159 results on '"Bretteville, Alexis"'
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2. IP-LC-MSMS Enables Identification of Three Tau O-GlcNAcylation Sites as O-GlcNAcase Inhibition Pharmacodynamic Readout in Transgenic Mice Overexpressing Human Tau
3. Dexmedetomidine increases tau phosphorylation under normothermic conditions in vivo and in vitro
4. Systemic immune‐checkpoint blockade with anti‐PD1 antibodies does not alter cerebral amyloid‐β burden in several amyloid transgenic mouse models
5. Anesthesia and tau pathology
6. Tau pathology modulates Pin1 post-translational modifications and may be relevant as biomarker
7. Proximity Ligation Assay: A Tool to Study Endogenous Interactions Between Tau and Its Neuronal Partners
8. A transgenic rat that develops Alzheimer's disease-like amyloid pathology, deficits in synaptic plasticity and cognitive impairment
9. Early Axonopathy Preceding Neurofibrillary Tangles in Mutant Tau Transgenic Mice
10. Biochemical Isolation of Insoluble Tau in Transgenic Mouse Models of Tauopathies
11. Deregulation of Protein Phosphatase 2A and Hyperphosphorylation of τ Protein Following Onset of Diabetes in NOD Mice
12. Pin1 allows for differential Tau dephosphorylation in neuronal cells
13. Diazaspirononane Nonsaccharide Inhibitors of O-GlcNAcase (OGA) for the Treatment of Neurodegenerative Disorders
14. Biochemistry of Tau in Alzheimerʼs disease and related neurological disorders
15. Additional file 1: of Tau phosphorylation regulates the interaction between BIN1â s SH3 domain and Tauâ s proline-rich domain
16. Additional file 10: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
17. Additional file 5: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
18. Additional file 8: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
19. Additional file 9: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
20. Additional file 7: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
21. Additional file 4: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
22. of Tau phosphorylation regulates the interaction between BIN1â s SH3 domain and Tauâ s proline-rich domain
23. Additional file 2: of Tau phosphorylation regulates the interaction between BIN1â s SH3 domain and Tauâ s proline-rich domain
24. Additional file 6: of Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
25. Genetically Engineered iPSC-Derived FTDP-17 MAPT Neurons Display Mutation-Specific Neurodegenerative and Neurodevelopmental Phenotypes
26. Systemic immune-checkpoint blockade with anti-PD1 antibodies does not alter cerebral amyloid-β burden in several amyloid transgenic mouse models
27. Tau phosphorylation regulates the interaction between BIN1’s SH3 domain and Tau’s proline-rich domain
28. P4-032: MOLECULAR CHARACTERISATION OF BRIDGING INTEGRATOR 1 (BIN1) INTERACTION WITH TAU
29. Specificity of Anti-Tau Antibodies when Analyzing Mice Models of Alzheimer's Disease: Problems and Solutions
30. Two-dimensional electrophoresis of tau mutants reveals specific phosphorylation pattern likely linked to early tau conformational changes.
31. Insulin dysfunction and Tau pathology
32. Neurogenesis and cell cycle-reactivated neuronal death during pathogenic tau aggregation.
33. Subacute per OS lithium treatment decreases Tau phosphorylation and neurofibrillary tangles load in G272V/P301S mutant Tau transgenic mice
34. Anesthesia-induced hypothermia mediates decreased ARC gene and protein expression through ERK/MAPK inactivation
35. Distinct phosphorylation pattern of Peptidyl prolyl isomerase Pin1 during neurofibrillary degeneration in Alzheimer and Tau transgenic mouse brains.
36. Alzheimer's disease-like tau neuropathology leads to memory deficits and loss of functional synapses in a novel mutated tau transgenic mouse without any motor deficits.
37. p25/Cdk5-mediated retinoblastoma phosphorylation is an early event in neuronal cell death
38. P3-001: Hypothermia-induced hyperphosphorylation: A new model to study tau kinase inhibitors
39. Dimethyl Sulfoxide Induces Both Direct and Indirect Tau Hyperphosphorylation
40. Hypothermia-induced hyperphosphorylation: a new model to study tau kinase inhibitors
41. Two-Dimensional Electrophoresis of Tau Mutants Reveals Specific Phosphorylation Pattern Likely Linked to Early Tau Conformational Changes
42. Acceleration and persistence of neurofibrillary pathology in a mouse model of tauopathy following anesthesia
43. Tau Aggregates: Toxic, Inert, or Protective Species?
44. O4-01-07: Subacute per OS lithium treatment decreases Tau phosphorylation and neurofibrillary tangles load in G272V/P301S mutant Tau transgenic mice
45. Alzheimer's Disease-Like Tau Neuropathology Leads to Memory Deficits and Loss of Functional Synapses in a Novel Mutated Tau Transgenic Mouse without Any Motor Deficits
46. P1-049: Alzheimer-type neurofibrillary lesions and neurofilamentous inclusions are cell-type specific in transgenic mice expressing mutated tau proteins G272V/P301S
47. S3-03-06: Tau phosphorylation, tauopathies, mitosis-related disease
48. P1-062: Alzheimer's disease-like tau neuropathology leads to memory deficits and loss of functional synapses in a novel mutated tau transgenic mouse without any motor deficits
49. p25/Cdk5-mediated retinoblastoma phosphorylation is an early event in neuronal cell death
50. MOLECULAR CHARACTERISATION OF BRIDGING INTEGRATOR 1 (BIN1) INTERACTION WITH TAU
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