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Systemic hypertension is a cardiovascular disease increasingly present in geriatric cats. Its consequences on the target organs are also particularly on the eye,at the level of the retina. The afflicted individuals may present lesions of choroidopathy and hypertensive retinopathy. The objectives of the reported study were to evaluate the clinical and ophthalmological findings, the causes of systemic hypertension in these cats and to discuss the visual prognosis of the individuals. This retrospective study was carried out on a number of 14 patients. The selection criteria were represented by cats suffering from hypertensive retinopathy having a systolic blood pressure greater than 160 mmHg and having an ocular lesion: retinal vascular tortuosity, effusion, hemorrhage, retinal detachment or hyphema. Medical records were reviewed for individuals’ reports, the medical history, ophthalmic examination findings, and ocular ultrasound scans. Most cats were referred because they had decreased visual acuity (42.9%), a history of chronic kidney disease or even for hyphema, glaucoma and anisocoria. Auscultatory cardiac abnormalities were diagnosed in half of the individuals. Chronic kidney disease concomitant with systemic arterial hypertension was the most common (n=12), followed by hyperthyroidism (n=1). The four cats with blood pressure monitoring (n=4) after the administration of amlodipine showed a decrease in blood pressure. [ABSTRACT FROM AUTHOR]

Indifferently of animal species (including humans) in magnesium deficiencies usually we have one drop of this mineral from bone tissue. This phenomenon is more intense in young animals and is directly correlated with severity of magnesium deficits. The total number of clinical cases was 15 dogs, belong of German Sheppard, Dobermann, Great Dane, Siberian Husky breeds with clinical symptoms of rickets and with ages between 6 to 9 mounts. Group I has included patients with moderate signs of rickets and the group II has included patients with more severe form . Our biochemical analysis was; serum Calcium (SCa), serum Magnesium (SMg), erythrocyte Magnesium (EMg), an inorganic Phosphate (inorg.P), serum Creatinine (SCr) and Alkaline Phosphatase (AP); from urine we determine : urinary Magnesium (UMg), urinary Creatinine (UCr) and Magnesium excretion fraction (MgEF). Median values of seric and erythrocytes Magnesium in both groups were below reference values, but in group 2 were lower than in group 1. Dismagnesiemia is followed by one abrupt decreses in magnesiuria and reduction of excretion fraction of magnesium. This mean that global magnesium reserve is dropping and magnesium activity in cell is deficencity. Rickets osteodystrophia can be associated with slow hipomagnesium tetany. [ABSTRACT FROM AUTHOR]