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1. A randomized, double-blind, placebo-controlled trial of antidepressants in Parkinson disease

Author: I H, Richard, M P, McDermott, R, Kurlan, J M, Lyness, P G, Como, N, Pearson, S A, Factor, J, Juncos, C, Serrano Ramos, M, Brodsky, C, Manning, L, Marsh, L, Shulman, H H, Fernandez, K J, Black, M, Panisset, C W, Christine, W, Jiang, C, Singer, S, Horn, R, Pfeiffer, D, Rottenberg, J, Slevin, L, Elmer, D, Press, H C, Hyson, W, McDonald, and Katie, Kompoliti
Subjects: Adult, Male, medicine.drug_class, Serotonin reuptake inhibitor, Venlafaxine Hydrochloride, Venlafaxine, Placebo, Severity of Illness Index, Drug Administration Schedule, Double-Blind Method, Norepinephrine reuptake inhibitor, medicine, Humans, Serotonin–norepinephrine reuptake inhibitor, Psychiatric Status Rating Scales, Depressive Disorder, Adrenergic Uptake Inhibitors, Hamilton Rating Scale for Depression, Parkinson Disease, Cyclohexanols, Paroxetine, Antidepressive Agents, Delayed-Action Preparations, Anesthesia, Female, Neurology (clinical), Psychology, Selective Serotonin Reuptake Inhibitors, medicine.drug
Abstract: Objective: To evaluate the efficacy and safety of a selective serotonin reuptake inhibitor (SSRI) and a serotonin and norepinephrine reuptake inhibitor (SNRI) in the treatment of depression in Parkinson disease (PD). Methods: A total of 115 subjects with PD were enrolled at 20 sites. Subjects were randomized to receive an SSRI (paroxetine; n = 42), an SNRI (venlafaxine extended release [XR]; n = 34), or placebo (n = 39). Subjects met DSM-IV criteria for a depressive disorder, or operationally defined subsyndromal depression, and scored >12 on the first 17 items of the Hamilton Rating Scale for Depression (HAM-D). Subjects were followed for 12 weeks (6-week dosage adjustment, 6-week maintenance). Maximum daily dosages were 40 mg for paroxetine and 225 mg for venlafaxine XR. The primary outcome measure was change in the HAM-D score from baseline to week 12. Results: Treatment effects (relative to placebo), expressed as mean 12-week reductions in HAM-D score, were 6.2 points (97.5% confidence interval [CI] 2.2 to 10.3, p = 0.0007) in the paroxetine group and 4.2 points (97.5% CI 0.1 to 8.4, p = 0.02) in the venlafaxine XR group. No treatment effects were seen on motor function. Conclusions: Both paroxetine and venlafaxine XR significantly improved depression in subjects with PD. Both medications were generally safe and well tolerated and did not worsen motor function. Classification of Evidence: This study provides Class I evidence that paroxetine and venlafaxine XR are effective in treating depression in patients with PD.
Published: 2012
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2. Naturalistic evaluation of entacapone in patients with signs and symptoms of<scp>L</scp>-dopa wearing-off

Author: Robyn Schecter, Oksana Suchowersky, M. Panisset, Mandar Jog, and Bonita Rehel
Subjects: Male, Canada, medicine.medical_specialty, Levodopa, Activities of daily living, Parkinson's disease, Catechols, Drug Administration Schedule, Antiparkinson Agents, Quality of life, Rating scale, Surveys and Questionnaires, Internal medicine, Nitriles, medicine, Humans, Entacapone, Aged, Aged, 80 and over, Dose-Response Relationship, Drug, business.industry, Parkinson Disease, Drug Tolerance, General Medicine, Middle Aged, medicine.disease, nervous system diseases, Treatment Outcome, Clinical Global Impression, Physical therapy, Female, Observational study, business, Follow-Up Studies, medicine.drug
Abstract: To evaluate the effectiveness of entacapone in the management of levodopa wearing-off in Parkinson's disease (PD) in a naturalistic, real-life setting.This prospective, open-label, observational study included patients with idiopathic PD. Patients were eligible for inclusion if they had been taking 3-5 doses of levodopa per day for ≥2 months and had shown signs of levodopa wearing-off for ≥1 month. Subjects received entacapone (recommended dose: 1 × 200 mg tablet with each levodopa dose) for 28 days. Patients were asked to complete a wearing-off questionnaire and the eight-question Parkinson's Disease Questionnaire Quality of Life assessment (PDQ-8). Activities of daily living (both in the on and off states) were assessed using the Unified Parkinson's Disease Rating Scale (UPDRS) part II. Clinical Global Impression (CGI) of severity of PD-related symptoms was assessed using a modified CGI tool. Patient global assessment of severity of PD symptoms was also obtained.A total of 341 patients were enrolled by 68 physicians across Canada. At Day 28, 56.9% of the subjects indicated improvement compared to baseline on the modified CGI of change (CGI-C); 21.4% reported no change. Improvements were also observed on the UPDRS II and the PDQ-8. Benefit from entacapone appeared to be relatively uniform across subgroups (e.g., number of daily levodopa doses, use of other anti-PD medications).The results of this study may be biased due to factors inherent in open-label, community-based trials (e.g., compliance). This is, however, reflective of everyday clinical practice.In this naturalistic, real-life study, the addition of entacapone to levodopa therapy provided benefits in quality of life and activities of daily living for a substantial proportion of PD patients experiencing wearing-off.
Published: 2008
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3. Power calculations and placebo effect for future clinical trials in progressive supranuclear palsy

Author: Stamelou, M. Schöpe, J. Wagenpfeil, S. Del Ser, T. Bang, J. Lobach, I.Y. Luong, P. Respondek, G. Oertel, W.H. Boxer, A. Höglinger, G.U. Williams, D. Lafontaine, A.L. Marras, C. Jog, M. Panisset, M. Lang, A. Parker, L. Stewart, A.J. Corvol, J.-C. Azulay, J.-P. Couratier, P. Mollenhauer, B. Lorenzl, S. Ludolph, A. Benecke, R. Hoglinger, G. Lipp, A. Reichmann, H. Woitalla, D. Chan, D. Zermansky, A. Burn, D.J. Lees, A. Boxer, A. Miller, B.L. Lobach, I.V. Roberson, E. Honig, L. Zamrini, E. Pahwa, R. Bordelon, Y. Driver-Dunkley, E. Lessig, S. Lew, M. Womack, K. Boeve, B. Ferrara, J. Hillis, A. Kaufer, D. Kumar, R. Xie, T. Gunzler, S. Zesiewicz, T. Dayalu, P. Golbe, L. Grossman, M. Jankovic, J. McGinnis, S. Santiago, A. Tuite, P. Isaacson, S. Leegwater-Kim, J. Litvan, I. Grossman, M. Knopman, D.S. Schneider, L.S. Doody, R.S. Golbe, L. Koestler, M. Jack, C.R. Van Deerlin, V. Randolph, C. Gozes, I. Whitaker, S. Hirman, J. Gold, M. Morimoto, B.H. Gómez, J.C. Tijero, B. Berganzo, K. García de Yebenes, J. Lopez Sendón, J.L. Garcia, G. Tolosa, E. Buongiorno, M.T. Bargalló, N. Burguera, J.A. Martinez, I. Ruiz-Martínez, J. Narrativel, I. Vivancos, F. Ybot, I. Aguilar, M. Quilez, P. Boada, M. Lafuente, A. Hernandez, I. López-Lozano, J.J. Mata, M. Kupsch, A. Lipp, A. Ebersbach, G. Schmidt, T. Hahn, K. Hoglinger, G. Hollerhage, M. Reichmann, H. Wolz, M. Schneider, C. Klingelhofer, L. Berg, D. Maetzler, W. Srulijes, K.K. Ludolph, A. Kassubek, J. Steiger, M. Tyler, K. Morris, L. Lees, A. Ling, H. Hauser, R. McClain, T. Truong, D. Jenkins, S. Litvan, I. Houghton, D. Ferrara, J. Bordelon, Y. Gratiano, A. Golbe, L. Mark, M. Uitti, R. Ven Gerpen, J. Bhatia, K. Bordelon, Y.M. Colosimo, C. Dodel, R. Josephs, K.A. Morris, H. Mueller, U. Paviour, D. Schellenberg, G. Steele, J. van Swieten, J.C. Whitwell, J. Tau Restoration on PSP (TAUROS) Investigators The MDS-Endorsed PSP Study Group
Subjects: eye diseases
Abstract: Background: Two recent randomized, placebo-controlled trials of putative disease-modifying agents (davunetide, tideglusib) in progressive supranuclear palsy (PSP) failed to show efficacy, but generated data relevant for future trials. Methods: We provide sample size calculations based on data collected in 187 PSP patients assigned to placebo in these trials. A placebo effect was calculated. Results: The total PSP-Rating Scale required the least number of patients per group (N=51) to detect a 50% change in the 1-year progression and 39 when including patients with ≤ 5 years disease duration. The Schwab and England Activities of Daily Living required 70 patients per group and was highly correlated with the PSP-Rating Scale. A placebo effect was not detected in these scales. Conclusions: We propose the 1-year PSP-Rating Scale score change as the single primary readout in clinical neuroprotective or disease-modifying trials. The Schwab and England Activities of Daily Living could be used as a secondary outcome. © 2016 International Parkinson and Movement Disorder Society.
Published: 2016

