for delineating the tumor biology and biomarkers critical for understanding their cooperation in cancer oncogenesis and future targeting in therapeutic strategies.

Published
2015
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9. 058 A Rare Case of Spinal and Bulbar Muscular Atrophy (SMBA) Diagnosed by Hypertestosteronemia During Infertility Treatment

Author

Tai, T., primary, Igarashi, H., additional, Takesige, Y., additional, Nakamura, Y., additional, Hattori, H., additional, Nakajo, Y., additional, Aono, N., additional, Kasajima, M., additional, Yoshinaga, K., additional, Koizumi, M., additional, Hashimoto, T., additional, Toya, M., additional, Kumagai, J., additional, and Kyono, K., additional

Published
2022
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12. Energy release rates for an edge delamination of a laminated beam subjected to thermal gradient

Author

Toya, M., Oda, M., Kado, A., and Saitoh, T.

Subjects
Strains and stresses -- Research, Mechanics -- Research, Science and technology
Abstract

Energy release rates for an edge delamination of a laminated beam subjected to through-thickness temperature gradient are analyzed on the basis of the classical beam theory. The decomposition of the energy release rate into mode 1 and mode II components is made by combining the analyses of the energy release rates by Toya (1992) and the two-dimensional elasticity solutions for a split-beam element by Suo and Hutchinson (1990). The energy release rate is a quadratic function of the temperatures of the top and bottom surfaces of the beam. The transition of the type of crack growth between pure mode II and mixed mode type occurs at the temperature difference corresponding to the minimum energy release rate. Numerical analyses based on finite-element method are also carried out, which show that the theory agrees well with numerical results when temperature jump across the delaminated surfaces is relatively small as compared with the temperature difference between the top and bottom surfaces of the layered beam. [DOI: 10.1115/1.1978917]

Published
2005

13. Energy release rates for an interface crack embedded in a laminated beam subjected to three-point bending

Author

Toya, M., Aritomi, M., and Chosa, A.

Subjects
Finite element method -- Usage, Composite construction -- Fatigue, Science and technology
Abstract

A classical beam theory was used to analyze an asymmetric three-point bending of a simple layered beam with an internal interface crack. The delaminated beam was regarded as two lapped beams hinged at both ends to determine the axial forces. Numerical analyses based on the finite element method showed that delamination growth occurs in mixed mode subscribes to the fracturing process.

Published
1997

17. International Marketing Ethics: A Literature Review and Research Agenda

Author

La Toya M. Russell and Rajshekhar G. Javalgi

Subjects
Economics and Econometrics, business.industry, 05 social sciences, Public relations, General Business, Management and Accounting, Applied ethics, Marketing mix, Marketing science, Globalization, Internationalization, Arts and Humanities (miscellaneous), Multinational corporation, Information ethics, Political science, 0502 economics and business, 050211 marketing, Business and International Management, Business ethics, business, Law, 050203 business & management
Abstract

Globalization has changed the nature of business in the twenty-first century (Doh et al. in Bus Ethics Q 20(3):481–502, 2010). With the increased internationalization of multinational corporations, the need to address international marketing ethics arises (Carrigan et al. in Int Market Rev 22(5):481–493, 2005). Given the diversity of environments and cultures, ethical issues are numerous and complicated (Iyer in J Bus Ethics 31(1):3–24, 2001). The understanding of international marketing ethics is critical to academics as well as practitioners. This paper is a literature review (1960s through 2013) of the study of ethics in international marketing. In order to develop a comprehensive review of articles, we review topics such as ethical problems, moral philosophies, and culture and ethics. We also discuss the methodological procedures that are used in international marketing ethics. Researchers and practitioners alike can use this research on international marketing ethics to discuss the phenomena and subsequently lead to future work research.

Published
2015

19. A socio-marketing analysis of the concept of cute and its consumer culture implications

Author

Thomas Brashear Alejandro, Elad Granot, and La Toya M. Russell

Subjects
Marketing, Economics and Econometrics, Sociology and Political Science, Social Psychology, Advertising, Social marketing, Consumer Culture, Arts and Humanities (miscellaneous), Market analysis, Relevance (information retrieval), Sociology, Business and International Management, Consumer culture theory, Consumer behaviour
Abstract

Cute has become the favored language of (the predominantly female) popular consumer culture. This paper examines the roots of “cute” and its evolution with reference to its relevance to marketers. We follow the cultural appropriation of Japan’s “Kawaii” by the Western “cute”, and introduce a social, marketing-oriented description and analysis of the concept. We present its socio-cultural, experiential, symbolic, and ideological relevance to consumption in general, and to consumer culture theory in particular. This examination steers clear of scientific generalizations prevalent in consumer behavior research and aims, instead, to illuminate the cultural dimensions of the consumption cycle, and allow a better understanding of what it is like to form social attachment and loyalty in the context of cute consumption and consumer culture theory. Especially interesting questions arise regarding the trans-social acculturating elements of cute, as well as the apparent mainstreaming (cultural appropriation) of the cute Asian subculture into the dominant Western consumer and material cultures.

