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1. Protein network analysis reveals selectively vulnerable regions and biological processes in FTD

2. Author Correction: Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores (Nature Communications, (2021), 12, 1, (3417), 10.1038/s41467-021-22491-8)

3. New insights into the genetic etiology of Alzheimer's disease and related dementias

4. Data-driven staging of genetic frontotemporal dementia using multi-modal MRI

5. Genetic architecture of sporadic frontotemporal dementia and overlap with Alzheimerʼs and Parkinsonʼs diseases

7. New insights into the genetic etiology of Alzheimer's disease and related dementias

9. Practice effects in genetic frontotemporal dementia and at-risk individuals: a GENFI study

11. A C6orf10/LOC101929163 locus is associated with age of onset in C9orf72 carriers

15. Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores

16. Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores

17. Progression of Behavioral Disturbances and Neuropsychiatric Symptoms in Patients With Genetic Frontotemporal Dementia

18. Common variants in Alzheimer's disease and risk stratification by polygenic risk scores

19. When do the expectations of others matter? Experimental evidence on distributional justice and guilt aversion

20. Common variants in Alzheimer's disease and risk stratification by polygenic risk scores

21. Progression of Behavioral Disturbances and Neuropsychiatric Symptoms in Patients With Genetic Frontotemporal Dementia

23. Sphingolipid synthesis inhibition by myriocin administration enhances lipid consumption and ameliorates lipid response to myocardial ischemia reperfusion injury

25. Correction to:A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity (Acta Neuropathologica, (2019), 138, 2, (237-250), 10.1007/s00401-019-02026-8)

26. Correction to: A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity (Acta Neuropathologica, (2019), 138, 2, (237-250), 10.1007/s00401-019-02026-8)

27. Analysis of brain atrophy and local gene expression in genetic frontotemporal dementia

28. Age at symptom onset and death and disease duration in genetic frontotemporal dementia: an international retrospective cohort study

30. Apathy in presymptomatic genetic frontotemporal dementia predicts cognitive decline and is driven by structural brain changes

31. Mendelian randomization implies no direct causal association between leukocyte telomere length and amyotrophic lateral sclerosis

32. Age at symptom onset and death and disease duration in genetic frontotemporal dementia: an international retrospective cohort study

33. Solar geoengineering may lead to excessive cooling and high strategic uncertainty

34. Correction to: A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity

36. Frontotemporal dementia: impact of P301L tau mutation on a healthy carrier

40. Cognitive reserve and TMEM106B genotype modulate brain damage in presymptomatic frontotemporal dementia: a GENFI study

42. Cerebral perfusion changes in presymptomatic genetic frontotemporal dementia: a GENFI study

43. Genetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasia

45. Identification of evolutionarily conserved gene networks mediating neurodegenerative dementia

46. Clinical value of cerebrospinal fluid neurofilament light chain in semantic dementia

47. Novel CSF biomarkers in genetic frontotemporal dementia identified by proteomics

48. Genetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasia

49. Spatiotemporal analysis for detection of pre-symptomatic shape changes in neurodegenerative diseases: Initial application to the GENFI cohort

50. Perfect and imperfect strangers in social dilemmas

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