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2. Mendelian Susceptibility to Mycobacterial Disease: Retrospective Clinical and Genetic Study in Mexico

Author

Peñafiel Vicuña, Ana Karen, Yamazaki Nakashimada, Marco, León Lara, Ximena, Mendieta Flores, Elizabeth, Nuñez Núñez, María Enriqueta, Lona-Reyes, Juan Carlos, Hernández Nieto, Leticia, Ramírez Vázquez, María Guadalupe, Barroso Santos, Joel, López Iñiguez, Álvaro, González, Yolanda, Torres, Martha, Lezana Fernández, José Luis, Román Montes, Carla M., Medina-Torres, Edgar Alejandro, González Serrano, Edith, Bustamante Ogando, Juan Carlos, Lugo Reyes, Saúl, Zavaleta Martínez, Oscar, Staines Boone, Aidé Tamara, Venegas Montoya, Edna, Aguilar Gómez, Nancy Evelyn, Soudeé, Camille, Jouanguy, Emmanuelle, Puel, Anne, Boisson-Dupuis, Stéphanie, Pedraza Sánchez, Sigifredo, Casanova, Jean-Laurent, Espinosa Rosales, Francisco, Espinosa Padilla, Sara, Bustamante, Jacinta, and Blancas Galicia, Lizbeth

Published
2023
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3. Neumonía necrosante en un paciente con deficiencia selectiva de IgA.

Author

Núnez-Núñez, María Enriqueta, Monraz-Monteón, Denisse, Lona-Reyes, Juan Carlos, pozos-Ochoa, Luis Iván, Magallón-Picazo, Diego, and Bayardo-Gutierrez, Beatriz

Abstract

Copyright of Revista Alergia de Mexico is the property of Coleg. Mexicano de Inmunologia Clinica y Alergia A.C.; Soc. Lat. de Alergia, Asma e Inmunologia and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2024
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13. Mendelian Susceptibility to Mycobacterial Disease: Retrospective Clinical and Genetic Study in Mexico

Author

Peñafiel Vicuña, Ana Karen, primary, Yamazaki Nakashimada, Marco, additional, León Lara, Ximena, additional, Mendieta Flores, Elizabeth, additional, Nuñez Núñez, María Enriqueta, additional, Lona-Reyes, Juan Carlos, additional, Hernández Nieto, Leticia, additional, Ramírez Vázquez, María Guadalupe, additional, Barroso Santos, Joel, additional, López Iñiguez, Álvaro, additional, González, Yolanda, additional, Torres, Martha, additional, Lezana Fernández, José Luis, additional, Román Montes, Carla M., additional, Medina-Torres, Edgar Alejandro, additional, González Serrano, Edith, additional, Bustamante Ogando, Juan Carlos, additional, Lugo Reyes, Saúl, additional, Zavaleta Martínez, Oscar, additional, Staines Boone, Aidé Tamara, additional, Venegas Montoya, Edna, additional, Aguilar Gómez, Nancy Evelyn, additional, Soudeé, Camille, additional, Jouanguy, Emmanuelle, additional, Puel, Anne, additional, Boisson-Dupuis, Stéphanie, additional, Pedraza Sánchez, Sigifredo, additional, Casanova, Jean-Laurent, additional, Espinosa Rosales, Francisco, additional, Espinosa Padilla, Sara, additional, Bustamante, Jacinta, additional, and Blancas Galicia, Lizbeth, additional

Published
2022
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15. Mendelian Susceptibility to Mycobacterial Disease: Retrospective Clinical and Genetic Study in Mexico

Author

VICUÑA, ANA KAREN PEÑAFIEL, primary, Yamazaki-Nakashimada, MARCO, additional, León-Lara, Ximena, additional, FLORES, ELIZABETH MENDIETA, additional, NUÑEZ, MARÍA ENRIQUETA NUÑEZ, additional, LONA-REYES, JUAN CARLOS, additional, NIETO, LETICIA HERNADEZ, additional, VÀZQUEZ, MARÍA GUADALUPE RAMÍREZ, additional, SANTOS, JOEL BARROSO, additional, IÑIGUEZ, ALVARO LÓPEZ, additional, GONZÁLEZ, YOLANDA, additional, TORRES, MARTHA, additional, FERNANDEZ, JOSÉ LUIS LEZANA, additional, Román-Montes, CARLA M, additional, MEDINA-TORRES, EDGAR ALEJANDRO, additional, SERRANO, EDITH GONZÁLEZ, additional, OGANDO, JUAN CARLOS BUSTAMANTE, additional, REYES, SAUL LUGO, additional, MARTÍNEZ, OSCAR ZAVALETA, additional, BOONE, AIDÉ TAMARA SATINES, additional, MONTOYA, EDNA VENEGAS, additional, GOMEZ, NANCY EVELYN AGUILAR, additional, SOUDEÉ, CAMILLE, additional, JOUANGUY, EMMANUELLE, additional, PUEL, ANNE, additional, Boisson-Dupuis, Stéphanie, additional, SÁNCHEZ, SIFREDO PEDRAZA, additional, CASANOVA, JEAN LAURENT, additional, ROSALES, FRANCISCO ESPINOSA, additional, Padilla, SARA Espinosa-, additional, Bustamante, Jacinta, additional, and Blancas-Galicia, Lizbeth, additional

