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3. Cell-autonomous effects of APOE4 in restricting microglial response in brain homeostasis and Alzheimer’s disease

4. Correction: Trem2 H157Y increases soluble TREM2 production and reduces amyloid pathology

5. Peripheral apoE4 enhances Alzheimer’s pathology and impairs cognition by compromising cerebrovascular function

6. C9ORF72 poly(GA) aggregates sequester and impair HR23 and nucleocytoplasmic transport proteins

7. Heterochromatin anomalies and double-stranded RNA accumulation underlie C9orf72 poly(PR) toxicity

9. Investigating the Pathogenic Interplay of Alpha-Synuclein, Tau, and Amyloid Beta in Lewy Body Dementia: Insights from Viral-Mediated Overexpression in Transgenic Mouse Models

10. Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation

13. Poly(GR) impairs protein translation and stress granule dynamics in C9orf72-associated frontotemporal dementia and amyotrophic lateral sclerosis

15. LRP1 in vascular mural cells modulates cerebrovascular integrity and function in the presence of APOE4

16. C90RF72 repeat expansions in mice cause TDP-43 pathology, neuronal loss, and behavioral deficits

17. APOE genotype modifies benefits of calorie restriction against aging.

18. APOE2 Exacerbates TDP ‐43 Related Toxicity in the Absence of Alzheimer Pathology

21. APOE ε2 is associated with increased tau pathology in primary tauopathy

22. Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of C9ORF72-repeat induced toxicity

23. Two FTD-ALS genes converge on the endosomal pathway to induce TDP-43 pathology and degeneration

24. Cell-autonomous effects of APOE4in restricting microglial response in brain homeostasis and Alzheimer’s disease

25. Plasma PolyQ-ATXN3 Levels Associate With Cerebellar Degeneration and Behavioral Abnormalities in a New AAV-Based SCA3 Mouse Model

26. APOE3-Jacksonville (V236E) variant reduces self-aggregation and risk of dementia: Implications for therapeutic strategy

29. APOE3 -Jacksonville (V236E) variant reduces self-aggregation and risk of dementia

30. Hexanucleotide Repeat Expansions in c9FTD/ALS and SCA36 Confer Selective Patterns of Neurodegeneration In Vivo

31. Two FTD-ALS genes converge on the endosomal pathway to induce TDP-43 pathology and degeneration.

32. Loss of Tmem106b leads to cerebellum Purkinje cell death and motor deficits

33. Loss of Tmem106b leads to cerebellum Purkinje cell death and motor deficits

34. APOE2 is associated with longevity independent of Alzheimer’s disease

35. Author response: APOE2 is associated with longevity independent of Alzheimer’s disease

36. APOE2promotes longevity independent of Alzheimer’s disease

37. Additional file 5 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

38. Additional file 6 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

39. Additional file 1 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

40. Additional file 2 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

41. Additional file 7 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

42. Additional file 3 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

43. Additional file 1 of Clusterin ameliorates tau pathology in vivo by inhibiting fibril formation

44. Additional file 4 of Astrocyte-derived clusterin suppresses amyloid formation in vivo

45. APOE2 is associated with longevity independent of Alzheimer’s disease

46. Additional file 1: of Enhanced phosphorylation of T153 in soluble tau is a defining biochemical feature of the A152T tau risk variant

47. APOE4 exacerbates α-synuclein pathology and related toxicity independent of amyloid

48. TIA1 Mutations in Amyotrophic Lateral Sclerosis and Frontotemporal Dementia Promote Phase Separation and Alter Stress Granule Dynamics

49. Additional file 1: of Loss of Tmem106b is unable to ameliorate frontotemporal dementia-like phenotypes in an AAV mouse model of C9ORF72-repeat induced toxicity

50. Vascular apoE4 Impairs Brain Cognition by Modulating Glio-Vascular Functions

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