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167. Targeted deletion of one or two copies of the G protein β subunit Gβ5 gene has distinct effects on body weight and behavior in mice.

168. Altered expression and subcellular distribution of GRK subtypes in the dopamine-depleted rat basal ganglia is not normalized byl-DOPA treatment.

169. Cone arrestin binding to JNK3 and Mdm2: conformational preference and localization of interaction sites.

170. Dopamine depletion and subsequent treatment withl-DOPA, but not the long-lived dopamine agonist pergolide, enhances activity of the Akt pathway in the rat striatum.

171. Each rhodopsin molecule binds its own arrestin.

172. Arrestin2.

176. Cognitive effects of dopamine depletion in the context of diminished acetylcholine signaling capacity in mice

179. Designer adhesion GPCR tells its signaling story

180. Dopamine D3receptor is selectively and transiently expressed in the developing whisker barrel cortex of the rat

181. Developmental Regulation of Expression of the D3Dopamine Receptor in Rat Nucleus Accumbens and Islands of Calleja1

182. D3Receptors and the Actions of Neuroleptics in the Ventral Striatopallidal System of Schizophrenics

183. GRKs as Modulators of Neurotransmitter Receptors.

184. Distribution of Dopamine D3Receptor Expressing Neurons in the Human Forebrain: Comparison with D2Receptor Expressing Neurons

185. Comparison of [3H]Paroxetine and [3H]Cyanoimipramine for Quantitative Measurement of Serotonin Transporter Sites in Human Brain

186. Structural basis of GPCR coupling to distinct signal transducers: implications for biased signaling.

187. Arrestin‐3 binds parkin and enhances parkin‐dependent mitophagy.

188. GPCR Binding and JNK3 Activation by Arrestin-3 Have Different Structural Requirements.

191. The Role of Arrestin-1 Middle Loop in Rhodopsin Binding.

192. Short Arrestin-3-Derived Peptides Activate JNK3 in Cells.

193. Biological Role of Arrestin-1 Oligomerization.

194. Mdm2 enhances ligase activity of parkin and facilitates mitophagy.

195. Cleavage of arrestin-3 by caspases attenuates cell death by precluding arrestin-dependent JNK activation.

196. Non-visual arrestins regulate the focal adhesion formation via small GTPases RhoA and Rac1 independently of GPCRs.

197. Arrestin-2 and arrestin-3 differentially modulate locomotor responses and sensitization to amphetamine.

198. G Protein-coupled Receptor Kinases of the GRK4 Protein Subfamily Phosphorylate Inactive G Protein-coupled Receptors (GPCRs).

199. Overexpression of GRK6 rescues l-DOPA-induced signaling abnormalities in the dopamine-depleted striatum of hemiparkinsonian rats.

200. Arrestin-3 Binds c-Jun N-terminal Kinase 1 (JNK1) and JNK2 and Facilitates the Activation of These Ubiquitous JNK Isoforms in Cells via Scaffolding.

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