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1. [Chromosome aberrations in F1 (NZBxNZW) mice--an experimental model of systemic lupus erythematosus].

Author

Skazkina OIa, Lil'p IG, Pleskovskaia GN, Tugolukova SS, and Korogodina IuV

Subjects
Animals, Autoimmune Diseases genetics, Bone Marrow drug effects, Bone Marrow ultrastructure, Female, Male, Mice, Mitomycin, Mitomycins pharmacology, Chromosome Aberrations, Disease Models, Animal, Lupus Erythematosus, Systemic genetics, Mice, Inbred NZB genetics
Abstract

Spontaneous and mitomycin-C-induced chromosomal aberration level was studied in bone marrow cells of mice F1 (NZBXNZW), an experimental model of systemic lupus erythematosus and in C57BL/6J mice. The chromosome instability level is found to correspond to the degree of the autoimmune process: F1 (NZBXNZW) mice with the highest titer of autoantibodies to DNA has higher chromosomal mutability.

Published
1984

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