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1. Monoamine oxidase-A promotes protective autophagy in human SH-SY5Y neuroblastoma cells through Bcl-2 phosphorylation

2. Alcohol-related brain damage in humans.

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3. Pale body-like inclusion formation and neurodegeneration following depletion of 26S proteasomes in mouse brain neurones are independent of α-synuclein.

4. The Calcineurin-TFEB-p62 Pathway Mediates the Activation of Cardiac Macroautophagy by Proteasomal Malfunction

5. Does video feedback & peer observation offer a valid method of reinforcing oral presentation training for undergraduate biochemists?

6. Monoamine oxidase-A promotes protective autophagy in human SH-SY5Y neuroblastoma cells through Bcl-2 phosphorylation

7. The Calcineurin-TFEB-p62 Pathway Mediates the Activation of Cardiac Macroautophagy by Proteasomal Malfunction

8. Up, Up and Away! : Mattie's Magical Animal Dreamworld #1 Ed. 1

9. The Castle of Cupcakes : Mattie's Magical Animal Dreamworld #2 Ed. 1

10. Continued 26S proteasome dysfunction in mouse brain cortical neurons impairs autophagy and the Keap1-Nrf2 oxidative defence pathway

11. Heterozygosity for the proteasomal Psmc1 ATPase is insufficient to cause neuropathology in mouse brain, but causes cell cycle defects in mouse embryonic fibroblasts

12. CRMP2 Hyperphosphorylation is Characteristic of Alzheimer's Disease and not a Feature Common to Other Neurodegenerative Diseases

13. Protein Degradation in Neurodegeneration: The Ubiquitin Pathway

14. Immunoreactivity to Lys63-linked polyubiquitin is a feature of neurodegeneration

15. The UPS and autophagy in chronic neurodegenerative disease: Six of one and half a dozen of the other—Or not?

16. Is malfunction of the ubiquitin proteasome system the primary cause of α-synucleinopathies and other chronic human neurodegenerative disease?

17. Id4 is required for the correct timing of neural differentiation

18. Proteasome Dysfunction Activates Autophagy and the Keap1-Nrf2 Pathway*

19. Can neurodegeneration be separated from neuropathological hallmarks of chronic idiopathic human neurodegenerative disease? A perspective from modelling!

20. The wakefulness promoting agent modafinil ameliorates experimental autoimmune encephalomyelitis

21. Ubiquitin-like protein conjugation and the ubiquitin-proteasome system as drug targets

22. Diverse polyubiquitin chains accumulate following 26S proteasomal dysfunction in mammalian neurones

23. Review: the ubiquitin-proteasome system: contributions to cell death or survival in neurodegeneration

24. Depletion of 26S proteasomes in mouse brain neurons causes neurodegeneration and Lewy-like inclusions resembling human pale bodies

25. Contributor contact details

26. The ubiquitin-proteasome system and neurodegenerative disorders

27. Parkinson's Disease Society 6th Spring Conference for Researchers

28. Pale Body-Like Inclusion Formation and Neurodegeneration following Depletion of 26S Proteasomes in Mouse Brain Neurones are Independent of α-Synuclein

29. The composition of cherry tomatoes and its relation to consumer acceptability

30. Can neurodegeneration be separated from neuropathological hallmarks of chronic idiopathic human neurodegenerative disease? A perspective from modelling!

31. Implications for oxidative stress and astrocytes following 26S proteasomal depletion in mouse forebrain neurones

32. Proteasome Dysfunction Activates Autophagy and the Keap1-Nrf2 Pathway.