4. Cerebrolysin in Alzheimer's disease: a randomized, double-blind, placebo-controlled trial with a neurotrophic agent

Author: Manfred Windisch, H. Moessler, M. Panisset, and Serge Gauthier
Subjects: Male, Heterozygote, medicine.medical_specialty, Placebo-controlled study, Placebo, Placebos, Central nervous system disease, Disability Evaluation, chemistry.chemical_compound, Apolipoproteins E, Cognition, Double-Blind Method, Gene Frequency, Alzheimer Disease, Internal medicine, medicine, Humans, Dementia, Amino Acids, Psychiatry, Adverse effect, Alleles, Biological Psychiatry, Aged, Aged, 80 and over, Psychiatric Status Rating Scales, Depression, Middle Aged, medicine.disease, Psychiatry and Mental health, Neuroprotective Agents, Phenotype, Treatment Outcome, Neurology, chemistry, Cerebrolysin, Female, Neurology (clinical), Age of onset, Alzheimer's disease, Psychology
Abstract: Cerebrolysin (Cere) is a compound with neurotrophic activity. It has been shown to be effective in the treatment of Alzheimer's disease (AD) in earlier trials. In this multicenter, randomized, double-blind, placebo-controlled, parallel-group study, patients were injected intravenously with placebo or 30 mL Cere five days per week for four weeks. Effects on cognition and global function were evaluated with the Alzheimer Disease Assessment Scale-Cognitive Subscale (ADAS-Cog) and the Clinicians Interview-based Impression of Change with Caregiver Input scale (CIBIC+) 4, 12, 24 weeks after the beginning of the injections. 192 patients were enrolled, 95 were randomized to placebo, and 97 to Cere. At baseline, there was a significant difference between groups for age, age of onset of dementia, and the number of patients with hallucinations. At week 12 there was a significant difference on the CIBIC+ (p = 0.033) in favor of Cere. The number of CIBIC+ responders (scoreor = 4), was significantly higher (p = 0.007), with 68 (76%) in the Cere group and 51 (57%) in the placebo group. Trends were noted in the Disability Assessment in Dementia scale and the Cornell Depression Scale. Adverse events were recorded in 73% of placebo and 64% of Cere patients. Most common adverse events were headaches, dizziness, weight loss and anxiety.Cere treatment was well tolerated and resulted in significant improvements in the global score two months after the end of active treatment.
Published: 2002
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5. LRRK2 Screening in a Canadian Parkinson's Disease Cohort

Author: Lemuel Racacho, Dennis E. Bulman, M. Panisset, Fabin Han, and David Grimes
Subjects: Gynecology, medicine.medical_specialty, Parkinson's disease, business.industry, Medical screening, General Medicine, medicine.disease, LRRK2, Surgery, Central nervous system disease, Neurology, Cohort, medicine, Neurology (clinical), business
Abstract: Background:Mutations in the leucine-rich repeat kinase 2 gene (LRRK2) have become the most common known cause for developing Parkinson's disease. The frequency of mutations described in the literature varies widely depending on the population studied with most reports focusing only on screening for the most common G2019S mutation in exon 41.Methods:In this study seven exons (19, 24, 25, 31, 35, 38, and 41) in LRRK2 where mutations have been reported were screened in 230 unselected Parkinson's disease patients using denaturing high-performance liquid chromatography.Results:The sequencing of samples with heteroduplex profiles revealed five novel and two known intronic sequence variants. In our cohort, we were unable to detect any of the known mutations in these exons or identify novel mutations within the LRRK2 gene.Conclusions:Therefore, despite the availability of diagnostic LRRK2 genetic testing it is unlikely to yield a positive result in this population.
Published: 2007
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6. R1514Q substitution in Lrrk2 is not a pathogenic Parkinson's disease mutation

Author: Karen Williams, Carolyn Peterson, S. Narayan, Margaret F. Turk, Julie H. Carter, C. Schell, Carlos Singer, Chad W. Christine, Paul J. Tuite, Robyn Schacherer, J. Whetteckey, S. Phipps, Diane K. Marek, William C. Nichols, John M. Bertoni, A. H. Rajput, Kenneth Marek, An Tran, P. Ryan, J. Hevezi, Joan Werner, Kelvin L. Chou, S. Chouinard, James Sutton, Margaret C. Lannon, T. Ajax, Joan Young, Deborah Judd, L. Zelaya, David Grimes, Magali Fernandez, Theresa A. Zesiewicz, Mark Stacy, Peggy Gray, Debra Berry, Michael J. Aminoff, C. Horn, C. Costan-Toth, J. Mannetter, Patricia Simpson, Susan Rolandelli, Tatiana Foroud, T. Tra, S. Wilson, Judith Dobson, Nestor Galvez-Jimenez, Donna Schwieterman, Shirley Uy, K. Price, J. Wojcieszek, Anette Nieves, Paul Atchison, Susan Bennett, L. Klassen, A. Podichetty, Vincent Calabrese, Becky Dunlop, D. Kamp, Holly Delgado, Sandra Roque, Maureen A. Leehey, Richard Camicioli, Julie So, Jayaraman Rao, Kelly E. Lyons, Kapil D. Sethi, A. Wang, Lynn Marlor, David Oakes, S. Culver, Juan Sanchez-Ramos, L. Woodward, J. Danielson, Jeannine Petit, Joann Belden, E. Licari, M. Meacham, Deborah Fontaine, Sharon Evans, C. Stone, S. Morehouse, Christopher F. O'Brien, G. Podskalny, J. Fraser, Anthony E. Lang, W.R. Wayne Martin, Carmen Serrano, H. Poiffaut, Stewart A. Factor, Joanne Wojcieszek, S. Belber, L. Davis, C. Allen, J. Hall, Judy Richman, Joseph Jankovic, Carson Reider, Stephen G. Reich, Stephanie Thomas, Kathy Davis, Richard B. Dewey, Karen Marder, T. Demarcaida, A. Kaczmarek, Lauren Seeberger, C. Halter, Mary Lou Klimek, Donald S. Higgins, Miodrag Velickovic, Joanna Hamann, Eric Siemers, E. Ohmann, C. Dingmann, Galit Kleiner-Fisman, Shari Niswonger, Theresa Derian, Maryan DeAngelis, Aileen Shinaman, Tilak Mendis, M. Rundle, Susan Mendick, L. Giffin, Karen Blindauer, Paul Gordon, Andrew Feigin, L. Shulman, Maureen Cook, Brian Wulbrecht, Rajesh Pahwa, T. Foroud, Un Jung Kang, Arthur Watts, Oksana Suchowersky, C. Joubert, J. Vo, Mandar Jog, M. Panisset, Roberta Winnick, Ronald F. Pfeiffer, Barbara Shannon, Jean P. Hubble, Clifford W. Shults, T. Gales, Tanya Simuni, M. Wolff, Hubert H. Fernandez, Pam Andrews, Karyn Boyar, Brad A. Racette, Vicki Hunt, Christine Hunter, Daniel D. Truong, L. Good, Robert L. Rodnitzky, P. Rodriguez, Sandra K. Kostyk, T. Shirley, Cheryl Halter, Peter A LeWitt, W. Weiner, Ryan J. Uitti, Lisa Scollins, Marc L. Gordon, J. Carpenter, Alice Rudolph, Lewis Sudarsky, Robert A. Hauser, Cliff Shults, Bala V. Manyam, Francis O. Walker, Juliette Harris, Marguerite Wieler, K. Dustin, Kelli Williamson, Brenda Pfeiffer, William C. Koller, Frederick J. Marshall, V. Hagen, A. Campbell, B. Hutchinson, L. Elmer, Anja Rudolph, K. Haas, Tori Ross, Rachel Saunders-Pullman, Nathan Pankratz, E. Aiken, Mariann DiMinno, Peggy Roberge, Arif Dalvi, B. Hayward, Mayank Pathak, David Simon, Michael W. Pauciulo, Holly A. Shill, M. Marotta-Kollarus, K. Ligon, Alok Sahay, Joseph H. Friedman, Neal Hermanowicz, E. Julian-Baros, Irenita Gardiner, N. Luong, Danna Jennings, R. Kurlan, and P. M. Conneally
Subjects: Adult, Male, Parkinson's disease, Adolescent, Genotype, Arginine, Guanine, Protein Serine-Threonine Kinases, Biology, Leucine-Rich Repeat Serine-Threonine Protein Kinase-2, medicine.disease_cause, Diagnosis, Differential, chemistry.chemical_compound, Degenerative disease, medicine, Humans, Point Mutation, Aged, Aged, 80 and over, Genetics, Mutation, Substitution (logic), Adenine Nucleotide Translocator 1, Genetic Variation, Parkinson Disease, Middle Aged, medicine.disease, LRRK2, nervous system diseases, Amino Acid Substitution, Neurology, chemistry, Female, Neurology (clinical), Carrier Proteins
Abstract: Mutations in LRRK2 were first reported as causing Parkinson's disease (PD) in late 2004. Since then, approximately a dozen LRRK2 substitutions have been identified that are believed to be pathogenic mutations. The substitution of adenine for guanine at nucleotide 4541 (4541G>A) in LRRK2 was recently reported. This substitution resulted in the replacement of an arginine at position 1514 with a glutamine (R1514Q). Although this substitution was not found in a large cohort of controls, its pathogenicity could not be verified. We have now genotyped the R1514Q substitution in a sample of 954 PD patients from 429 multiplex PD families. This substitution was identified in 1.8% of the PD patients; however, the majority of the PD sibships segregating this substitution were discordant for this putative mutation. In addition, the R1514Q substitution was detected in 1.4% of neurologically evaluated, control individuals. These data suggest that the R1514Q variant is not a pathogenic LRRK2 mutation. We believe it is imperative that the causative nature of any newly identified genetic variant be determined before it is included in any panel for diagnostic testing.
Published: 2007
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7. The relationship between extrapyramidal signs and cognitive performance in patients with Alzheimer's disease enrolled in the CERAD study

Author: Alan J. Lerner, M. Panisset, Christopher M. Clark, Douglas C. Ewbank, Gerda G. Fillenbaum, John C. Morris, Albert Heyman, Rachelle S. Doody, and Victor W. Henderson
Subjects: medicine.medical_specialty, Clinical Dementia Rating, Parkinsonism, Cognitive disorder, Neuropsychology, medicine.disease, medicine, Dementia, Verbal fluency test, Neurology (clinical), Effects of sleep deprivation on cognitive performance, Alzheimer's disease, Psychiatry, Psychology
Abstract: The objective of this study was to determine the relationship between the presence of extrapyramidal signs and the severity of cognitive and functional impairment in patients with Alzheimer's disease (AD). Eleven university medical centers in the United States and France participated in the Consortium to Establish a Registry for Alzheimer's Disease (CERAD) study of extrapyramidal signs in AD. Forty-seven patients with probable AD who had extrapyramidal signs were matched by sex, race, education, and age with 132 probable AD patients without extrapyramidal signs. The main outcome measures were the Clinical Dementia Rating, Blessed Dementia Scale, and the CERAD Neuropsychology Battery (verbal fluency, naming, Mini-Mental State Examination, word list learning, word list recall, savings ratio, word list recognition, and constructional praxis). AD patients with extrapyramidal signs performed more poorly than AD patients without parkinsonism on various neuropsychological tests, even after controlling for the Clinical Dementia Rating and reported duration of cognitive impairment. The severity of the extrapyramidal manifestations was related to the degree of cognitive and functional impairment. Muscular rigidity and bradykinesia were the most frequent extrapyramidal signs associated with AD. Patients with AD associated with extrapyramidal signs have greater cognitive and functional impairment than AD patients without clinical evidence of parkinsonism.
Published: 1997
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8. Visuo-spatial dysgnosia and Balint's syndrome as major symptoms of probable Alzheimer's disease