Published
2013

21. Effect of cancer treatment on female reproductive outcomes

Author

Sasaki, C., primary, Aono, N., additional, Nakajo, Y., additional, Hattori, H., additional, Tanaka, Y., additional, Inoue, H., additional, Koizumi, M., additional, Toya, M., additional, Hashimoto, T., additional, Igarashi, H., additional, and Kyono, K., additional

Published
2017
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22. Aripiprazole: A New Atypical Antipsychotic Drug

Author

Toya M. Bowles and Gary M Levin

Subjects
medicine.drug_class, Aripiprazole, Atypical antipsychotic, Quinolones, 030204 cardiovascular system & hematology, Pharmacology, Akathisia, 030226 pharmacology & pharmacy, Partial agonist, Piperazines, 03 medical and health sciences, 0302 clinical medicine, Extrapyramidal symptoms, medicine, Humans, Pharmacology (medical), Adverse effect, business.industry, Clinical trial, Pharmacodynamics, Schizophrenia, Controlled Clinical Trials as Topic, medicine.symptom, business, Antipsychotic Agents, medicine.drug
Abstract

OBJECTIVE:To review the pharmacology, pharmacokinetics, clinical efficacy, and safety profile of aripiprazole for the treatment of schizophrenia.DATA SOURCES:Information was selected from MEDLINE (1995–August 2002). Abstracts, scientific posters, and presentations were also used.STUDY SELECTION/DATA EXTRACTION:All published information regarding the pharmacokinetic, pharmacodynamic, and clinical characteristics of aripiprazole was considered. Studies providing a comprehensive description of aripiprazole were selected.DATA SYNTHESIS:Aripiprazole is a dopamine partial agonist and a serotonin-2A antagonist; it is dosed 10–30 mg/d, with no initial titration necessary. Short-term clinical trials demonstrated efficacy in acute exacerbations, and long-term studies showed that aripiprazole can maintain remission of schizophrenia. Most adverse events were mild. The incidence of extrapyramidal symptoms was low, with akathisia being the most common.CONCLUSIONS:Aripiprazole currently demonstrates comparable efficacy and safety for use in schizophrenia.

Published
2003

23. Constitutively active Akt1 cooperates with KRas(G12D) to accelerate in vivo pancreatic tumor onset and progression

Author

Veethika Pandey, Lisette Dominguez, Lina Spinel, Toya M. Albury, Jacqueline Talarchek, Sarah B. Gitto, Andres J. Klein-Szanto, Deborah A. Altomare, and Joseph R. Testa

Subjects
Male, Cancer Research, Pathology, medicine.medical_specialty, Genotyping Techniques, Blotting, Western, Gene Expression, AKT2, Mice, Transgenic, Biology, medicine.disease_cause, lcsh:RC254-282, Polymerase Chain Reaction, Polymorphism, Single Nucleotide, Article, Proto-Oncogene Proteins p21(ras), 03 medical and health sciences, Mice, 0302 clinical medicine, Pancreatic tumor, Pancreatic cancer, medicine, Animals, 030304 developmental biology, 0303 health sciences, Mucins, lcsh:Neoplasms. Tumors. Oncology. Including cancer and carcinogens, medicine.disease, Actins, 3. Good health, Mice, Inbred C57BL, Pancreatic Neoplasms, medicine.anatomical_structure, 030220 oncology & carcinogenesis, Mutation, Adenocarcinoma, CA19-9, Female, KRAS, Pancreas, Carcinogenesis, Proto-Oncogene Proteins c-akt, Carcinoma, Pancreatic Ductal
Abstract

BACKGROUND AND AIMS: Pancreatic adenocarcinoma is a deadly disease characterized by metastatic progression and resistance to conventional therapeutics. Mutation of KRAS is the most frequent early event in pancreatic tumor progression. AKT isoforms are frequently activated in pancreatic cancer, and reports have implicated hyperactivation of AKT1, as well as AKT2, in pancreatic tumor formation. The objective here is to delineate the role of AKT in facilitating in vivo pancreatic tumor progression in the context of KRAS mutation and predisposition to pancreatic cancer. METHODS: Mice with Akt1 and KRas mutant alleles expressed using the pancreas Pdx promoter were mated to characterize the incidence and frequency of histologic and genetic alterations known to occur commonly in human pancreatic ductal adenocarcinoma. RESULTS: Active Akt1 (Akt1Myr, containing a myristoylation sequence) cooperated with active mutant KRasG12D to accelerate pancreatic carcinoma onset and progression and increase phosphorylation of downstream effectors in the Akt pathway. Mucin and smooth muscle actin expression was found in and around pancreatic intraepithelial neoplasms (PanINs), and accelerated time to metastasis was found in Akt1Myr/KRasG12D mice. CONCLUSIONS: In contrast to prior reports of pancreatic KRas mutant mice mated with mice deficient for various tumor suppressor genes, which resulted in aggressive disease within a few months of age, Akt1Myr/KRasG12D mice enabled the study of PanINs and spontaneous pancreatic transformation more characteristic of human pancreatic progression in elderly individuals. The Akt1Myr/KRasG12D model holds promise for delineating the tumor biology and biomarkers critical for understanding their cooperation in cancer oncogenesis and future targeting in therapeutic strategies.

