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55. Correction of bleeding in experimental severe hemophilia A by systemic delivery of factor VIII-encoding mRNA

59. The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis

62. Anti-inflammatory Activity of the Protein Z-Dependent Protease Inhibitor

66. The Proteolytic Inactivation of Protein Z-Dependent Protease Inhibitor by Neutrophil Elastase Might Promote the Procoagulant Activity of Neutrophil Extracellular Traps in Sepsis.

67. The VWF/LRP4/αVβ3-axis represents a novel pathway regulating proliferation of human vascular smooth muscle cells

68. Development of a dual hybrid AAV vector for endothelial-targeted expression of von Willebrand factor

69. A compact integrated microfluidic oxygenator with high gas exchange efficiency and compatibility for long-lasting endothelialization

72. Gain-of-Function Variant p.Pro2555Arg of von Willebrand Factor Increases Aggregate Size through Altering Stem Dynamics

74. A Combination of Two Variants p. (Val510 =) and p. (Pro2145Thrfs * 5), Responsible for von Willebrand Disease Type 3 in a Caribbean Patient

76. VWF/LRP4/αVβ3-axis represents a novel pathway regulating proliferation of human vascular smooth muscle cells.

80. A Thrombin-Activatable Factor X Variant Corrects Hemostasis in a Mouse Model for Hemophilia A

81. Relevance of platelet desialylation and thrombocytopenia in type 2B von Willebrand disease: preclinical and clinical evidence

82. A mutation in the gene coding for the sialic acid transporter SLC35A1 is required for platelet life span but not proplatelet formation

90. Aspects fonctionnels et cliniques de l'emicizumab, un anticorps bispécifique utilisé dans le traitement de l'hémophilie A.

91. Thrombin generation on vascular cells in the presence of factor VIII and/or emicizumab

92. Von Willebrand factor: how unique structural adaptations support and coordinate its complex function

93. Removal of Mannose-Ending Glycan at Asn2118 Abrogates FVIII Presentation by Human Monocyte-Derived Dendritic Cells.

94. A mutation in the gene coding for the sialic acid transporter SLC35A1 is required for platelet life span but not proplatelet formation

95. Protein kinase C signaling dysfunction in von Willebrand disease (p.V1316M) type 2B platelets

97. The contribution of platelet glycoprotein receptors to inflammatory bleeding prevention is stimulus and organ dependent

100. Soluble Siglec-5 associates to PSGL-1 and displays anti-inflammatory activity

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