Author: F. Boiler, M. Agostini, M. Panisset, and P. Davous
Subjects: Pediatrics, medicine.medical_specialty, genetic structures, business.industry, Visual form, Posterior cortical atrophy, Disease, medicine.disease, Apraxia, Bálint's syndrome, Neurology, Severe dementia, medicine, Right posterior, Dementia, Neurology (clinical), business, Neuroscience
Abstract: We describe three patients, respectively 57-, 54- and 55-years-old at onset of the disease, who developed over many years a progressive cortical visual dysfunction without overt dementia. At onset, the visual symptoms were mainly a visuo-spatial dysgnosia, but they progressively worsened to a near complete Balint syndrome in the three cases. In all of them, memory and language were only mildly affected during the first years of evolution, but a constructive and/or dressing apraxia and an optic ataxia in the left visual hemifield suggested in two cases a predominant dysfunction in the right hemisphere. Imaging of the brain by CT scan and MRI showed no focal atrophy, but the metabolic SPECT was early abnormal in all three cases showing a right posterior metabolic defect in two cases and a bilateral posterior hypometabolism in one case. Follow-up of the patients showed that they all developed language and memory disturbances leading to severe dementia after 5–8 years of evolution. It is suggested that these patients are affected by a visual form of Alzheimer's disease which is probably the most frequent cause of progressive Balint-Holmes syndrome.
Published: 1996
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9. Fourth meeting of the European Neurological Society 25–29 June 1994 Barcelona, Spain