Published
2014

24. Successful Management of Prolonged γ-Hydroxybutyrate and Alcohol Withdrawal

Author

Roger W. Sommi, Toya M. Bowles, and Mohsen Amiri

Subjects
Adult, Male, Hallucinations, Dose, Alcohol abuse, Alcohol, Diaphoresis, Lorazepam, Irritability, Tachypnea, chemistry.chemical_compound, Humans, Medicine, Pharmacology (medical), Ethanol, business.industry, Central Nervous System Depressants, medicine.disease, Muscle Rigidity, Substance Withdrawal Syndrome, chemistry, Anesthesia, Concomitant, Fluid Therapy, Anticonvulsants, medicine.symptom, Sodium Oxybate, business, Anesthetics, Intravenous, medicine.drug
Abstract

A 27-year-old man was admitted with tremulousness, diaphoresis, tachypnea (28 breaths/min), full-body rigidity, irritability, paranoia, and auditory and visual hallucinations 2 days after stopping long-term gamma-hydroxybutyrate (GHB) and 8 hours after stopping alcohol intake. He received intravenous fluids and tapering dosages of lorazepam to control agitation and rigidity, and recovered with no significant sequelae after 8 days. Abrupt cessation of GHB after high-dosage abuse can precipitate a clinically significant withdrawal syndrome. Lorazepam should be considered for treatment of GHB withdrawal. Concomitant alcohol abuse may mask early GHB withdrawal symptoms and exacerbate withdrawal.

Published
2001

28. Ethical Issues of Small Business Owners: A Regional Perspective and a Conceptual Framework.

Author

Russell, La Toya M.

Subjects
SMALL business, ETHICS, EXECUTIVES, BUSINESSPEOPLE, GLOBALIZATION
Abstract

Previous research has shown that ethics is very important when conducting business domestically and internationally. There has been extensive ethics research done as it relates to managers in large firms and the ethical issues they encounter. What appears to be a limited area are the ethical issues encountered by entrepreneurs and small business owners. In this article, we will discuss some of the issues encountered by the entrepreneurs and small business owners. We will also discuss some of the ethical issues encountered by entrepreneurs and small business owners specifically in the Middle East and North Africa (MENA) region. [ABSTRACT FROM AUTHOR]

Published
2018
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31. Assessment of human serotonin 1A receptor polymorphisms and SSRI responsiveness

Author

Gary M. Levin, Toya M. Bowles, Megan J. Ehret, Taimour Y. Langaee, Julie A. Johnson, Jennifer Y. Tan, and William J. Millard

Subjects
Adult, medicine.medical_specialty, Population, Drug Resistance, Single-nucleotide polymorphism, Serotonergic, Polymorphism, Single Nucleotide, Internal medicine, Genetics, medicine, Escitalopram, Humans, education, 5-HT receptor, Aged, Pharmacology, Aged, 80 and over, education.field_of_study, business.industry, Depression, General Medicine, Middle Aged, Antidepressive Agents, Endocrinology, Receptor, Serotonin, 5-HT1A, 5-HT6 receptor, Molecular Medicine, Serotonin, business, Pharmacogenetics, Selective Serotonin Reuptake Inhibitors, medicine.drug
Abstract

Background: Depression is thought to involve, in part, dysregulation of serotonergic neurotransmission. In depressed individuals, the number of serotonin receptors, including the 5-hydroxytryptamine (serotonin)-1A (5-HT1A) autoreceptors, are increased. Clinical improvement with selective serotonin reuptake inhibitors (SSRIs) is not usually observed until several weeks after treatment initiation. This delay may be due to the time it takes for the autoreceptors to downregulate. Roughly one-third of patients with depression do not respond to an initial trial of antidepressant medication treatment, possibly as a result of structural variations in the 5-HT1A receptor. Aims: This study was designed to determine the allelic frequency of seven 5-HT1A receptor polymorphisms in a depressed versus a nondepressed population, and in SSRI responders versus nonresponders. All the polymorphisms studied are single nucleotide polymorphisms (SNPs) in the HTR1A gene, which encodes 5-HT1A. Seven prevalent SNPs were included in the analysis. Results: The study showed no relationship between any of the HTR1A polymorphisms and SSRI responders versus nonresponders. Conclusion: While the study has several limitations, the results are consistent with a growing body of literature that suggests that the pharmacogenetics of depression (an inherently complex disorder) may turn out to be multifactorial, and may include the HTR1A gene in concert with other serotonin-related genes.