Published
2022
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19. Late treatment of paraquat poisoning in pediatric patients: Report of two cases

Author

Ramos-Gutiérrez, Ruth Yesica, Real-Ornelas, Gabriel Alejandro, González Ruiz, Héctor Andrés, Lona-Reyes, Juan Carlos, Acuña-Chávez, Nancy, and Barriga-Marín, Javier Alvaro

Subjects
Tratamiento inmunosupresor, Intoxicación con paraquat, Paraquat poisoning, Paciente pediátrico, México, Children, Immunosuppressive treatment
Abstract

El paraquat (PQ) pertenece al grupo de herbicidas de los bipiridilos. Su presentación es en forma líquida o en granulado, usándose con una concentración al 5 %, para uso en jardinería y al 20 % para uso agrícola. En la intoxicación en humanos el órgano blanco es el pulmón. Los pacientes desarrollan insuficiencia respiratoria que puede explicarse por una inicial actividad que involucra un gran estrés oxidativo, con presencia de radicales libres de oxígeno y peroxidación lipídica, con sus consecuentes daños, además de infiltración por polimorfonucleares que con su reacción de liberación empeoran la neumonitis. Puede haber mejoría de la neumonitis y el daño en algunos órganos, pero pronto la aparición de fibrosis pulmonar lleva a falta de respuesta a la administración de oxígeno y a la muerte por insuficiencia respiratoria en algunos días a semanas. De acuerdo con la cantidad ingerida varía la evolución de la severidad del cuadro clínico. Se presentan dos pacientes pediátricos con intoxicación por PQ, a quienes se les inició tratamiento inmunosupresor después de 48 horas de la exposición. Uno de los pacientes se intoxicó de manera no intencional y otro por suicidio. Los dos pacientes recibieron tratamiento similar, sin embargo, el paciente con intención suicida falleció días después de la exposición. Se hace una revisión de la literatura sobre el tratamiento administrado. Paraquat (PQ) belongs to the bipyridyls herbicides. Its presentation is liquid or granulated, being used at concentrations of 5 %, in gardening and 20 % in agricultural use. In human poisoning, the target organ is the lung. The patients develop respiratory insufficiency that can be explained by an initial activity that involves a great oxidative stress, with the presence of oxygen free radicals and lipid peroxidation, with its consequent damages, in addition to polymorphonuclear infiltration that with its liberation reaction worsen pneumonitis. There may be improvement of pneumonitis, but the appearance of pulmonary fibrosis will lead to a lack of response to the administration of oxygen and death due to respiratory failure in a few days to a few weeks. According to the amount ingested, the evolution of the severity of the clinical picture varies. We present two pediatric patients with PQ poisoning, who were started on immunosuppressant treatment after 48 hours of exposure. One of the patients was poisoned incidentally and the other one by suicide. The two patients received similar treatment, however, the patient with suicidal intention died days after the exposure. A review of the literature on the treatment offered is made.

Published
2019

26. Infección del torrente sanguíneo por Candida glabrataen una unidad de cuidados intensivos neonatales con prescripción de profilaxis con fluconazol

Author

Lona-Reyes, Juan Carlos, Gómez-Ruiz, Larissa María, Cordero-Zamora, Araceli, Chávez-Rodríguez, Mariana, Gallegos-Marín, Juan Antonio, and Jiménez-Hernández, Denisse Sinaí

Abstract

Candida glabrataes un patógeno emergente con capacidad de desarrollar tolerancia y resistencia a los antifúngicos azólicos, lo que genera incertidumbre sobre la utilidad de la profilaxis antifúngica en recién nacidos.

Published
2020
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29. Calidad de vida en niños con cáncer mediante PedsQL Cancer Module©.