Author: H. Hattig, C. Delli Pizzi, M. C. Addonizio, Michelle Davis, A. R. Giovagnoli, L. Florensa, M. Roth, J. de Kruijk, Francisco Lacruz, Ph. Dewailly, A. Toygar, C. Avendano, P.P. De Deyn, J. F. Hurtevent, F. Lomeila, T. W. Wong, Gordon T. Plant, M. Bud, H. J. Willison, DH Miller, D. W. Langdon, R. Cioni, J. Servan, A. Kaygisiz, E. Racadot, D. B. Schens, E. Picciola, L. Falip, C. Bouchard, J. Jotova, A. Jorge-Santamaria, P. Misra, A. Dufour, C. P. Panagopoulos, A. Venneri, B. Sredni, B. Angelard, M. Janelidze, M. Carreno, J. Obenberger, J. Pouget, H. W. Moser, R. Kaufmann, J. A. Molina, D. Linden, A. Martin Urda, E. Uvestad, A. Krone, J. P. Cochin, J. Mallecourt, A. Cambon-Thomsen, K. Violleau, P. Osschmann, A. M. Durocher, E. Bussaglia, D. M. Danielle, H. Efendi, C. Van Broeckhoven, K. G. Jordan, W. Rautenberg, C. Iniguez, J. M. Delgado, Graham Watson, M. Lawden, Gareth J. Barker, K. Stiasny, James T. Becker, G. Campanella, E. Peghi, A. Poli, A. Haddad, T. Yamawaki, Giacomo P. Comi, S. Sotgiu, B. Ersmark, A. Pomes, M. Ziegler, P. Ferrante, P. Ruppi, H. KuÇukoglu, R. Bouton, U. K. Rinne, P. Vieregge, M. Dary, P. Giunti, Peter J. Goadsby, S. Jung, E. Secor, A. Steinberg, N. Vila, M. A. Hernandez, M. Cursi, A. Enqelhardt, A. Engelhardt, J. Veitch, F. Di Silverio, F. Arnaud, B. Neundörfer, R. Brucher, Dominique Caparros-Lefebvre, B. Meyer, Marianne Dieterich, M. H. Snidaro, R. Gomez, R. Cerbo, M. Ragno, J. M. Vance, S. Nemni, A. Caliskan, F. Barros, I. Velcheva, D. Ceballos-Baumann, V. Barak, A. Avila, N. Antonova, F. Resche, S. Pappata, L. Varela, S. R. Silveira Santos, A. Cammarota, L. Naccache, Y. Nara, E. Tournier-Lasserves, R. Mobner, T. Chase, A. Ensenyat, J. Ulrich, G. Giegerich, M. Rother, M. Revilla, N. Nitschke, K. Honczarenko, E. Basart Tarrats, J. Blin, B. Jacob, J. Santamaria, S. Knezevic, J. L. Castillo, M. Antem, J. Colomer, O. Busse, Didier Hannequin, S. Carrier, J. B. Ruidavets, C. Rozman, J. Bogoussslavsky, J. Pascual Calvet, E. Monros, J. M. Polo, M. Zucconl, Javier Muruzabal, R. R. Allen, R. Rivolta, K. Haugaard, A. Nespolo, K. Hoang-Xuang, G. Bussone, T. Avramidis, E. Corsini, Christiana Franke, T. Vinogradova, H. Boot, K. Vestergaard, G. H. Jansen, N. Argentino, M. Raltzig, W. Linssen, Mark B. Pepys, P. Roblot, L. Lauritzen, E. Fainardi, D. Morin, T. X. Arbizu Urdiain, J. Wollenhaupt, S. Bostantjopoulou, G. Pavesi, A. D. Forman, Giovanni Fabbrini, D. Jean, J. J. Archelos, M. I. Blanchs, M. Del Gobbo, Anna Carla Turconi, Ch. Derouesné, Elio Scarpini, A. Visbeck, P. Castejon, J. P. Renou, F. Mounier-Vehier, G. Potagas, Ch. Duyckaerts, A. Filla, R. Schneider, G. Ronen, K. Nagata, J. P. Vedel, A. Henneberg, G. van Melle, C. Baratti, H. Knott, M. C. Prevett, A. Bes, B. Metin, Jos V. Reempts, L. Martorell, Mefkure Eraksoy, H. O. Handwerker, D. S. Younger, O. Oktem, D. Frongillo, C. Soriano-Soriano, L. Niehaus, F. Zipp, A. Tartaro, S Newman, R. H. Browne, P. Davous, R. Sanchez, M. Muros, M. E. Kornhuber, A. Lavarone, M. Mohr, M. R. Garcia, S. Russell, H. Kellar-Wood, M. R. Tola, B. Ostermeyer, Ch. Tzekov, K. Sartor, E. B. Ringelstein, P. P. Gazzaniga, Paul Krack, H. Fidaner, H. Rico, T. Dbaiss, F. Alameda, E. Torchiana, L. Rumbach, I. Charques, J. M. Bogaard, C. D. Frith, L. J. Rappelle, R. Brenner, A. Joutel, K. Fuxe, G. HÄcker, M. J. Blaser, J. Valls-SolÇ, G. Ulm, M. Alberdi, A. Bock, F. W. Bertelsmann, U. Wieshmann, J. Visa, J. R. Lupski, D. D'Amico, L. M. P. Ramos, A. A. Vanderbark, R. Horn, M. Warmuth, Dietmar Kühne, Mark S. Palmer, C. Ehrenheim, E. Canga, S. Viola, O. Scarpino, P. Naldi, R. Almeida, A. A. Raymond, J. Gamez, Stephan Arnold, A. DiGiovanni, J. Dalmau, C. C. Chari, H. F. Beer, J. C. Koetsier, J. Iriarte, E. Yunis, J. Casadevall, E. Le Guern, E. Stenager, S. R. Benbadis, J. M. Warter, F. Burklin, I. Theodorou, L. Johannesen, G. A. Graveland, X. Leclerc, I. Vecchio, L. Ozelius, G. Nicoletti, R. K. Gherardi, E. Esperet, M. L. Delodovici, F. Cattin, F. Paiau, Giorgio Sacilotto, C. A. J. Broere, D. Chavdarov, J. P. Willmer, C. H. Hawkes, Th. Naegele, E. Ellie, E. Dartigues, M. J. Guardiola, S. Hesse, Z. Levic, Marco Rovaris, P. Saugeir-Veber, B. A. Yaqub, H. F. Durwen, R. Larumbe, J. Ballabrina, M. Sendtner, J. Röther, M. Horstink, C. Kluglein, M.P. Montesi, H. Apaydin, J. Montoya, E. Waubant, Ch. Verellen-Dunoulin, A. Nicolai, J. Lopez-Delval, R. Lemon, G. Cantinho, E. Granieri, A. Zeviani, Wolfgang H. Oertel, U. Ficola, V. Di Piero, V. Fragola, K. Sabev, M. V. Guitera, I. Turki, F. Bolgert, P. Ingrand, J. M. Gobernado, L. M. E. Grimaldi, S. Baybas, B. Eymard, Y. Rolland, Y. Robitaille, Ta. Pampols, P. J. Koehler, A. Carroacedo, J. Vilchez, S. Di Vittorio, I. R. Rise, T. Nagy, M. Kuffner, E. Palazzini, A. Ott, J. Pruim, T. X. Arbizu, E. Manetti, C. Cervera, S. Felber, G. Gursoy, J. Scholz, G. A. Buscaino, M. S. Chen, A. Pascual, J. Hazan, J. U. Gajda, J. G. Cea, G. Bottini, G. Damalik, F. Le Doze, G. Bonaldi, J. M. Hew, C. Messina, A. M. Kennedy, J. M. Carney, N. 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Brasic, W. Heide, I. Santilli, W. M. Korn, D. Selcuki, M. J. Barrett, D. Krieger, T. Leon, T. Houallah, M. Tournilhac, C. Nos, D. Chavot, F. Barbieri, F. J. Jimenez-Jimenez, J. Muruzabal, K. Poeck, A. Sennlaub, L. M. Iriarte, L. G. Lazzarino, C. Sanz, P. A. Fischer, S. D. Shorvon, R. Hoermann, F. Delecluse, M. Krams, O. Corabianu, F. H. Hochberg, Christopher J. Mathias, B. Debachy, C. M. Poser, L. Delodovici, A. Jimenez-Escrig, F. Baruzzi, F. Godenberg, D. Cucinotta, P. J. Garcia Ruiz, K. Maier-Hauff, P. R. Bar, R. Mezt, R. Jochens, S. Karakaneva, C. Roberti, E. Caballero, Joseph E. Parisi, M. Zamboni, T. Lacasa, B. Baklan, J. C. Gautier, J. A. Martinez-Matos, W. Pollmann, G. Thomas, L. Verze, E. Chleide, R. Alvarez Sala, I. Noel, E. Albuisson, O. Kastrup, S. I. Rapoport, H. J. Braune, H. Lörler, M. Le Merrer, A. Biraben, S. Soler, S. J. Taagholt, U. Meyding-Lamadé, K. Bleasdale-Barr, Isabella Moroni, Y. Campos, J. Matias-Guiu, G. Edan, M. G. Bousser, John B. Clark, J. Garcia de Yebenes, N. K. Olsen, P. Hitzenberger, S. Einius, Aj Thompson, Ch. J. Vecht, T. Crepin-Leblond, Klaus L. Leenders, A. Di Muzio, L. Georgieva, René Spiegel, K. Sabey, D. Ménégalli, J. Meulstee, U. Liszka, P. Giral, C. Sunol, J. M. Espadaler, A. D. Crockar, K. Varli, G. Giraud, P. J. Hülser, A. Benazzouz, A. Reggio, M. Salvatore, K. Genc, M. Kushnir, S. Barbieri, J. Ph. Azulay, M. Gianelli, N. Bathien, A. AlMemar, F. Hentati, I. Ragueneau, F. Chiarotti, R. C. F. Smits, A. K. Asbury, F. Lacruz, B. Muller, Alan J. Thompson, Gordon Smith, K. Schmidt, C. Daems Monpeun, Juergen Weber, A. Arboix, G. R. Fink, A. M. Cobo, M. Ait Kaci Ahmed, E. Gencheva, Israel-Biet, G. Schlaug, P. De Jonghe, Philip Scheltens, K. Toyka, P. Gonzalez-Porque, A. Cila, J. M. Fernandez, P. Augustin, J. Siclia, S. Medaglini, D. E. Ziogas, A. Feve, L. Kater, G. J. E. Rinkel, D. Leppert, Rüdiger J. Seitz, S. Ried, C. Turc-Carel, G. Smeyers, F. Godinho, M. Czygan, M. Rijntjes, E. Aversa, M. Frigo, Leif Østergaard, J. L. Munoz Blanco, A. Cruz-Matinez, J. De Reuck, C. Theillet, T. Barroso, V. Oikonen, Florence Lebert, M. Kilinc, C. Cordon-Cardon, G. Stoll, E. Thiery, F. Pulcinelli, J. Solski, M. Schmiegelow, L. J. Polman, P. Fernandez-Calle, C. Wikkelso, M. Ben Hamida, M. Laska, E. Kott, W. Sulkowski, C. Lucas, N. M. Bornstein, D. Schmitz, M. W. Lammers, A. de Louw, R. J. S. Wise, P. A. van Darn, C. Antozzi, P. Villanueva, P. H. E. Hilkens, C. Constantin, W. Ricart, A. Wolf, M. Gamba, P. Maguire, Alessandro Padovani, B. M. Patten, Marie Sarazin, H. Ackermann, L. Durelli, S. Timsit, Sebastian Jander, B. W. Scheithauer, G. Demir, J. P. Neau, P. Barbanti, A. Brand, N. AraÇ, V. Fischer-Gagnepain, R. Marchioli, G. Serratrice, C. Maugard-Louboutin, G. T. Spencer, D. Lücke, G. Mainardi, K. Harmant Van Rijckevorsel, G. B. Creel, R. Manzanares, Francesco Fortunato, A. May, J. Workman, K. Johkura, E. Fernandez, Carlo Colosimo, L. Calliauw, L. Bet, Félix F. Cruz-Sánchez, M. Dhib, H. Meinardi, F. Carrara, J. Kuehnen, C. Peiro, H. Lassmann, K. Skovgaard Olsen, A. McDonald, L. Sciulli, A. Cobo, A. Monticelli, B. Conrad, J. Bagunya, J. Benitez, V. Desnizza, B. Dupont, O. Delrieu, D. Moraes, J. J. Heimans, F. Garcia Rio, M. Matsumto, A. Fernandez, R. Nermni, R. Chalmers, M. J. Marchau, F. Aguado, P. Velupillai, P. J. Martin, P. Tassan, V. Demarin, A. Engelien, T. Gerriets, Comar, J. L. Carrasco, J. P. Pruvo, A. Lopez de Munain, D. Pavitt, J. Alarcon, Chris H. Polman, B. Guldin, N. Yeni, Hartmut Brückmann, N. Wilczak, H. Szwed, R. Causaran, G. Kyriazis, M. E. Westarp, M. Gasparini, N. Pecora, J. M. Roda, E. Lang, V. Scaioli, David R. Fish, D. Caputo, O. Gratzl, R. Mercelis, A. Perretti, G. Steimetz, I. Link, C. Rigoletto, A. Catafau, G. Lucotte, M. Buti, G. Fagiolari, A. Piqueras, C. Godinot, J. C. Meurice, Erodriguez J. Dominigo, F. Lionnet, H. Grzelec, David J. Brooks, P. M. G. Munro, F. X. Weilbach, M. Maiwald, W. Split, B. Widjaja-Cramer, V. Ozturk, J. Colas, E. Brizioli, J. Calleja, L. Publio, M. Desi, R. Soffietti, P. Cortinovis-Tourniaire, E. F. Gonano, G. Cavaletti, S. Uselli, K. Westerlind, H. Betuel, C. O. Dhiver, H. Guggenheim, M. Hamon, R. Fazio, P. Lehikoinen, A. Esser, B. Sadzot, G. Fink, Angelo Antonini, D. Bendahan, V. Di Carlo, G. Galardi, A. F. Boller, M. Aksenova, Del Fiore, V. de la Sayette, H. Chabriat, A. Nicoletti, A. Dilouya, M. L. Harpin, E. Rouillet, J. Stam, A. Wolters, M. R. Delgado, Eduardo Tolosa, G. Said, A. J. Lees, L. Rinaldi, A. Schulze-Bonhage, MA Ron, C. Lefebvre, E. W. Radü, R. Alvarez, M. L. Bots, P. Reganati, S. Palazzi, A. Poggi, N. J. Scolding, V. Sazdovitch, T. Moreau, E. Maes, M. A. Estelies, P. Petkova, Jose-Felix Marti-Masso, G De La Meilleure, N. Mullatti, M. Rodegher, N. C. Notermans, T. A. T. Warner, S. Aktan, J. P. Louboutin, L. Volpe, C. Scheidt, W. Aust, C. M. Wiles, U. Schneider, S. K. Braekken, W. R. Willems, K. Usuku, Peter M. Rothwell, C. Talamon, M. L. Sacchetti, A. Codina, M. H. Marion, A. Santoro, J. Roda, A. Bordoni, D. J. Taylor, S. Ertas, H. H. Emmen, J. Vichez, V. BesanÇon, R. E. Passingham, M. L. Malosio, A. Vérier, M. Bamberg, A. W. Hansen, E. Mostacero, G. Gaudriault, Marie Vidailhet, B. Birebent, K. Strijckmans, F. Giannini, T. Kammer, I. Araujo, J. Nowicki, E. Nikolov, A. Hutzelmann, R. Gherardi, J. Verroust, L. Austoni, A. Scheller, A. Vazquez, S. Matheron, H. Holthausen, J. M. Gerard, M. Bataillard, S. Dethy, V. H. Patterson, V. Ivanez, N. P. Hirsch, F. Ozer, M. Sutter, C. Jacomet, M. Mora, Bruno Colombo, A. Sarropoulos, T. H. Papapetropoulos, M. Schwarz, D. S. Dinner, N. Acarin, B. Iandolo, J. O. Riis, P. R. J. Barnes, F. Taroni, J. Kazenwadel, L. Torre, A. Lugaresi, I. L. Henriques, S. Pauli, S. Alfonso, Pedro Quesada, A. S. T. Planting, J. M. Castilla, Thomas Gasser, M. Van der Linden, A. Alfaro, E. Nobile-Orazio, G. Popova, W. Vaalburg, F. G. A. van der Mech, L. Williams, F. Medina, J. P. Vernant, J. Yaouanq, B. Storch-Hagenlocher, A. Potemkowski, R. Riva, M. H. Mahagne, M. Ozturk, Ve. Drory, N. Konic, C. Jungreis, A. Pou Serradell, J. L. Gauvrit, G. J. Chelune, S. Hermandez, T. Dingus, L. Hewer, Ch. Koch, M. N. Metz-Lutz, G. Parlato, M. Sinaki, Charles Pierrot-Deseilligny, H. C. Diener, J. Broeckx, J. Weill-Fulazza, M. L. Villar, M. Rizzo, O. Ganslandt, C. Duran, N. A. Fletcher, G. Di Giovacchino, Susan T. Iannaccone, C. Kolig, N. Fabre, H. A. Crockard, Rita Bella, M. Tazir, E. Papagiannuli, K. Overgaard, Emma Ciafaloni, I. Lorenzetti, F. Viader, P. A. H. Millac, I. Montiel, L. H. Visser, M. Palomar, P. L. Murgia, H. Pedersen, Rafael Blesa, S. Seddigh, W. O. Renier, I. Lemahieu, H. M. L. Jansen, L. Rosin, J. Galofre, K. Mattos, M. Pondal, G. M. Hadjigeorgiou, D. Francis, L. Cantin, D. Stegeman, M. Rango, A. B. M. F. Karim, S. Schraff, B. Castellotti, I. Iriarte, E. Laborde, T. J. Tjan, R. Mutani, D. Toni, B. Bergaasco, J. G. Young, C. Klotzsch, A. Zincone, X. Ducrocq, M. Uchuya, O. J. Kolar, A. Quattrone, T. Bauermann, Nereo Bresolin, J. Vallée, B. C. Jacobs, A. Campos, Werner Poewe, J. A. Villanueva, A. W. Kornhuber, A. Malafosse, E. Diez-Tejedor, G. Jungreia, M. J. A. Puchner, A. Komiyama, O. Saribas, V. Volpini, L. Geremia, S. Bressi, A. Nibbio, Timothy E. Bates, T. z. Tzonev, E. Ideman, G. A. Damlacik, G. Martino, G. Crepaldi, T. Martino, Kjell Någren, E. Idiman, D. Samuel, J. M. Perez Trullen, Y. van der Graaf, J. O. Thorell, M. J. M. Dupuis, E. Sieber, R. D'Alessandro, C. Cazzaniga, J. Faiss, A. Tanguy, A. Schick, I. Hoksergen, A. Cardozo, R. Shakarishvili, G. K. Wennlng, J. L. Marti-Vilalta, J. Weissenbach, I. L. Simone, Amalia C. Bruni, Darius J. Adams, C. Weiller, A. Pietrangeli, F. Croria, C. Vigo-Pelfrey, Patricia Limousin, A. Ducros, G. Conti, O. Lindvall, E. Richter, M. Zuffi, A. Nappo, T. Riise, J. Wijdenes, M. J. Fernandez, J. Rosell, P. Vermersh, S. Servidei, M. S. C. Verdugo, F. Gouttiere, W. Solbach, M. Malbezin, I. S. Watanabe, A. Tumac, W. I. McDonald, D. A. Butterfield, P. P. Costa, F. deRino, F. Bamonti, J. M. Cesar, C. H. Lahoz, I. Mosely, M. Starck, M. H. Lemaitre, K. M. Stephan, S. Tex, R. Bokonjic, I. Mollee, L. Pastena, M. Gutierrez, F. Boiler, M. C. Martinez-Para, M. Velicogna, O. Obuz, A. Grinspan, M. Guarino, L. M. Cartier, E. Ruiz, D. Gambi, S. Messina, M. Villa, Michael G. Hanna, J. Valk, Leone Pascual, M. Clanet, Z. Argov, B. Ryniewicz, E. Magni, B. Berlanga, K. S. Wong, C. Gellera, C. Prevost, F. Gonzalez-Huix, R. Petraroli, J. E. G. Benedikz, I. Kojder, C. Bommelaer, L. Perusse, M. R. Bangioanni, Guy M. McKhann, A. Molina, C. Fresquet, E. Sindern, Florence Pasquier, M. J. Rosas, M. Altieri, O. Simoncini, M. Koutroumanidis, C. A. F. Tulleken, M. Dary-Auriol, S. Oueslati, H. Kruyer, I. Nishisho, C. R. Horning, A. Vital, G. V. Czettritz, J. Ph. Neau, B. Mihout, A. Ameri, M. Francis, S. Quasthoff, D. Taussig, S. Blunt, P. Valentin, C. Y. Gao, O. Heinzlef, H. d'Allens, C. Coudero, M. Erfas, G. Borghero, P. J. Modrego Pardo, M. C. Patrosso, N. L. Gershfeld, P. A. J. M. Boon, O. Sabouraud, M. Lara, J. Svennevig, G. L. Lenzi, A. Barrio, H. Villaroya, JosÇ M. Manubens, O. Boespflug-Tanguy, M. Carreras, D. A. Costiga, J. P. Breux, S. Lynn, C. Oliveras Ley, A. G. Herbaut, J. Nos, C. Tornali, Y. A. Hekster, J. L. Chopard, J. M. Manubens, P. Chemouilli, A. Jovicic, F. Dworzak, S. Smirne, S. E. Soudain, B. Gallano, D. Lubach, G. Masullo, G. Izquierdo, A. Pascual Leone Pascual, A. Sessa, V. Freitas, O. Crambes, L. Ouss, G. W. Van Dijk, P. Marchettini, P. Confalonieri, M. Donaghy, A. Munnich, M. Corbo, and M. E. L. van der Burg
Subjects: Neurology, business.industry, Media studies, Library science, Medicine, Neurology (clinical), business
Published: 1994
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10. Will the penicillin be exceeded?