Published
2007

32. Quality and safety of ART therapies

Author

Thornhill, A., primary, Dibouni, Z. A. -, additional, Shah, T., additional, Wheat, S., additional, Teitelbaum, M., additional, Walker, M., additional, Bissonette, F., additional, Yuzpe, A. L., additional, Leveille, M. C., additional, Gysler, M., additional, Johnson, M., additional, Sprague, A., additional, Villena, J. G., additional, Aparicio, J. L., additional, Gimenez, J., additional, Ten, J., additional, Perez, R. B., additional, Scholten, I., additional, Chambers, G. M., additional, van Loendersloot, L., additional, van der Veen, F., additional, Repping, S., additional, Gianotten, J., additional, Hompes, P. G. A., additional, Ledger, W., additional, Mol, B. W. J., additional, Dior, U. P., additional, Laufer, N., additional, Granovsky-Grisaru, S., additional, Yagel, S., additional, Yaffe, H., additional, Gielchinsky, Y., additional, Nelen, W. L. D. M., additional, Huppelschoten, A. G., additional, Verkerk, E. W., additional, Adang, E. M. M., additional, Kremer, J. A. M., additional, Davies, M., additional, Rumbold, A., additional, Marino, J., additional, Willson, K., additional, Moore, V., additional, Giles, L., additional, Shebl, O., additional, Ebner, T., additional, Tews, G., additional, Haas, D., additional, Oppelt, P., additional, Mayer, R. B., additional, Sanges, F., additional, Maggiulli, R., additional, Albricci, L., additional, Romano, S., additional, Scarica, C., additional, Schimberni, M., additional, Giallonardo, A., additional, Vettraino, G., additional, Ubaldi, F., additional, Rienzi, L., additional, Figueira, R. C. S., additional, Braga, D. P. A. F., additional, Setti, A. S., additional, Iaconelli, A., additional, Borges, E., additional, Obrado, E. C., additional, Barredo, D. R., additional, Navarro, L. L., additional, Rodriguez, A. V., additional, Rague, P. N. B., additional, Lletget, B. C., additional, Padro, R. T., additional, Oron, G., additional, Sokal-Arnon, T., additional, Zeadna, A., additional, Son, W. Y., additional, Holzer, H., additional, Tulandi, T., additional, Nakamura, Y., additional, Hattori, H., additional, Sato, Y., additional, Kuchiki, M., additional, Sakamoto, E., additional, Doshida, M., additional, Toya, M., additional, Kyono, K., additional, Nakajo, Y., additional, Hirata, K., additional, Xin, Z. M., additional, Zhu, H., additional, Sun, Y. P., additional, Jin, H. X., additional, Song, W. Y., additional, Rodriguez, A., additional, Poisot, F., additional, Rodriguez, F., additional, Coll, O., additional, Vassena, R., additional, Vernaeve, V., additional, Ye, Y., additional, Wang, L., additional, Wang, N., additional, Le, F., additional, Jin, F., additional, Zheng, Y., additional, Lou, Y., additional, Pan, P. P., additional, Wang, L. Y., additional, Hu, C. X., additional, Liu, S. Y., additional, Zheng, Y. M., additional, Li, L. J., additional, Liu, X. Z., additional, Xu, X. R., additional, Huang, H. F., additional, Lin, S. L., additional, Li, M., additional, Lian, Y., additional, Chen, L. X., additional, Liu, P., additional, Kawwass, J. F., additional, Crawford, S., additional, Kissin, D. M., additional, Session, D. R., additional, Boulet, S., additional, and Jamieson, D. J., additional

Published
2013
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34. Prediabetes linked to excess glucagon in transgenic mice with pancreatic active AKT1.

Author

Albury-Warren, Toya M., Pandey, Veethika, Spinel, Lina P., Masternak, Michal M., and Altomare, Deborah A.

