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8. Changes in Renal Function One Year after Radical Nephrectomy for Cancer Compared to Kidney Donation

Author

Matheus We, Ferreira U, de Andrade Ct, de Lima Ml, and Mazzali M

Subjects
medicine.medical_specialty, urogenital system, business.industry, medicine.medical_treatment, Kidney donation, Urology, medicine, Renal function, Cancer, urologic and male genital diseases, business, medicine.disease, Nephrectomy
Abstract

Purpose: Adaptive changes to acute reduction in renal mass after radical nephrectomy can cause Chronic Kidney Disease (CKD) more frequently in patients with renal cancer compared to kidney donors. The aim of this study was to compare changes in Estimated Glomerular Filtration Rate (eGFR) 1-year after radical nephrectomy for cancer vs kidney donation and to identify risk factors for CKD.

Published
2020

9. The rise and fall of an extraordinary Ca-rich transient

Author

S. J. Prentice, K. Maguire, A. Flörs, S. Taubenberger, C. Inserra, C. Frohmaier, T. W. Chen, J. P. Anderson, C. Ashall, P. Clark, M. Fraser, L. Galbany, A. Gal-Yam, M. Gromadzki, C. P. Gutiérrez, P. A. James, P. G. Jonker, E. Kankare, G. Leloudas, M. R. Magee, P. A. Mazzali, M. Nicholl, M. Pursiainen, K. Skillen, S. J. Smartt, K. W. Smith, C. Vogl, D. R. Young

Published
2020
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20. Adeguatezza, dignità, negligenza: il problema della salute in carcere

Author

BUZZELLI, SILVIA, Scuto, S, Nobile de Santis, P, Giungi, A, Brusa, G, Manzelli, G, Mazzali, M, Brocchi, F, Limentani, C, Di Mauro, S, Crosti, B, Naldi, A, Moreno M, Spazzali, G., Buzzelli, S, Scuto, S, Nobile de Santis, P, Giungi, A, Brusa, G, Manzelli, G, Mazzali, M, Brocchi, F, Limentani, C, Di Mauro, S, Crosti, B, Naldi, A, Moreno, M, and Spazzali, G

Subjects
sistema penitenziario, salute, diritto alla, standard europei, IUS/16 - DIRITTO PROCESSUALE PENALE
Published
2014

24. Il carcere 'normale' e le sue alternative

Author

Buzzelli, S, Mazzali, M, Manzelli, G, Panarello, G, Di Rosa, G, Foschini, P, BUZZELLI, SILVIA, Foschini, P., Buzzelli, S, Mazzali, M, Manzelli, G, Panarello, G, Di Rosa, G, Foschini, P, BUZZELLI, SILVIA, and Foschini, P.

Published
2015

25. Anemia in Chronic Renal Disease: Evaluation of Inflammatory Activity on Erythropoiesis and Iron Metabolism in Patients not Submitted to Dialysis Treatment

Author

Mazzali M, Plotegher L, Grotto Hz, Gentil Af, and De Lima Ga

Subjects
medicine.medical_specialty, biology, business.industry, Anemia, medicine.medical_treatment, Neopterin, urologic and male genital diseases, medicine.disease, Gastroenterology, General Biochemistry, Genetics and Molecular Biology, chemistry.chemical_compound, chemistry, Iron-deficiency anemia, Erythropoietin, hemic and lymphatic diseases, Internal medicine, medicine, biology.protein, Hemoglobin, business, Intensive care medicine, Dialysis, Soluble transferrin receptor, medicine.drug, Kidney disease
Abstract

BACKGROUND Inflammatory activity is one of the factors involved in the physiopathology of anemia in patients with chronic kidney disease (CKD). The majority of studies on anemia, inflammation, and disturbances of iron metabolism have focused on patients in end-stage renal failure and dialysis therapy. However, anemia and inflammation are present in patients in previous stages of renal failure. The objective of this study was to evaluate the possible influence of inflammatory activity on erythropoiesis and iron metabolism in CKD patients without dialytic treatment. METHODS 114 CKD adult patients were studied. Patients with anemia (n = 72) were compared with those without anemia (n = 46). Anemic patients were classified as renal anemia (n = 46) or iron deficiency anemia (n = 26). In addition the total group was analyzed according to the degree of renal dysfunction. Iron status, erythropoiesis activity (soluble transferrin receptor and erythropoietin determinations), and inflammatory activity (C-reactive protein, interleukin-6, interleukin-lp, and neopterin determinations) were measured using commercial kits. Reticulocyte hemoglobin content (Ret-Y) was also determined. RESULTS Interleukin-6, interleukin-li, and neopterin concentrations were higher in the anemic group when compared with those without anemia and controls. There was no difference in C-reactive protein values between CKD with and without anemia, although both of them had showed elevated levels when compared with controls. Ret-Y values were lower in iron deficiency anemia when compared with renal anemia and controls. An inverse correlation between interleukin-6 and hemoglobin (r = -0.4287, p= 0.0002) was observed only in the renal anemia group. It was observed that anemia has a tendency to worsen as renal function deteriorates. Reticulocyte count was lower and neopterin concentrations were higher in more advanced renal failure stages. CONCLUSIONS Inflammatory factors contribute to anemia in renal patients in all stages of renal failure. High levels of neopterin in CKD patients suggest that neopterin contributes to impaired erythropoietin production and anemia in CKD patients.