Author: M, PANISSET and A, FRAPPIER
Subjects: Penicillins
Published: 2010

11. Clinical trials in cognitive impairment in the elderly

Author: M. Panisset, Françoise Forette, and François Boller
Subjects: Aging, medicine.medical_specialty, Activities of daily living, business.industry, Cognition, Disease, medicine.disease, Clinical trial, Alzheimer Disease, Activities of Daily Living, mental disorders, Tacrine, medicine, Clinical Global Impression, Humans, Dementia, Geriatrics and Gerontology, Alzheimer's disease, Psychiatry, Vascular dementia, Intensive care medicine, business
Abstract: The pharmacological treatment of dementia is particularly difficult because of the uncertainty of the diagnosis and of the underlying pathogenetic processes, and because of the variable severity of the syndrome. This paper suggests several lines for future therapeutic trials in dementia. These include the need to work with homogenous populations and to take into account the natural history of the disease, the pharmacologic characteristics of the drugs, and the particular ethical considerations pertaining to demented patients. We review the diagnostic criteria for dementia, Alzheimer's disease (AD) and vascular dementia. We discuss the different types of therapeutic trial design, the problem of the duration of the protocol, the tools to evaluate the cognitive effects and the activities of daily living, and the use of a Clinical Global Impression of Change (CGIC). The results of some therapeutic trials in AD and vascular dementia are given as examples.
Published: 1992
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12. Multiple cranial neuropathy as a feature of internal carotid artery dissection

Author: M Panisset and B H Eidelman
Subjects: Adult, Carotid Artery Diseases, Male, Skull Neoplasms, Dissection (medical), Diagnosis, Differential, Aneurysm, medicine, Humans, Paralysis, Paresis, Advanced and Specialized Nursing, Internal carotid artery dissection, medicine.diagnostic_test, business.industry, Cranial nerves, Intracranial Aneurysm, Magnetic resonance imaging, Anatomy, Middle Aged, medicine.disease, Magnetic Resonance Imaging, Cranial Nerve Diseases, Cerebral Angiography, Aortic Dissection, Angiography, Neurology (clinical), medicine.symptom, Tomography, X-Ray Computed, Cardiology and Cardiovascular Medicine, business, Carotid Artery, Internal, Cerebral angiography
Abstract: The traditional presentation of spontaneous internal carotid artery dissection includes ipsilateral hemicranial headache, oculosympathetic paresis, and contralateral focal cerebral ischemic deficits. However, we describe two cases with multiple cranial nerve involvement ipsilateral to the dissection as the principal feature. The first patient, a 36-year-old man, had involvement of the 9th, 10th, 11th, and 12th cranial nerves. The second case was a 53-year-old man with abnormalities of the 5th, 7th, 9th, 10th, and 12th cranial nerves. In both, magnetic resonance imaging revealed a ring-like area of abnormal signal intensity surrounding the carotid artery at the skull base. Carotid angiography was consistent with the suggestion of dissection on the magnetic resonance studies in both cases. The patients recovered without anticoagulation. Internal carotid artery dissection may thus present with multiple cranial nerve palsies, which could be mistaken for an infiltrating tumor of the skull base. Magnetic resonance imaging is useful in identifying the condition.
Published: 1990
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13. Neurofilament M gene in a French-Canadian population with Parkinson's disease

Author: M. Panisset, Dennis E. Bulman, David Grimes, and Fabin Han
Subjects: Male, Canada, Parkinson's disease, Population, DNA Mutational Analysis, Polymerase Chain Reaction, Central nervous system disease, Neurofilament Proteins, Medicine, Humans, education, Gene, Aged, education.field_of_study, business.industry, Parkinson Disease, General Medicine, Middle Aged, medicine.disease, Molecular biology, Neurofilament-M, Neurology, Mutation, French canadian, Female, Neurology (clinical), business, Neuroscience
Abstract: Background:Recently, a single base pair substitution (G1747A) mutation of the neurofilament M (NF-M) gene was reported in a French-Canadian patient with early onset Parkinson’s disease (PD). Three unaffected siblings were found to be heterozygotes for the NF-M Gly336Ser mutation but, to date, no other affected PD individuals have been found with a similar mutation. No other individuals with Parkinson’s disease and of similar ethnic background have been screened for this mutation.Methods:We screened 102 French-Canadian patients with definite PD and 45 French-Canadian controls for this substitution in the NF-M gene using a PCR-restriction enzyme digestion method.Results:None of the patients or controls carried this mutation.Conclusion:Our results would indicate that this mutation is not common even in a PD population of similar ethnic background and suggest this change represents a rare variant. However, these results do not exclude the possibility that other mutations in this gene could be present.
Published: 2005

14. Differential memory impairment in dementia with Lewy bodies and Alzheimer's disease

Author: M, Simard, R, van Reekum, D, Myran, M, Panisset, T, Cohen, M, Freedman, S, Black, and B, Suvajac
Subjects: Aged, 80 and over, Lewy Body Disease, Male, Analysis of Variance, Memory Disorders, Spatial Behavior, Middle Aged, Memory, Short-Term, Alzheimer Disease, Space Perception, Humans, Female, Perceptual Masking, Aged
Abstract: This study was conducted in order to elucidate the functioning of the Central Executive System of Working Memory (WM) and to clarify the status of other cognitive functions in Dementia with Lewy bodies (DLB) and Alzheimer's disease (AD). Fourteen DLB, 22 AD, and 23 control subjects were assessed with the dual task paradigm and other cognitive tests. When compared with controls, DLB subjects performed more poorly in concurrent conditions on semantic WM tasks, and AD subjects performed more poorly on the spatial WM task. The DLB subjects had an inferior verbal span and AD subjects, an inferior recall on the CVLT. These data suggest relative impairments of verbal and semantic WM in DLB and relative impairments of spatial WM and verbal episodic memory in AD.
Published: 2004

15. 2.240 RASAGILINE IMPROVES THE QUALITY OF SLEEP IN PATIENTS WITH PARKINSON'S DISEASE: RESULTS OF THE RASAGILINE EFFECT ON SLEEP TRIAL (REST)

Author: S. Chouinard and M. Panisset
Subjects: Rasagiline, medicine.medical_specialty, Parkinson's disease, business.industry, Quality of sleep, medicine.disease, Sleep in non-human animals, chemistry.chemical_compound, Physical medicine and rehabilitation, Neurology, chemistry, Medicine, In patient, Neurology (clinical), Geriatrics and Gerontology, business, Rest (music)
Published: 2012
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16. Linkage stratification and mutation analysis at the Parkin locus identifies mutation positive Parkinson's disease families