Subjects
*PREDIABETIC state, *GLUCAGON, *TRANSGENIC mice, *PANCREATIC proteins, *PROTEIN kinase B, *ISLET cell tumor
Abstract

Protein kinase B/AKT has three isoforms (AKT1-3) and is renowned for its central role in the regulation of cell growth and proliferation, due to its constitutive activation in various cancers. AKT2, which is highly expressed in insulin-responsive tissues, has been identified as a primary regulator of glucose metabolism as Akt2 knockout mice (Akt2-/-) are glucoseintolerant and insulin-resistant. However, the role of AKT1 in glucose metabolism is not as clearly defined. We previously showed that mice with myristoylated Akt1 (AKT1Myr) expressed through a bicistronic Pdx1-TetA and TetO-MyrAkt1 system were susceptible to islet cell carcinomas, and in this study we characterized an early onset, prediabetic phenotype. Beginning at weaning (3 weeks of age), the glucose-intolerant AKT1Myr mice exhibited non-fasted hyperglycemia, which progressed to fasted hyperglycemia by 5 months of age. The glucose intolerance was attributed to a fasted hyperglucagonemia, and hepatic insulin resistance detectable by reduced phosphorylation of the insulin receptor following insulin injection into the inferior vena cava. In contrast, treatment with doxycycline diet to turn off the transgene caused attenuation of the non-fasted and fasted hyperglycemia, thus affirming AKT1 hyperactivation as the trigger. Collectively, this model highlights a novel glucagon-mediated mechanism by which AKT1 hyperactivation affects glucose homeostasis and provides an avenue to better delineate the molecular mechanisms responsible for diabetes mellitus and the potential association with pancreatic cancer. [ABSTRACT FROM AUTHOR]

Published
2016
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35. QUALITY AND SAFETY OF ART THERAPIES

Author

Caballero, P., primary, Alonso, J., additional, Cortes, S., additional, Caballero Campo, M., additional, Gago, M., additional, Nunez-Calonge, R., additional, Ricciarelli, E., additional, Gomez Palomares, J. L., additional, Bruna Catalan, I., additional, Hernandez, E. R., additional, Grzegorczyk-Martin, V., additional, Belaisch-Allart, J., additional, Mayenga, J. M., additional, Kulski, O., additional, Plachot, M., additional, Darby, H. C., additional, Florensa Bargallo, M., additional, Perals Vazquez, N., additional, Esbert Algam, M., additional, Belles Fernandez, M., additional, Ballesteros Boluda, A., additional, Calderon de Oya, G., additional, Alegre de Miquel, M., additional, Choudhary, M., additional, Ramineni, A., additional, Stewart, J., additional, Cabello, Y., additional, Fernandez-Shaw, S., additional, Mercader, A., additional, Herrer, R., additional, Arroyo, G., additional, Del Rio, F., additional, Carrera, M., additional, Fernandez Sanchez, M., additional, Sumimoto, T., additional, Kataoka, N., additional, Ogata, H., additional, Mizuta, S., additional, Tokura, Y., additional, Yamada, S., additional, Ogata, S., additional, Mizusawa, Y., additional, Matsumoto, Y., additional, Okamoto, E., additional, Kokeguchi, S., additional, Shiotani, M., additional, Nagai, Y., additional, Otsuki, J., additional, Maeda, K., additional, Momma, Y., additional, Takahashi, K., additional, Chuko, M., additional, Miwa, A., additional, Nagai, A., additional, Seggers, J., additional, Haadsma, M. L., additional, La Bastide-van Gemert, S., additional, Heineman, M. J., additional, Kok, J. H., additional, Middelburg, K. J., additional, Roseboom, T. J., additional, Schendelaar, P., additional, Van den Heuvel, E. R., additional, Hadders-Algra, M., additional, Jongbloed-Pereboom, M., additional, La Bastide-Van Gemert, S., additional, Heineman, K. R., additional, Bos, A. F., additional, Kondapalli, L. A., additional, Shaunik, A., additional, Molinaro, T. A., additional, Ratcliffe, S. J., additional, Barnhart, K. T., additional, Haadsma, M., additional, Keating, P., additional, Van Hoften, J. C., additional, Veenstra-Knol, H. E., additional, Cobben, J. M., additional, Pirkevi, C., additional, Atayurt, Z., additional, Yelke, H., additional, Kahraman, S., additional, Desmyttere, S., additional, Verpoest, W., additional, Haentjens, P., additional, Verheyen, G., additional, Liebaers, I., additional, Bonduelle, M., additional, Winter, C., additional, Van Acker, F., additional, De Schrijver, F., additional, Nekkebroeck, J., additional, Pariente-Khayat, A., additional, de Laubier, A., additional, Fehily, D., additional, Lemardeley, G., additional, Merlet, F., additional, Creusvaux, H., additional, Nakajo, Y., additional, Sakamoto, E., additional, Doshida, M., additional, Toya, M., additional, Nasu, I., additional, Kyono, K., additional, Schats, R., additional, Vergouw, C. G., additional, Kostelijk, E. H., additional, Doejaaren, E., additional, Hompes, P. G. A., additional, Lambalk, C. B., additional, Nakamura, Y., additional, Takisawa, T., additional, Shibuya, Y., additional, Sato, Y., additional, Sato, K., additional, Berard, A., additional, Chaabane, S., additional, Sheehy, O., additional, Blais, L., additional, Fraser, W., additional, Bissonnette, F., additional, Monnier, P., additional, Tan, S. L., additional, Trasler, J., additional, Subramaniam, A., additional, Chiappetta, R., additional, Mania, A., additional, Trew, G., additional, Lavery, S. A., additional, van den Akker, O., additional, Purewal, S., additional, Bunnell, C., additional, Lashen, H., additional, Terriou, P., additional, Giorgetti, C., additional, Porcu-Buisson, G., additional, Roger, V., additional, Chinchole, J. M., additional, Hamon, V., additional, Allemand-Sourieu, J., additional, Cravello, L., additional, Moreau, J., additional, Chabert-Orsini, V., additional, Belva, F., additional, Roelants, M., additional, De Schepper, J., additional, Devroey, P., additional, Painter, R. C., additional, Machin, L., additional, Fearon, K., additional, Morishima, K., additional, Fujimoto, A., additional, Oishi, H., additional, Hirata, T., additional, Harada, M., additional, Hasegawa, A., additional, Osuga, Y., additional, Yano, T., additional, Kozuma, S., additional, and Taketani, Y., additional

