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2. RAS-mutant leukaemia stem cells drive clinical resistance to venetoclax

3. Concurrent inhibition of oncogenic and wild-type RAS-GTP for cancer therapy

4. In vivo vulnerabilities to GPX4 and HDAC inhibitors in drug-persistent versus drug-resistant BRAFV600E lung adenocarcinoma

5. Pan-KRAS inhibitor disables oncogenic signalling and tumour growth

6. KRASG12C Inhibition with Sotorasib in Advanced Solid Tumors

7. Overall survival with circulating tumor DNA-guided therapy in advanced non-small-cell lung cancer

8. Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation.

10. Immune biomarkers and response to checkpoint inhibition of BRAFV600 and BRAF non-V600 altered lung cancers

14. Long-term benefit of sotorasib in patients with KRAS G12C–mutated non–small-cell lung cancer: plain language summary

15. Modeling lung adenocarcinoma metastases using patient-derived organoids

17. Chemical remodeling of a cellular chaperone to target the active state of mutant KRAS

18. Outcomes of Combination Platinum-Doublet Chemotherapy and Anti-PD(L)-1 Blockade in KRASG12C-Mutant Non-Small Cell Lung Cancer

19. Long-Term Outcomes and Molecular Correlates of Sotorasib Efficacy in Patients With Pretreated KRAS G12C-Mutated Non–Small-Cell Lung Cancer: 2-Year Analysis of CodeBreaK 100

22. Outcomes of Combination Platinum-Doublet Chemotherapy and Anti-PD(L)-1 Blockade in KRASG12C-Mutant Non-Small Cell Lung Cancer.

23. Supplementary Data from The KRASG12C Inhibitor MRTX849 Provides Insight toward Therapeutic Susceptibility of KRAS-Mutant Cancers in Mouse Models and Patients

24. Data from The KRASG12C Inhibitor MRTX849 Provides Insight toward Therapeutic Susceptibility of KRAS-Mutant Cancers in Mouse Models and Patients

25. Supplementary Figures from KRASG12C Mutation Is Associated with Increased Risk of Recurrence in Surgically Resected Lung Adenocarcinoma

26. Supplementary Data Table 2 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

27. Supplementary Tables from KRASG12C Mutation Is Associated with Increased Risk of Recurrence in Surgically Resected Lung Adenocarcinoma

28. Supplemental Tables 1-4, Supplemental Figures 1-2 from Effects of Co-occurring Genomic Alterations on Outcomes in Patients with KRAS-Mutant Non–Small Cell Lung Cancer

29. Supplementary Data Table 4 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

30. Supplementary Data Table 1 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

31. Supplementary Data Table 5 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

32. Supplementary Figure Legends from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

33. Supplementary Data S1-S3 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

34. Supplementary Data Table 3 from Adaptive Resistance to Dual BRAF/MEK Inhibition in BRAF-Driven Tumors through Autocrine FGFR Pathway Activation

37. Insights into how adeno-squamous transition drives KRAS inhibitor resistance.

40. Clinicopathologic characterization of ERK2 E322K mutation in solid tumors: Implications for treatment and drug development.

42. With one eye on the future

44. Immune biomarkers and response to checkpoint inhibition of BRAFV600 and BRAF non-V600 altered lung cancers

45. Diverse alterations associated with resistance to KRAS(G12C) inhibition

48. Tumor adaptation and resistance to RAF inhibitors

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