Author: William C. Nichols, J. Hubble, H. Fernandez, M. Aminoff, C. O'Brien, D. Truong, D. A. Grimes, A. Rudolph, F. Walker, L. Elmer, S. Factor, R. Kurlan, K. Blindauer, Paul J. Tuite, L. Seeberger, Richard B. Dewey, J. Rao, D. Jennings, P. M. Conneally, R. Pahwa, R. Hauser, W. Koller, T. Ajax, M. Fernandez, J. Sutton, B. Racette, R. Uitti, K. Sethi, A. Feigin, J. Jankovic, R. Pfeiffer, C. Serrano Ramos, M. Stacy, L. Shulman, M. F. Gordon, L. Sudarsky, K. Marek, J. Bertoni, V. Calabresse, M. Velickovic, R. Rodnitzyk, Juan Sanchez-Ramos, Sean K. Uniacke, P. Gordon, A. Dalvi, N. Hermanowicz, T. Zesiewicz, T. Simuni, U. Jung Kang, C. Shults, P. Lewitt, M. Leehey, Mayank Pathak, Tatiana Foroud, J. Carter, R. Saunders Pullman, B. Manyam, M. Panisset, Michael W. Pauciulo, G. D. Podakalny, T. Mendis, D. Simon, J. Friedman, W. Martin, Richard Camicioli, Joanne Wojcieszek, N. Pankratz, C. Halter, K. Marder, S. Reich, A. Nieves, A. Rajput, and O. Suchowersky
Subjects: Adult, Genetic Markers, Parkinson's disease, Genetic Linkage, Ubiquitin-Protein Ligases, DNA Mutational Analysis, Physiology, Biology, Parkin, Central nervous system disease, Ligases, Degenerative disease, Genetics, medicine, Humans, Genetic Testing, First-degree relatives, Genetics (clinical), Aged, Lewy body, Genetic Carrier Screening, Family aggregation, Parkinson Disease, Odds ratio, Middle Aged, medicine.disease, Mutation, Letter to JMG
Abstract: Parkinson's disease (PD) is one of the most common neurological disorders in humans with an overall prevalence of 1:1000 with the incidence increasing to as high as 3.4% among people aged 75 years.1,2 The clinical phenotype includes resting tremor, muscular rigidity, bradykinesia, and postural instability. The signs and symptoms of the disease are the consequence of a striatal deficiency of dopamine resulting from neuronal death in the substantia nigra. It is characterised by the presence of the Lewy body, an intracytoplasmic inclusion body found in many brain regions which is not entirely specific to, but is a highly sensitive marker for, Parkinson's disease. The pathogenesis of idiopathic Parkinson's disease is unknown. For the overwhelming majority of PD patients, the disease has previously been thought to occur sporadically. However, there is increasing evidence of a genetic contribution to the disorder.1,3 Recently, two studies have investigated familial aggregation of PD using large, population based, case-control studies. Elbaz et al 4 reported an odds ratio of 3.2 for the presence of PD in first degree relatives (parents and sibs) of 175 cases as compared to 481 controls. Analyses stratified by age showed this aggregation to be stronger for younger PD patients. Familial aggregation of PD in Iceland was studied using a cohort of 772 cases, with 560 having onset of disease at >50 years of age.5 In this study, the odds ratio for PD was 6.7 for sibs and 3.2 for offspring of affected subjects. These studies are consistent with others reporting the risk to be anywhere from two to 14 times higher for first degree relatives as compared to the risk in members of unaffected families.6–9 Genetic linkage analyses in families with either autosomal dominant forms of PD or with an autosomal recessive, juvenile form …
Published: 2002

17. Manganese neurotoxicity, a continuum of dysfunction: results from a community based study

Author: D, Mergler, M, Baldwin, S, Bélanger, F, Larribe, A, Beuter, R, Bowler, M, Panisset, R, Edwards, A, de Geoffroy, M P, Sassine, and K, Hudnell
Subjects: Adult, Male, Manganese, Time Factors, Dose-Response Relationship, Drug, Iron, Manganese Poisoning, Age Factors, Environmental Exposure, Mercury, Middle Aged, Neuropsychological Tests, Random Allocation, Sex Factors, Lead, Motor Skills, Surveys and Questionnaires, Multivariate Analysis, Humans, Female, Nervous System Diseases, Aged
Abstract: Excessive manganese (Mn) has been associated with neurobehavioral deficits and neurological and/or neuropsychiatric illness, but the level at which this metal can cause adverse neurotoxic effects, particularly with long-term exposure, is still unknown. The objective of the present study was to assess nervous system functions in residents exposed to manganese from a variety of environmental sources. A random stratified sampling procedure was used to select participants; persons with a history of workplace exposure to Mn and other neurotoxic substances were excluded. A self-administered questionnaire provided data on socio-demographic variables. Blood samples were analyzed for total manganese (MnB), lead, mercury and serum iron. Nervous system assessment included computer and hand-administered neurobehavioral tests, computerized neuromotor tests, sensory evaluation and a neurological examination. The present analyses include 273 persons (151 women and 122 men); MnB range: 2.5 micrograms/L-15.9 micrograms/L (median: 7.3 micrograms/L). Multivariate analyses were used and neuro-outcomes were examined with respect to MnB, taking into account potential confounders and covariables. Results were grouped according to neurofunctional areas and MANOVA analyses revealed that higher MnB (7.5 micrograms/L) was significantly associated with changes in coordinated upper limb movements (Wilks' lambda = 0.92; p = 0.04) and poorer learning and recall (men: Wilks' lambda = 0.77; p = 0.002; women: Wilks' lambda = 0.86; p = 0.04). Further analyses revealed that with increasing log MnB (Simple regression: p0.05) performance on a pointing task was poorer, frequency dispersion of hand-arm tremor decreased, while harmonic index increased, and the velocity of a pronation/supination arm movement was slower. An Mn-age interaction was observed for certain motor tasks, with the poorest performance observed among those _50 y and in the higher MnB category. Differences between genders suggest that men may be at greater risk than women, although effects were also observed in women. These findings are consistent with the hypothesis that Mn neurotoxicity can be viewed on a continuum of dysfunction, with early, subtle changes at lower exposure levels.
Published: 1999

18. Preliminary evidence of neurotoxicity associated with eating fish from the Upper St. Lawrence River Lakes

Author: D, Mergler, S, Bélanger, F, Larribe, M, Panisset, R, Bowler, M, Baldwin, J, Lebel, and K, Hudnell
Subjects: Adult, Male, Meat, Fishes, Quebec, Food Contamination, Mercury, Middle Aged, Neuropsychological Tests, Treatment Outcome, Lead, Socioeconomic Factors, Surveys and Questionnaires, Animals, Humans, Female, Nervous System Diseases, Aged
Abstract: Pollution of hydrographic basins has affected the flora and fauna that thrive in these aquatic ecosystems, and fish, which constitute an important food resource, often contain a plethora of potentially toxic chemicals. In a major research project on early neurotoxic effects of environmental exposure to manganese among residents in Southwest Quebec, fish consumption from 2 lakes of the Upper St. Lawrence River System, was surveyed as a potential confounding factor. Participants were selected using a random, stratified sampling strategy from lists of the Quebec Health Plan. Following exclusions, 273 men and women between 20-69 years were retained for the present analysis. A total of 103 (37.7%) reported eating fish from the Upper St. Lawrence. Although fisheaters and non-fisheaters were similar for most socio-demographic variables, significantly more fisheaters (65.2%) reported consuming alcoholic beverages as compared to non-fisheaters (42.4%) (Chi Sq.0.01). To eliminate this possible bias, fisheaters were matched to non-fisheaters for the variables sex, alcohol consumption (never or occasionally vs. regularly), age (+/-5y) and education (+/-2y). A total of 63 matched pairs were thus created. Paired analyses (t-test or Signed Rank) showed that fisheaters had higher levels of blood organic mercury and lead. Analysis of nervous system functions revealed that both groups performed similarly on tests of sensory function, visual memory and recognition, fine motor performance and some motor tests, but fisheaters performed significantly more poorly (p0.05) on tests requiring cognitive flexibility, word naming, auditory recall, and more complex motor tasks. The profile of deficits is consistent with diminished capacity for information processing. These observations were made within a study that was not specifically designed to examine the effects of fish eating from these two lakes, and the characterization of fish dietary habits has many limitations. Nevertheless, the findings are sufficiently compelling to warrant further studies, since fish from the Upper St. Lawrence Lakes are known to contain multiple neurotoxic substances.
Published: 1998

19. Current diagnostic methods and outcome variables for clinical investigation of Alzheimer's disease

Author: S, Gauthier and M, Panisset
Subjects: Diagnosis, Differential, Psychiatric Status Rating Scales, Treatment Outcome, Alzheimer Disease, Humans, Aged
Abstract: The diagnosis of Alzheimer's disease (AD) is done by a careful history, requiring reliable informants and serial observations. The main differential diagnosis is depression, delirium, and inappropriate use of psychotropic drugs. Other common causes of dementia such as vascular, Lewy body disease, frontal lobe degeneration, can be distinguished by the pattern of symptoms and findings on the physical examination. A minimal amount of laboratory investigation is usually required. The natural history of AD, with progressive involvement of cognition, activities of daily living and behaviour, justifies the need of outcome variables addressing these specific symptomatic domains. These are complemented by global clinical assessment tools for disease staging and disease progression. A new challenge is to select from outcome variables used in clinical investigations the most appropriate tools for regular clinical practice.
Published: 1998

20. Levodopa improves motor function without impairing cognition in mild non-demented Parkinson's disease patients. Parkinson Study Group

Author: J H, Growdon, K, Kieburtz, M P, McDermott, M, Panisset, and J H, Friedman
Subjects: Antiparkinson Agents, Levodopa, Male, Psychiatric Status Rating Scales, Analysis of Variance, Cognition, Humans, Dementia, Female, Parkinson Disease, Middle Aged, Psychomotor Performance, Aged
Abstract: The objective of the study was to determine the effects of short-term levodopa administration on motor, cognitive, and psychiatric aspects of Parkinson's disease (PD).The effects of levodopa on mental processes in PD are controversial. Opinions range from the claim that levodopa improves cognition to the opposite view that levodopa causes or exacerbates dementia, delusions, and hallucinations. Of the 800 idiopathic PD patients enrolled in the original DATATOP study, 387 reached the end point of functional disabilities sufficiently severe to require levodopa treatment. There were 263 men and 124 women who were comparable with regard to age, symptom duration of PD, and measures of PD severity. We compared test scores on motor performance, cognitive function, and psychiatric status before levodopa and again within 6 months after initiation of levodopa therapy.Levodopa administration improved all motor functions significantly. The improvement was significantly greater in women than in men. Levodopa administration did not worsen scores on any cognitive tests, and there were quantitatively small but significant improvements in tests of frontal lobe function. Levodopa exerted only minor effects on psychiatric measures. There were small but significant decreases in scores for depression, and increases in vivid dreams and hallucinations.Levodopa administration for up to 6 months in dosages sufficient to improve motor function has only small effects on cognitive function and psychiatric status in mild to moderate PD patients. We conclude that motor symptoms in early PD, which result from dopamine depletion, are dissociable from cognitive functions and psychiatric status, which may be more dependent on nondopaminergic mechanisms.
Published: 1998