Published
2012
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36. POSTER VIEWING SESSION - EMBRYOLOGY

Author

Fourati Ben Mustapha, S., primary, Khrouf, M., additional, Kacem Ben Rejeb, K., additional, Elloumi Chaabene, H., additional, Merdassi, G., additional, Wahbi, D., additional, Ben Meftah, M., additional, Zhioua, F., additional, Zhioua, A., additional, Azzarello, A., additional, Host, T., additional, Mikkelsen, A. L., additional, Theofanakis, C. P., additional, Dinopoulou, V., additional, Mavrogianni, D., additional, Partsinevelos, G. A., additional, Drakakis, P., additional, Stefanidis, K., additional, Bletsa, A., additional, Loutradis, D., additional, Rienzi, L., additional, Cobo, A., additional, Paffoni, A., additional, Scarduelli, C., additional, Capalbo, A., additional, Garrido, N., additional, Remohi, J., additional, Ragni, G., additional, Ubaldi, F. M., additional, Herrer, R., additional, Quera, M., additional, GIL, E., additional, Serna, J., additional, Grondahl, M. L., additional, Bogstad, J., additional, Agerholm, I. E., additional, Lemmen, J. G., additional, Bentin-Ley, U., additional, Lundstrom, P., additional, Kesmodel, U. S., additional, Raaschou-Jensen, M., additional, Ladelund, S., additional, Guzman, L., additional, Ortega, C., additional, Albuz, F. K., additional, Gilchrist, R. B., additional, Devroey, P., additional, Smitz, J., additional, De Vos, M., additional, Bielanska, M., additional, Leveille, M. C., additional, Borghi, E., additional, Magli, M. C., additional, Figueroa, M. J., additional, Mascaretti, G., additional, Ferraretti, A. P., additional, Gianaroli, L., additional, Szlit, E., additional, Leocata Nieto, F., additional, Maggiotto, G., additional, Arenas, G., additional, Tarducci Bonfiglio, N., additional, Ahumada, A., additional, Asch, R., additional, Sciorio, R., additional, Dayoub, N., additional, Thong, J., additional, Pickering, S., additional, Ten, J., additional, Carracedo, M. 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N., additional, Kiesel, L., additional, Kliesch, S., additional, Azambuja, R., additional, Okada, L., additional, Lazzari, V., additional, Dorfman, L., additional, Michelon, J., additional, Badalotti, M., additional, Badalotti, F., additional, Petracco, A., additional, Schwarzer, C., additional, Versieren, K., additional, De Croo, I., additional, Lierman, S., additional, De Vos, W., additional, Van den Abbeel, E., additional, Gerris, J., additional, Milacic, I., additional, Borogovac, D., additional, Veljkovic, M., additional, Arsic, B., additional, Jovic Bojovic, D., additional, Lekic, D., additional, Pavlovic, D., additional, Garalejic, E., additional, Albertini, D. F., additional, De Ponti, E., additional, Sanges, F., additional, Talevi, R., additional, Papini, L., additional, Mollo, V., additional, Rienzi, L. F., additional, Gualtieri, R., additional, Orteg, C., additional, Choi, J., additional, Lee, H., additional, Ku, S., additional, Kim, S., additional, Choi, Y., additional, Kim, J., additional, Moon, S., additional, Demilly, E., additional, Assou, S., additional, Moussaddykine, S., additional, Dechaud, H., additional, Hamamah, S., additional, Takisawa, T., additional, Doshida, M., additional, Hattori, H., additional, Nakamura, Y., additional, Kyoya, T., additional, Shibuya, Y., additional, Nakajo, Y., additional, Tasaka, A., additional, Toya, M., additional, Kyono, K., additional, Novo, S., additional, Penon, O., additional, Gomez, R., additional, Barrios, L., additional, Duch, M., additional, Santalo, J., additional, Esteve, J., additional, Nogues, C., additional, Plaza, J. A., additional, Perez-Garcia, L., additional, Ibanez, E., additional, Chavez, S., additional, Loewke, K., additional, Behr, B., additional, Reijo Pera, R., additional, Huang, S., additional, Wang, H., additional, Soong, Y., additional, Chang, C., additional, Okimura, T., additional, Kuwayama, M., additional, Mori, C., additional, Morita, M., additional, Uchiyama, K., additional, Aono, F., additional, Kato, K., additional, Takehara, Y., additional, Kato, O., additional, Minasi, M., additional, Casciani, V., additional, Arizzi, L., additional, Mencacci, C., additional, Piscitelli, C., additional, Cucinelli, F., additional, Tocci, A., additional, Wydooghe, E., additional, Vandaele, L., additional, Dewulf, J., additional, Van