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1. Sall genes regulate hindlimb initiation in mouse embryos.

2. Sall4 regulates posterior trunk mesoderm development by promoting mesodermal gene expression and repressing neural genes in the mesoderm.

3. The zinc finger transcription factor Sall1 is required for the early developmental transition of microglia in mouse embryos.

4. Development of the Proximal-Anterior Skeletal Elements in the Mouse Hindlimb Is Regulated by a Transcriptional and Signaling Network Controlled by Sall4 .

5. Sall4 regulates neuromesodermal progenitors and their descendants during body elongation in mouse embryos.

6. Anephrogenic phenotype induced by SALL1 gene knockout in pigs.

7. Sall1 Regulates Microglial Morphology Cell Autonomously in the Developing Retina.

8. Re-expression of Sall1 in podocytes protects against adriamycin-induced nephrosis.

9. Cooperative Action between SALL4A and TET Proteins in Stepwise Oxidation of 5-Methylcytosine.

10. Sall1 is a transcriptional regulator defining microglia identity and function.

11. Sall4 controls differentiation of pluripotent cells independently of the Nucleosome Remodelling and Deacetylation (NuRD) complex.

12. Sall1 transiently marks undifferentiated heart precursors and regulates their fate.

13. A mouse model of Townes-Brocks syndrome expressing a truncated mutant Sall1 protein is protected from acute kidney injury.

14. Sall1 in renal stromal progenitors non-cell autonomously restricts the excessive expansion of nephron progenitors.

15. Preferential Propagation of Competent SIX2+ Nephronic Progenitors by LIF/ROCKi Treatment of the Metanephric Mesenchyme.

16. Sall4-Gli3 system in early limb progenitors is essential for the development of limb skeletal elements.

17. Sall4 is essential for mouse primordial germ cell specification by suppressing somatic cell program genes.

18. Sall1 maintains nephron progenitors and nascent nephrons by acting as both an activator and a repressor.

19. Preformed Wolffian duct regulates Müllerian duct elongation independently of canonical Wnt signaling or Lhx1 expression.

20. Histone lysine-specific demethylase 1 (LSD1) protein is involved in Sal-like protein 4 (SALL4)-mediated transcriptional repression in hematopoietic stem cells.

21. Islet1 deletion causes kidney agenesis and hydroureter resembling CAKUT.

22. Sall4 Is Transiently Expressed in the Caudal Wolffian Duct and the Ureteric Bud, but Dispensable for Kidney Development.

23. Sall1 regulates cortical neurogenesis and laminar fate specification in mice: implications for neural abnormalities in Townes-Brocks syndrome.

24. Functional antagonism between Sall4 and Plzf defines germline progenitors.

25. Notch2 activation in the embryonic kidney depletes nephron progenitors.

26. A mouse line expressing Sall1-driven inducible Cre recombinase in the kidney mesenchyme.

27. Overexpression of Sall1 in vivo leads to reduced body weight without affecting kidney development.

28. Sall4 is essential for stabilization, but not for pluripotency, of embryonic stem cells by repressing aberrant trophectoderm gene expression.

29. Sall4 regulates cell fate decision in fetal hepatic stem/progenitor cells.

30. Sall genes regulate region-specific morphogenesis in the mouse limb by modulating Hox activities.

31. Sall1 regulates mitral cell development and olfactory nerve extension in the developing olfactory bulb.

32. Mouse homolog of SALL1, a causative gene for Townes-Brocks syndrome, binds to A/T-rich sequences in pericentric heterochromatin via its C-terminal zinc finger domains.

33. The murine homolog of SALL4, a causative gene in Okihiro syndrome, is essential for embryonic stem cell proliferation, and cooperates with Sall1 in anorectal, heart, brain and kidney development.

34. Essential roles of Sall family genes in kidney development.

35. Essential roles of Sall1 in kidney development.

36. [Renal development and its molecular mechanism].

37. Sall1, a causative gene for Townes-Brocks syndrome, enhances the canonical Wnt signaling by localizing to heterochromatin.

38. Identification of kidney mesenchymal genes by a combination of microarray analysis and Sall1-GFP knockin mice.

39. Kidney development conserved over species: essential roles of Sall1.

40. Zinc finger protein sall2 is not essential for embryonic and kidney development.

41. Sall4 controls differentiation of pluripotent cells independently of the Nucleosome Remodelling and Deacetylation (NuRD) complex

42. Sall4 controls differentiation of pluripotent cells independently of the Nucleosome Remodelling and Deacetylation (NuRD) complex

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