21. Current diagnostic methods and outcome variables for clinical investigation of Alzheimer’s disease

Author: S. Gauthier and M. Panisset
Subjects: Pediatrics, medicine.medical_specialty, Diagnostic methods, medicine.diagnostic_test, business.industry, Physical examination, Disease, medicine.disease, medicine, Dementia, Delirium, Differential diagnosis, medicine.symptom, business, Lewy body disease, Depression (differential diagnoses)
Abstract: The diagnosis of Alzheimer’s disease (AD) is done by a careful history, requiring reliable informants and serial observations. The main differential diagnosis is depression, delirium, and inappropriate use of psychotropic drugs. Other common causes of dementia such as vascular, Lewy body disease, frontal lobe degeneration, can be distinguished by the pattern of symptoms and findings on the physical examination. A minimal amount of laboratory investigation is usually required.
Published: 1998
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22. The relationship between extrapyramidal signs and cognitive performance in patients with Alzheimer's disease enrolled in the CERAD Study. Consortium to Establish a Registry for Alzheimer's Disease

Author: C M, Clark, D, Ewbank, A, Lerner, R, Doody, V W, Henderson, M, Panisset, J C, Morris, G G, Fillenbaum, and A, Heyman
Subjects: Male, Psychiatric Status Rating Scales, Cognition, Basal Ganglia Diseases, Alzheimer Disease, Humans, Female, Registries, Aged
Abstract: The objective of this study was to determine the relationship between the presence of extrapyramidal signs and the severity of cognitive and functional impairment in patients with Alzheimer's disease (AD). Eleven university medical centers in the United States and France participated in the Consortium to Establish a Registry for Alzheimer's Disease (CERAD) study of extrapyramidal signs in AD. Forty-seven patients with probable AD who had extrapyramidal signs were matched by sex, race, education, and age with 132 probable AD patients without extrapyramidal signs. The main outcome measures were the Clinical Dementia Rating, Blessed Dementia Scale, and the CERAD Neuropsychology Battery (verbal fluency, naming, Mini-Mental State Examination, word list learning, word list recall, savings ratio, word list recognition, and constructional praxis). AD patients with extrapyramidal signs performed more poorly than AD patients without parkinsonism on various neuropsychological tests, even after controlling for the Clinical Dementia Rating and reported duration of cognitive impairment. The severity of the extrapyramidal manifestations was related to the degree of cognitive and functional impairment. Muscular rigidity and bradykinesia were the most frequent extrapyramidal signs associated with AD. Patients with AD associated with extrapyramidal signs have greater cognitive and functional impairment than AD patients without clinical evidence of parkinsonism.
Published: 1997

23. Effect of Exercise on the Clinical Progression of Parkinson Disease (P06.082)

Author: P. Gou, S. Chouinard, N. Jodoin, S. Diab, and M. Panisset
Subjects: Neurology (clinical)
Published: 2012
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24. Severe impairment battery. A neuropsychological test for severely demented patients

Author: M Roudier, M Panisset, François Boller, and J Saxton
Subjects: Male, Longitudinal study, medicine.medical_specialty, Pediatrics, Psychometrics, Population, Neuropsychological Tests, Severity of Illness Index, Cognition, Arts and Humanities (miscellaneous), Severity of illness, medicine, Dementia, Humans, Psychiatry, education, Aged, Aged, 80 and over, education.field_of_study, medicine.diagnostic_test, Neuropsychology, Neuropsychological test, medicine.disease, Female, Neurology (clinical), Alzheimer's disease, Psychology
Abstract: Objective: Patients with progressive dementia invariably evolve to a stage where they can no longer be tested by standard neuropsychological tests. We studied the use of the Severe Impairment Battery (SIB) in such patients. Design: Case series. Setting: Geriatric long-term facility. Patients: Sixty-nine patients who met the criteria of the Diagnostic and Statistical Manual for Mental Disorders, Revised Third Edition , for dementia were selected. The diagnosis of probable Alzheimer's disease was established according to the guidelines suggested by the National Institute of Neurologic and Communicative Diseases and Stroke/Alzheimer's Disease and Related Disorders Association. There were 18 men and 51 women. The mean age of the population was 82.99±5.66 years. The mean Mini-Mental State Examination (MMSE) score was 10.71±6.14. Main Outcome Measure: To characterize the cognitive profile and evolution of severely demented patients by means of the SIB. Results: The mean score on the SIB was 92.52±31.92, with a possible maximum of 133 points. Subgroups of patients with the most severe degree of dementia (MMSE scores of 0 to 5 and 6 to 11) showed significant differences in their scores on the SIB. In contrast, no differences were found between subgroups with MMSE scores of 6 to 11,12 to 17, and greater than 17. Fifteen patients who had MMSE scores of less than 6 had SIB scores ranging from 7 to 81. All cognitive domains showed a deterioration across the four severity groups as determined by the MMSE scores and also during a longitudinal study performed on 26 patients. Conclusion: Our study indicates that the SIB is useful for the neuropsychological evaluation of severely demented patients and for their follow-up.
Published: 1994

25. Trial designs and outcome variables in anti-dementia drug testing

Author: M. Panisset and S. Gauthier
Subjects: Protocol (science), Drug, medicine.medical_specialty, business.industry, media_common.quotation_subject, Disease, medicine.disease, Outcome (game theory), Natural history, Primary progressive aphasia, medicine, Dementia, Intensive care medicine, business, Vascular dementia, media_common
Abstract: Designing a protocol for a therapeutic trial in Alzheimer’s disease (AD) is a very complex undertaking and is based on knowledge accumulated over the years about the manifestations of the disease and its diagnostic criteria, its natural history and its pathophysiology. Knowledge about the new compound from its early pre-clinical and clinical phases of development is equally important. Selection of validated outcome variables and a well coordinated project are the other keys for the successful and scientific validation of new medication for the treatment of AD.
Published: 1994
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26. [A battery of neuropsychological tests for severe dementia. An evaluation study]

Author: M, Panisset, M, Roudier, J, Saxton, and F, Boller
Subjects: Aged, 80 and over, Male, Alzheimer Disease, Humans, Reproducibility of Results, Female, Neuropsychological Tests, Aged
Abstract: All patients with Alzheimer's disease progress, more or less rapidly, to a stage where they can no longer be tested with the standard neuropsychological tools. The Severe Impairment Battery (SIB) is a neuropsychological tool used to evaluate patients with severe dementia. Its maximum score is 133. The purpose of this study was to assess the reliability of a French version of SIB. Fifty-four patients with Alzheimer's disease (mean Mini Mental State examination [MMS] 10.11 +/- 6.38) entered the study. Their mean SIB score was 88.4 +/- 33.3. The patients who were most affected (MMS below 5) were distributed by SIB from 7 to 80 points. An inter-examinator study of 36 patients yielded a correlation coefficient r = 0.9957 (P = 0.0001). In an inter-test study of 17 patients the correlation coefficient was also very high (r = 0.874, P = 0.0001). Thus, SIB seems to be a reliable tool to evaluate cognitive functions in cases of severe dementia. The distribution of scores obtained by SIB in patients with an MMS below 10 demonstrates that even within groups of subjects with severe dementia distinctions can be made between their remaining capabilities. Using SIB in these patients should improve our knowledge of the natural course of the disease and give better correlation between clinical data and neuropathological and neurobiochemical tests on the one hand, and enable us to adjust to their condition the departments where these patients are treated on the other hand.
Published: 1992

27. P3.105 Impaired visuomotor adaptation in Parkinson disease during a computer-mediated aiming task

Author: Léna Lhuisset, M. Lemay, and M Panisset
Subjects: business.industry, Dopaminergic, Motor commands, Adaptation (eye), Disease, Direct feedback, Task (project management), Neurology, Dendritic cell vaccine, Medicine, Neurology (clinical), Geriatrics and Gerontology, Computer mouse, business, Neuroscience
Abstract: Aiming requires the transformation of visual information concerning target locations into motor commands to allow the hand to move toward the targets. Additional sensorimotor transformations are necessary if a tool such as a computer mouse is used and/or if the movement is not directly seen, but monitored on a computer screen. The aim of this study was to determine the effect of using a display requiring additional sensorimotor transformations on aiming behavior of PD patients. Ten PD in the ON state (63.63±6.99 years) and 10 age-matched healthy participants (63.22±7.17 years) performed aiming movements toward three targets in 2 conditions: direct visual feedback where the starting base, the hand and the target were readily available, and indirect feedback where visual information (target, mouse-cursor and starting base) were displayed on a computer screen. For both groups, reaction times were shorter and movement times were longer in the indirect feedback condition compared to the direct feedback condition. PD undershot the target significantly more than control participants and their movements were less straight and less smooth during computer-mediated aiming than during direct aiming. Overall, these results indicate that, even though time parameters were affected by the condition for all participants, movement execution was less efficient during computer-mediated aiming for PD only, suggesting that PD are affected by the supplementary sensorimotor transformation required during computer-mediated aiming. These abnormalities appear not to be corrected by dopaminergic therapy. These findings may have consequences for some day-today living activities such as using computers or driving. P3.106 Dendritic cell vaccine against human a-synuclein aggregates Background: Immunization therapies for neurodegenerative diseases were initially focused on clearing extracellular deposits of abnormal proteins typified by b amyloid of Alzheimer's disease. Immunization of mice with full-length human a-synuclein (a-syn) has been reported to result in high relative affinity antibodies
Published: 2009
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28. Neural response and gambling strategies differ between Parkinson's disease patients with and without impulse control disorders: An fMRI study

Author: B Stemmer, M Panisset, A Dagher, and CA Erickson
Subjects: Parkinson's disease, Psychotherapist, Neurology, Cognitive Neuroscience, medicine, medicine.disease, Psychology, Impulse control
Published: 2009
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29. End-Stage Alzheimer's Disease: Glasgow Coma Scale and the Neurologic Examination

Author: M. Panisset, J. Saxton, and F. Boiler
Subjects: Arts and Humanities (miscellaneous), Neurology (clinical)
Published: 1995
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30. Validation of a questionnaire to detect dementia cases in families

Author: M. Panisset, C. Berr, and D. Feteanu
Subjects: Aging, medicine.medical_specialty, business.industry, General Neuroscience, Medicine, Dementia, Neurology (clinical), Geriatrics and Gerontology, business, Psychiatry, medicine.disease, Developmental Biology
Published: 1992
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31. A randomized, double-blind, placebo-controlled trial with cerebrolysin in Alzheimer's disease

Author: H. Moessler, M. Panisset, Serge Gauthier, and M. Windisch
Subjects: Pharmacology, medicine.medical_specialty, business.industry, Placebo-controlled study, Double blind, Psychiatry and Mental health, chemistry.chemical_compound, Neurology, chemistry, Cerebrolysin, Internal medicine, medicine, Pharmacology (medical), Neurology (clinical), business, Biological Psychiatry
Published: 2000
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32. Advances in the diagnosis of Alzheimer's disease

Author: Judes Poirier, M. Panisset, and Serge Gauthier
Subjects: medicine.medical_specialty, business.industry, Medicine, Disease, business, Intensive care medicine, Biological Psychiatry
Published: 1997
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33. Frame-Based Interactive Image Guidance for Functional Neurosurgery

Author: R. M. Comeau, T. M. Peters, M. Panisset, Abbas F. Sadikot, B. L. K. Davey, and Gilles Bertrand
Subjects: Frame based, business.industry, Medicine, Surgery, Computer vision, Neurology (clinical), Artificial intelligence, business, Functional neurosurgery, Image guidance
Published: 1996
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34. Frame-Based Interactive Image Guidance for Functional Neurosurgery Paper#757

Author: Abbas F. Sadikot, M. Panisset, Gilles Bertrand, B. L. K. Davey, R. M. Comeau, and T. M. Peters
Subjects: Frame based, business.industry, Medicine, Surgery, Computer vision, Neurology (clinical), Artificial intelligence, Functional neurosurgery, Image guidance, business
Published: 1996
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35. Naturalistic evaluation of entacapone in patients with signs and symptoms of L-dopa wearing-off .