Soom, A., additional, Moon, J. H., additional, Son, W. Y., additional, Mahfoudh, A., additional, Henderson, S., additional, Jin, S. G., additional, Shalom-Paz, E., additional, Dahan, M., additional, Holzer, H., additional, Mahmoud, K., additional, Triki-Hmam, C., additional, Terras, K., additional, Hfaiedh, T., additional, Ben Aribia, M. H., additional, Otsubo, H., additional, Egashira, A., additional, Tanaka, K., additional, Matsuguma, T., additional, Murakami, M., additional, Murakami, K., additional, Otsuka, M., additional, Yoshioka, N., additional, Araki, Y., additional, Kuramoto, T., additional, Smit, J. G., additional, Sterrenburg, M. D., additional, Eijkemans, M. J. C., additional, Al-Inany, H. G., additional, Youssef, M. A. F. M., additional, Broekmans, F. J. M., additional, Willoughby, K., additional, DiPaolo, L., additional, Deys, L., additional, Lagunov, A., additional, Amin, S., additional, Faghih, M., additional, Hughes, E., additional, Karnis, M., additional, Ashkar, F., additional, King, W. A., additional, Neal, M. 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P., additional, Kovacic, B., additional, Roglic, P., additional, Vlaisavljevic, V., additional, Sole, M., additional, Boada, M., additional, Coroleu, B., additional, Veiga, A., additional, Martiny, G., additional, Molinari, M., additional, Revelli, A., additional, Chimote, N. M., additional, Chimote, M., additional, Mehta, B., additional, Chimote, N. N., additional, Sheikh, N., additional, Nath, N., additional, Mukherjee, A., additional, Rakic, K., additional, Reljic, M., additional, Ingerslev, H. J., additional, Kirkegaard, K., additional, Hindkjaer, J., additional, Agerholm, I., additional, Kitasaka, H., additional, Fukunaga, N., additional, Nagai, R., additional, Yoshimura, T., additional, Tamura, F., additional, Kitamura, K., additional, Hasegawa, N., additional, Nakayama, K., additional, Katou, M., additional, Itoi, F., additional, Asano, E., additional, Deguchi, N., additional, Ooyama, K., additional, Hashiba, Y., additional, Asada, Y., additional, Michaeli, M., additional, Rotfarb, N., additional, Karchovsky, E., additional, Ruzov, O., additional, Atamny, R., additional, Slush, K., additional, Fainaru, O., additional, Ellenbogen, A., additional, Chekuri, S., additional, Chaisrisawatsuk, T., additional, Chen, P., additional, Pangestu, M., additional, Jansen, S., additional, Catt, S., additional, Molinari, E., additional, Racca, C., additional, Ryu, C., additional, Kang, S., additional, Lee, J., additional, Chung, D., additional, Roh, S., additional, Chi, H., additional, Yokota, Y., additional, Yokota, M., additional, Yokota, H., additional, Sato, S., additional, Nakagawa, M., additional, Komatsubara, M., additional, Makita, M., additional, Oyama, K., additional, Naruse, K., additional, Kilani, S., additional, Chapman, M. 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I., additional, Ricci, J., additional, Cavagna, M., additional, Pontes, A., additional, Vagnini, L. D., additional, Baruffi, R. L. R., additional, Franco Jr., J. G., additional, Felipe, V., additional, Vilela, M., additional, Tiveron, M., additional, Lombardi, C., additional, Viglierchio, M. I., additional, Marconi, G., additional, Rawe, V., additional, Wale, P. L., additional, Gardner, D. K., additional, Nakagawa, K., additional, Sugiyama, R., additional, Nishi, Y., additional, Kuribayashi, Y., additional, Jyuen, H., additional, Yamashiro, E., additional, Shirai, A., additional, Inoue, M., additional, Hovatta, O., additional, Tohonen, V., additional, Inzunza, J., additional, Parmegiani, L., additional, Cognigni, G. E., additional, Bernardi, S., additional, Ciampaglia, W., additional, Infante, F. E., additional, Tabarelli de Fatis, C., additional, Pocognoli, P., additional, Arnone, A., additional, Maccarini, A. M., additional, Troilo, E., additional, Filicori, M., additional, Radwan, P., additional, Polac, I., additional, Borowiecka, M., additional, Bijak, M., additional, and Radwan, M., additional

