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1. CRISPR-based gene editing enables FOXP3 gene repair in IPEX patient cells

2. Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19

3. Mapping the human genetic architecture of COVID-19

4. Prevalence and clinical challenges among adults with primary immunodeficiency and recombination-activating gene deficiency

6. Artificial Intelligence and Machine Learning for Inborn Errors of Immunity: Current State & Future Promise.

7. Enhancing tumor-infiltrating T cells with an exclusive fuel source.

8. Electronic health record signatures identify undiagnosed patients with common variable immunodeficiency disease.

10. APDS patients with immune-complex vasculitis and resolution with leniolisib.

11. Harnessing Biomaterials to Amplify Immunity in Aged Mice through T Memory Stem Cells.

12. Proceedings from the inaugural Artificial Intelligence in Primary Immune Deficiencies (AIPID) conference.

13. Correction: Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19.

14. Whole blood transcriptomics identifies subclasses of pediatric septic shock.

15. A Clinicopathological Categorization System for Clinical Research in Coccidioidomycosis.

16. Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19.

17. Tuning immunity through tissue mechanotransduction.

18. Systemic enhancement of antitumour immunity by peritumourally implanted immunomodulatory macroporous scaffolds.

20. Exome-wide association study to identify rare variants influencing COVID-19 outcomes: Results from the Host Genetics Initiative.

21. Rare predicted loss-of-function variants of type I IFN immunity genes are associated with life-threatening COVID-19.

22. Editorial: The cytoskeleton in T cell migration and activation.

23. Leveraging genomic diversity for discovery in an electronic health record linked biobank: the UCLA ATLAS Community Health Initiative.

24. Partial RAG deficiency in humans induces dysregulated peripheral lymphocyte development and humoral tolerance defect with accumulation of T-bet + B cells.

25. IL-10 promotes endothelial progenitor cell infiltration and wound healing via STAT3.

27. Immunomodulatory Microneedle Patch for Periodontal Tissue Regeneration.

29. Biallelic PI4KA variants cause neurological, intestinal and immunological disease.

31. Expanding the potential genes of inborn errors of immunity through protein interactions.

32. Massively scaled-up testing for SARS-CoV-2 RNA via next-generation sequencing of pooled and barcoded nasal and saliva samples.

33. Pre-existing conditions in Hispanics/Latinxs that are COVID-19 risk factors.

34. Swab-Seq: A high-throughput platform for massively scaled up SARS-CoV-2 testing.

36. Correction to: Infections in Infants with SCID: Isolation, Infection Screening and Prophylaxis in PIDTC Centers.

37. When Screening for Severe Combined Immunodeficiency (SCID) with T Cell Receptor Excision Circles Is Not SCID: a Case-Based Review.

38. The New "Wholly Trinity" in the Diagnosis and Management of Inborn Errors of Immunity.

39. Infections in Infants with SCID: Isolation, Infection Screening, and Prophylaxis in PIDTC Centers.

40. CADINS in an Adult with Chronic Sinusitis and Atopic Disease.

41. Multiplexed Functional Assessment of Genetic Variants in CARD11.

42. Unraveling the mechanobiology of immune cells.

43. MPEG1/Perforin-2 Haploinsufficiency Associated Polymicrobial Skin Infections and Considerations for Interferon-γ Therapy.

44. Augmenting T-cell responses to tumors by in situ nanomanufacturing.

45. Progressive B Cell Loss in Revertant X-SCID.

46. T-cell activation is modulated by the 3D mechanical microenvironment.

47. Mechanosensing through YAP controls T cell activation and metabolism.

48. Prior diagnoses and medications as risk factors for COVID-19 in a Los Angeles Health System.

49. Disseminated Coccidioidomycosis Treated with Interferon-γ and Dupilumab.

50. Correction: Dominant-negative mutations in human IL6ST underlie hyper-IgE syndrome.

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