Author: M. Jog, M. Panisset, O. Suchowersky, B. Réhel, R. Schecter, Jog, M, Panisset, M, Suchowersky, O, Réhel, B, and Schecter, R
Subjects: ENTACAPONE, DOPA, PARKINSON'S disease, QUESTIONNAIRES, QUALITY of life, CATECHOLAMINES, DRUG therapy for Parkinson's disease, ANTIPARKINSONIAN agents, CLINICAL trials, COMPARATIVE studies, DRUG tolerance, DRUG administration, DOSE-effect relationship in pharmacology, LONGITUDINAL method, RESEARCH methodology, MEDICAL cooperation, ORGANIC compounds, PHENOLS, RESEARCH, EVALUATION research, TREATMENT effectiveness, THERAPEUTICS
Abstract: Objective: To evaluate the effectiveness of entacapone in the management of levodopa wearing-off in Parkinson's disease (PD) in a naturalistic, real-life setting.Research Design and Methods: This prospective, open-label, observational study included patients with idiopathic PD. Patients were eligible for inclusion if they had been taking 3-5 doses of levodopa per day for ≥2 months and had shown signs of levodopa wearing-off for ≥1 month. Subjects received entacapone (recommended dose: 1 × 200 mg tablet with each levodopa dose) for 28 days. Patients were asked to complete a wearing-off questionnaire and the eight-question Parkinson's Disease Questionnaire Quality of Life assessment (PDQ-8). Activities of daily living (both in the on and off states) were assessed using the Unified Parkinson's Disease Rating Scale (UPDRS) part II. Clinical Global Impression (CGI) of severity of PD-related symptoms was assessed using a modified CGI tool. Patient global assessment of severity of PD symptoms was also obtained.Results: A total of 341 patients were enrolled by 68 physicians across Canada. At Day 28, 56.9% of the subjects indicated improvement compared to baseline on the modified CGI of change (CGI-C); 21.4% reported no change. Improvements were also observed on the UPDRS II and the PDQ-8. Benefit from entacapone appeared to be relatively uniform across subgroups (e.g., number of daily levodopa doses, use of other anti-PD medications).Study Limitations: The results of this study may be biased due to factors inherent in open-label, community-based trials (e.g., compliance). This is, however, reflective of everyday clinical practice.Conclusions: In this naturalistic, real-life study, the addition of entacapone to levodopa therapy provided benefits in quality of life and activities of daily living for a substantial proportion of PD patients experiencing wearing-off. [ABSTRACT FROM AUTHOR]
Published: 2008
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36. Neurochimie des troubles cognitifs dans la maladie de Parkinson

Author: M. Panisset and M.-A. Bedard
Subjects: Gynecology, Central nervous system disease, medicine.medical_specialty, business.industry, medicine, General Medicine, business, medicine.disease, General Biochemistry, Genetics and Molecular Biology
Abstract: Au cours des vingt dernieres annees, on a reconnu la frequence, la nature et la physiopathologie des troubles cognitifs dans la maladie de Parkinson (MP). Certains syndromes y sont plus frequents que dans la population generale, comme la maladie d'Alzheimer ou la maladie des corps de Lewy diffus. Toutefois, un tableau caracteristique de deterioration cognitive, connu sous le terme de syndrome sous-cortico-frontal (SSCF), semble le mieux decrire les troubles cognitifs de la majorite des patients atteints d'une MP. Le SSCF serait la consequence d'une desafferentation du cortex frontal ou, d'un point de vue fonctionnel, de la desactivation de programmes cognitifs demeures intacts. On ne connait que partiellement la biochimie des voies sous-cortico-frontales et des programmes cognitifs qu'elle sous-tend dans le SSCF. Bien que la plupart des etudes aient mis l'accent sur les lesions dopaminergiques, l'exploration des systemes cholinergiques, seuls ou dans leurs interactions avec les lesions dopaminergiques, s'avere beaucoup plus feconde.
Published: 1995
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37. Correlations between the 'severe impairment battery' and other scales for dementia

Author: V Bonavita, J Saxton, François Boller, M Panisset, S Carlomagno, Vincenzo Parlato, A Iavarone, and F Galeone
Subjects: Battery (electricity), Aging, medicine.medical_specialty, business.industry, General Neuroscience, medicine, Dementia, Neurology (clinical), Geriatrics and Gerontology, Audiology, medicine.disease, business, Developmental Biology
Published: 1992
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38. Rapport d'un travail d'expérimentation sur le contrôle de la mammite streptococcique par la vaccination curative et préventive et quelques mesures hygiéniques

Author: E. Brochu, M. Panisset, and M. Veilleux
Subjects: Food Science
Published: 1938
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39. STUDIES ON MYCOBACTERIA: I. CYTOCHEMICAL STUDY AND CHEMICAL ANALYSIS OF THE LIPIDS OF MYCOBACTERIUM MARIANUM

Author: V. Portelance, M. Panisset, and Soeur Marie Suzanne
Subjects: biology, Tubercle, Microorganism, Immunology, Genetics, Bacillus, General Medicine, biology.organism_classification, Molecular Biology, Applied Microbiology and Biotechnology, Microbiology, Mycobacterium marinum
Abstract: Several authors have reported that the composition of the lipids of the tubercle bacillus varies with the type and composition of the culture media. Major differences may therefore exist between the bacillary lipids of bacilli growing on culture media and those of such microorganisms parasitizing a host.The present paper deals with the cytochemical and chemical analyses of Mycobacterium marianum, which is, to our knowledge, the first strain of Mycobacterium to be stained by the Sudan black B method when grown on Sauton's medium. Details on the isolation and fractionation of several lipid fractions of Mycobacterium marianum are presented together with some of their properties. The results are discussed in relation to our working hypothesis that the chemical composition of Mycobacterium marianum grown under our conditions may resemble more closely that of bacilli growing in vivo than do most cultured mycobacteria.
Published: 1956
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40. [Attempted protection of mice with micro-doses of tubercle bacilli]

Author: R, Charbonneau, J C, Benoit, M, Panisset, and D, Mayrand
Subjects: Mice, Time Factors, Vaccination, BCG Vaccine, Immunization, Secondary, Animals, Tuberculosis, Mycobacterium tuberculosis, Mycobacterium
Published: 1974

41. [QUANTITATIVE STUDY OF THE VIABILITY OF BACTERIA BEFORE AND AFTER LYOPHILIZATION. PRESENTATION OF A METHOD OF ESTIMATION]

Author: M, QUEVILLON, J C, BENOIT, and M, PANISSET
Subjects: Bacteriological Techniques, Freeze Drying, Bacteria, Research, Mycobacterium bovis
Published: 1963

42. [Investigations on the mutagenic and tuberculostatic effect of certain analogs of vitamin K]

Author: M, PANISSET, H, SIMONNET, and M, DOBIJA
Subjects: Retinoids, Vitamin K, Bacteria, Heparin Antagonists, Mycobacterium tuberculosis, Antifibrinolytic Agents, Naphthoquinones
Published: 1952

43. [Intracerebral inoculation of Micrococcus in mice injected with penicillin: a possible method for the in vivo evaluation of antibiotics]

Author: M, PANISSET and M, DOBIJA
Subjects: Mice, Vaccination, Animals, Dermatologic Agents, Penicillins, Antibiotics, Antitubercular, Anti-Bacterial Agents, Micrococcus
Published: 1953

44. [Response of the epiploon in guinea pig to intraperitoneal BCG injection; quantitative interpretation of results]

Author: M, PANISSET and P, MAROIS
Subjects: Guinea Pigs, Vaccination, BCG Vaccine, Mycobacterium bovis, Omentum, Injections, Intraperitoneal
Published: 1953

45. [Enumeration of viable bacteria in suspensions of BCG vaccine]

Author: M, PANISSET and B, MARCIL
Subjects: Bacteria, Suspensions, Vaccination, BCG Vaccine, Humans, Mycobacterium bovis
Published: 1953

46. [The mode of action of fatlike adjuvants in immunity; variations in blood cholesterol during immunization]

Author: M, PANISSET, J, LAPIERRE, and E, MANKIEVICZ
Subjects: Cholesterol, Immunity, Humans, Immunization, Antigens, Antibodies
Published: 1951

47. [Antigen of Mycobacterium tuberculosis and specificity]

Author: M M, PANISSET
Subjects: Humans, Tuberculosis, Mycobacterium tuberculosis, Antigens, Antibodies
Published: 1952

48. [SELECTIVE EFFECT OF LYOPHILIZATION ON A BACTERIAL POPULATION: SPECIAL CASE OF BCG]

Author: M, QUEVILLON, J C, BENOIT, and M, PANISSET
Subjects: Bacteriological Techniques, Mice, Surface-Active Agents, Freeze Drying, Research, Animals, Tetrazolium Salts, Mycobacterium bovis
Published: 1964

49. Studies on an antibiotic substance extracted from the roots of Celastrus scandens

Author: M, PANISSET and A, NANTEL
Subjects: Humans, Celastrus, Anti-Bacterial Agents
Published: 1948

50. La nouvelle réglementation Vétérinaire française

Author: M, Panisset
Subjects: Articles
Published: 1938

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