Published
2011
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40. Session 03: Novel Embryonic Markers

Author

Tejera, A., primary, Herrero, J., additional, Ramsing, N., additional, Pellicer, A., additional, Garrido, N., additional, Grau, N., additional, De los Santos, M. J., additional, Meseguer, M., additional, Sakkas, D., additional, Henman, M., additional, Harrison, P., additional, Botros, L., additional, Henson, M., additional, Judge, K., additional, Roos, P., additional, Guerin, J. F., additional, Guillemin, Y., additional, Cornut, A., additional, Aouacheria, A., additional, Cortezzi, S. S., additional, Figueira, R. C. S., additional, Eberlin, M. N., additional, Braga, D. P. A. F., additional, Ferreira, C. R., additional, Souza, G. H. M. F., additional, Garcia, J. S., additional, Iaconelli, A., additional, Borges, E., additional, Ishikawa, T., additional, Nakajo, Y., additional, Ota, M., additional, Doshida, M., additional, Toya, M., additional, Kyono, K., additional, Drakakis, P., additional, Vlismas, A., additional, Partsinevelos, G., additional, Desmarais, B., additional, Bletsa, R., additional, Kallianidis, K., additional, Loutradis, D., additional, Antsaklis, A., additional, and Kiessling, A., additional

Published
2010
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43. Constitutively Active Akt1 Cooperates with KRasG12D to Accelerate In Vivo Pancreatic Tumor Onset and Progression.

Author

Albury, Toya M., Pandey, Veethika, Gitto, Sarah B., Dominguez, Lisette, Spinel, Lina P., Talarchek, Jacqueline, Klein-Szanto, Andres J., Testa, Joseph R., and Altomare, Deborah A.

Subjects
*PANCREATIC cancer, *ADENOCARCINOMA, *TUMORS, *PHOSPHORYLATION, *CARCINOGENESIS
Abstract

BACKGROUND AND AIMS: Pancreatic adenocarcinoma is a deadly disease characterized by metastatic progression and resistance to conventional therapeutics. Mutation of KRAS is the most frequent early event in pancreatic tumor progression. AKT isoforms are frequently activated in pancreatic cancer, and reports have implicated hyperactivation of AKT1, as well as AKT2, in pancreatic tumor formation. The objective here is to delineate the role of AKT in facilitating in vivo pancreatic tumor progression in the context of KRAS mutation and predisposition to pancreatic cancer. METHODS: Mice with Akt1 and KRas mutant alleles expressed using the pancreas Pdx promoter were mated to characterize the incidence and frequency of histologic and genetic alterations known to occur commonly inhuman pancreatic ductal adenocarcinoma. RESULTS: Active Akt1 (Akt1Myr, containing a myristoylation sequence) cooperated with active mutant KRasG12D to accelerate pancreatic carcinoma onset and progression and increase phosphorylation of downstream effectors in the Akt pathway. Mucin and smooth muscle actin expression was found in and around pancreatic intraepithelial neoplasms (PanINs), and accelerated time to metastasis was found in Akt1Myr/KRasG12D mice. CONCLUSIONS: In contrast to prior reports of pancreatic KRas mutant mice mated with mice deficient for various tumor suppressor genes, which resulted in aggressive disease within a few months of age, Akt1Myr/KRasG12D mice enabled the study of PanINs and spontaneous pancreatic transformation more characteristic of human pancreatic progression in elderly individuals. The Akt1Myr/KRasG12D model holds promise for delineating the tumor biology and biomarkers critical for understanding their cooperation in cancer oncogenesis and future targeting in therapeutic strategies. [ABSTRACT FROM AUTHOR]

Published
2015
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47. SUBJECTIVE WELL-BEING AND ITS INFLUENCE ON CONSUMER PURCHASE BEHAVIOR OF LUXURY ITEMS DO MATERIALISM AND AFFECTIVE AUTONOMY HAVE AN IMPACT?

Author

Russell, La Toya M.

Subjects
WELL-being, CONSUMER behavior, LUXURY, MATERIALISM, LEISURE class, AUTONOMY (Psychology)
Abstract

In previous literature, researchers have studied subjective well-being; however, there appears to be limited research on the relationship between subjective well-being and consumer purchase behavior of luxury items. In this paper, the author explores the relationship between subjective well-being, materialism and affective autonomy and consumer purchase behavior of luxury items. [ABSTRACT FROM AUTHOR]

Published
2012

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