Published
2013

26. Adeguatezza, dignità, negligenza: il problema della salute in carcere

Author

Buzzelli, S, Scuto, S, Nobile de Santis, P, Giungi, A, Brusa, G, Manzelli, G, Mazzali, M, Brocchi, F, Limentani, C, Di Mauro, S, Crosti, B, Naldi, A, Moreno, M, Spazzali, G, BUZZELLI, SILVIA, Moreno M, Spazzali, G., Buzzelli, S, Scuto, S, Nobile de Santis, P, Giungi, A, Brusa, G, Manzelli, G, Mazzali, M, Brocchi, F, Limentani, C, Di Mauro, S, Crosti, B, Naldi, A, Moreno, M, Spazzali, G, BUZZELLI, SILVIA, Moreno M, and Spazzali, G.

Published
2014

34. Could Uric acid have a Pathogenic Role in Chronic Allograft Dysfunction?

Author

Mazzali, M, Kazamel, R, Johnson, R, Mazzali, M, Kazamel, R, and Johnson, R

Abstract

Introduction: Chronic allograft dysfunction (CAD) is the primary cause of chronic graft failure after kidney transplantation. The pathogenesis of CAD involves both antigen-dependent and antigen-independent mechanisms. Serum uric acid could have a role in both mechanisms. Review: Hyperuricemia in subjects with renal transplantation is not usually viewed as clinically significant unless the subject develops gout. Drugs used in the treatment of hyperuricemia and gout are more likely to cause side effects in the kidney transplant reciepient. However, there are recent studies that raise the possibility that uric acid could have a role in CAD. Soluble uric acid has been shown to stimulate the proliferation of vascular smooth muscle, to inhibit endothelial cell proliferation and to reduce bioactive levels of endothelial NO. Studies in experimental models have found that hyperuricemia can cause hypertension associated with renal injury characterized by microvascular disease, tubulointerstitial disease, glomerular hypertrophy and glomerulosclerosis. Hyperuricemia was also found to worsen preexistent renal disease, and to be associated with the development of severe vascular lesions that are reminiscent of those observed in CAD. Furthermore, chronic cyclosporine nephropathy is very similar to what is observed in normal rats simply by raising uric acid levels. In addition, raising uric acid levels in rats receiving cyclosporine accelerates the nephropathy whereas lowering uric acid ameliorates the renal lesions. Conclusion: The pathogenic role of uric acid in CAD is still controversial. A controlled clinical trial would be the best approach to determine the effect of uric acid lowering treatment on the development of CAD and long term graft function. Keywords: chronic allograft dysfunction, cyclosporine, uric acid, hyperuricemia, vascular disease

Published
2010

42. Guidelines for Maintenance of Adult Patients With Brain Death and Potential for Multiple Organ Donations: The Task Force of the Brazilian Association of Intensive Medicine the Brazilian Association of Organs Transplantation, and the Transplantation Center of Santa Catarina

Author

Westphal, G.A., primary, Caldeira Filho, M., additional, Fiorelli, A., additional, Vieira, K.D., additional, Zaclikevis, V., additional, Bartz, M., additional, Wanzuita, R., additional, Teixeira, C., additional, Franke, C., additional, Machado, F.O., additional, Friedman, G., additional, Andrade, J., additional, Matos, J.D., additional, Lamgaro, D.M., additional, Silva, E., additional, Costa, G., additional, Coelho, M.E., additional, Oliveira, M.C., additional, Youssef, N.C.M., additional, Akamine, N., additional, Duarte, P., additional, Lisboa, R., additional, Mazzali, M., additional, and Ferraz Neto, B.H., additional

Published
2012
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49. OSTEOPONTIN IN NEPHROLITHIASIS: CELL CULTURE, ANIMAL, AND HUMAN STUDIES

Author

Wesson, J.A., additional, Kleinman, J.G., additional, Johnson, R.J., additional, Mazzali, M., additional, Beshensky, A.M., additional, Stietz, S., additional, Giachelli, C., additional, Liaw, L., additional, Alpers, C., additional, Couser, W.G., additional, Sorokina, E.A., additional, and Hughes, J., additional

Published
2002
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