Author

Ramírez-Zamora, Laura Miriam, Llamas-Peregrina, Norma Esthela, Lona-Reyes, Juan Carlos, and Sánchez-Zubieta, Fernando Antonio

Abstract

Copyright of Revista Mexicana de Pediatria is the property of Sociedad Mexicana de Pediatria and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts.)

Published
2015

30. [Association of comorbidities and symptomatic infection and multisystemic inflammatory syndrome due to SARS-CoV-2 in children].

Author

Lona-Reyes JC, Paredes-Casillas P, Sepúlveda-Marrón J, Barrón-Balderas A, Cruz-Revilla R, Meza-López C, Guerrero-Velasco JA, and Calderón-Escamilla JR

Subjects
Child, Child, Preschool, Cross-Sectional Studies, Hospitalization, Humans, Obesity, Syndrome, Systemic Inflammatory Response Syndrome epidemiology, COVID-19 complications, COVID-19 epidemiology, SARS-CoV-2
Abstract

Background: In children, infection by the new coronavirus (SARS-CoV-2) usually occurs asymptomatic or with mild clinical data, only a minor proportion have severe symptoms or a set of post-infectious signs and symptoms described as Pediatric Inflammatory Multisystemic Syndrome (PIMS)., Aim: To describe the association of comorbidities with symptomatic infection and PIMS due to SARS-CoV-2 in children., Methods: Analytical cross-sectional study, pediatric patients hospitalized were included. Active infection was diagnosed by polymerase chain reaction and/or antigenic tests. Patients with PIMS were identified by the definition proposed by the World Health Organization., Results: 375 patients were studied, the median age was 3.8 years. 47.7% (n: 179) had comorbidities, the most frequent were: solid neoplasms and/or hematological diseases 17.1% (n: 64), obesity 13.3% (n: 48) and chronic pneumopathies 9, 3% (n: 35). SARS-CoV-2 infection was present in 16.5% (n: 62/375) and PIMS in 10.4% (n. 39/375). Children with obesity showed a higher risk of infection (OR 2.21, 95% CI 1.05-4.6) and in those with cancer (OR 0.15, 95% CI 0.03-0.68) the PIMS risk was lower., Conclusions: The presence of comorbidities modifies the risk of infection by SARS-CoV-2 and PIMS.

Published
2021
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32. [Bloodstream infections with Candida glabrata in a neonatal intensive care unit that uses prophylaxis with fluconazole].

Author

Lona-Reyes JC, Gómez-Ruiz LM, Cordero-Zamora A, Chávez-Rodríguez M, Gallegos-Marín JA, and Jiménez-Hernández DS

Subjects
Antifungal Agents therapeutic use, Candida glabrata, Case-Control Studies, Fluconazole therapeutic use, Humans, Infant, Infant, Newborn, Intensive Care Units, Neonatal, Male, Microbial Sensitivity Tests, Candidiasis drug therapy, Candidiasis epidemiology, Candidiasis prevention & control, Sepsis drug therapy
Abstract

Background: Candida glabrata is an emerging pathogen with the ability to develop tolerance and resistance to azole antifungals, which creates uncertainty about the usefulness of antifungal prophylaxis in newborns., Aims: The aim of this study was to describe the factors associated with C. glabrata infection in a NICU that uses prophylaxis with fluconazole., Methods: A case-control study paired by gestational age was designed and conducted at the Civil Hospital of Guadalajara Dr. Juan I. Menchaca. Newborns with C. glabrata infection were studied and for each one a matched control was selected by gestational age. Odds ratios (OR) were estimated with 95% confidence intervals (95% CI) and McNemar test for contrast of hypothesis was applied., Results: Twenty-one infected patients were identified, from whom 66.7% were male; the median gestational age was 31.5 weeks. Increased risk of infection with C. glabrata was observed when there was a prescription of more than one antimicrobial scheme (OR 21, 95% CI, 1.23 - 358.3; p=0.006) and also among patients with surgical comorbidities (OR 8, 95% CI 1.01 - 63.9; p=0.04). During the study period, exposure to fluconazole showed no difference in the risk of infection., Conclusions: Neonates with more than one antimicrobial regimen and those with surgical comorbidities had a higher risk of C. glabrata infection., (Copyright © 2020 Asociación Española de Micología. Publicado por Elsevier España, S.L.U. All rights reserved.)

Published
2020
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33. [Prevalence of extended-spectrum beta-lactamases in enterobacteria of neonatal sepsis and associated factors].

Author

Lona-Reyes JC, Pérez-Ramírez RO, Rodríguez-Patiño V, Cordero-Zamora A, Gómez-Ruiz LM, and Llamas-Ramos L

Subjects
Adolescent, Adult, Child, Enterobacteriaceae classification, Female, Humans, Infant, Newborn, Intensive Care Units, Neonatal, Male, Microbial Sensitivity Tests, Middle Aged, Prevalence, Prospective Studies, Risk Factors, Young Adult, Anti-Bacterial Agents pharmacology, Cross Infection microbiology, Enterobacteriaceae drug effects, Enterobacteriaceae Infections microbiology, Neonatal Sepsis microbiology, beta-Lactamases biosynthesis
Abstract

Background: Infections caused by extended-spectrum beta-lactamases enterobacteria (ESBL-EP) have implications for neonatal morbidity and mortality., Aim: To describe the prevalence of ESBL-EP in neonatal sepsis and associated factors., Methods: A prospective cohort study was conducted from August 2016 to August 2017; newborn babies (NB) hospitalized in the Hospital Civil de Guadalajara "Dr. Juan I. Menchaca" were included. The ESBL-EP were investigated by double-disk synergy test and its association with clinical and demographic characteristics of the NB., Results: A total of 1,501 hospitalized NB were studied, with an average gestational age of 36.3 weeks. They were diagnosed 196 neonatal sepsis events, the most frequent etiologies were enterobacteria (45.5%). Resistance to ampicilin was found in 88.8% and to broad spectrum cephalosporins in more than 42% of the strains; 22.9% of them were ESBL phenotype. Apgar ≤ 7 at five minutes of life (OR 4.6; 95% CI 1.47-14.6) and gestational age < 37 weeks (OR 5.4; 95% CI 1.04-27.) increase the risk., Conclusion: In enterobacteria that cause neonatal sepsis, 22.9% were EP-ESBL; infection was more likely in patients with Apgar ≤ 7 at five minutes of age and in preterm infants.

Published
2019
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34. Etiology and antimicrobial resistance patterns in early and late neonatal sepsis in a Neonatal Intensive Care Unit.

Author

Lona Reyes JC, Verdugo Robles MÁ, Pérez Ramírez RO, Pérez Molina JJ, Ascencio Esparza EP, and Benítez Vázquez EA

Subjects
Age Factors, Anti-Bacterial Agents pharmacology, Cross-Sectional Studies, Drug Resistance, Bacterial, Enterobacteriaceae drug effects, Humans, Infant, Newborn, Intensive Care Units, Neonatal, Microbial Sensitivity Tests, Staphylococcus drug effects, Streptococcus drug effects, Anti-Bacterial Agents therapeutic use, Enterobacteriaceae Infections drug therapy, Neonatal Sepsis drug therapy, Neonatal Sepsis etiology, Staphylococcal Infections drug therapy, Streptococcal Infections drug therapy
Abstract

Introduction: Neonatal sepsis is one of the main causes of death among newborn infants. Empirical antimicrobial treatment is based on epidemiological information and antimicrobial susceptibility tests. The objective of this study was to describe etiologic agents and their antimicrobial susceptibility among newborn infants with early-onset neonatal sepsis (EONS) or late-onset neonatal sepsis (LONS) at a Neonatal Intensive Care Unit., Methods: Cross-sectional study conducted at a tertiary referral hospital in Western Mexico. Determination of antimicrobial resistance of microorganisms isolated in blood or cerebrospinal fluid of patients with EONS or nosocomial LONS., Results: Yeasts and bacteria were isolated from 235 cultures corresponding to 67 events of EONS and 166 events of LONS. Of all isolates, the most common bacteria were Enterobacteriaceae (51.5%), followed by Streptococcus spp. in EONS, and by Staphylococcus spp. in LONS. Of all nosocomial Enterobacteriaceae, 40% were extended spectrum beta-lactamase producing bacteria. Among Staphylococcus species, resistance to oxacillin was recorded in 65.5%. Among Enterobacteriaceae (n: 121), resistance to amikacin, piperacillin-tazobactam, and meropenem was below 3%. Non-fermenting bacteria did not show resistance to amikacin, ciprofloxacin or cefepime; however, the number of isolates was scarce., Conclusions: The most commonly identified bacteria in EONS were Enterobacteriaceae (67.6%) and Streptococcus spp. (17.6%), and Enterobacteriaceae (44.9%) and Staphylococcus spp. (34.7%) in LONS. Forty percent ofnosocomial Enterobacteriaceae were extended spectrum beta-lactamase producing bacteria, and 65.5% of Staphylococcus spp. showed resistance to oxacillin.

Published
2015
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