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1. Shiga Toxin–Producing Escherichia coli Infections Associated with Hemolytic Uremic Syndrome, Italy, 1988–2000

Author

Tozzi, A. E., Caprioli, A., Minelli, F., Gianviti, A., Petris, L., Edefonti, A., Giovanni Montini, Ferretti, A., Palo, T., Gaido, M., Rizzoni, G., Bettinelli, A., Capasso, G., Caringella, A., Coppo, R., Lama, G., Li Volti, S., Maffei, S., Maringhini, S., Miglietti, N., Pecoraro, C., Pela, I., Pennesi, M., Penza, R., Peratoner, L., Perfumo, F., Ratsche, I., Salvaggio, E., Setzu, C., Zacchello, G., Tozzi, Ae, Caprioli, A, Minelli, F, Gianviti, A, De Petris, L, Edefonti, A, Montini, G, Ferretti, A, De Palo, T, Gaido, M, Rizzoni, G, A., Bettinelli, Capasso, Giovambattista, Caringella, A, Coppo, R, Lama, G, Li Volti, S, Maffei, S, Maringhini, S, Miglietti, N, Pecoraro, C, Pela, I, Pennesi, M, Penza, R, Peratoner, L, Perfumo, F, Ratsche, I, Salvaggio, E, Setzu, C, and Zacchello, G.

Subjects
Microbiology (medical), Serotype, Adolescent, Population, lcsh:Medicine, Escherichia coli O157, medicine.disease_cause, Annual incidence, Shiga Toxin, Microbiology, lcsh:Infectious and parasitic diseases, fluids and secretions, Shiga toxin-producing Escherichia coli, Escherichia coli, Humans, Medicine, HUS, lcsh:RC109-216, Serotyping, education, Escherichia coli Infections, laboratory diagnosis, education.field_of_study, biology, business.industry, Research, Incidence, Incidence (epidemiology), lcsh:R, Shiga toxin, Virology, STEC, Infectious Diseases, Italy, Child, Preschool, Population Surveillance, Hemolytic-Uremic Syndrome, biology.protein, hemolytic uremic syndrome, bacteria, epidemiology, business
Abstract

The mean annual incidence of hemolytic uremic syndrome in persons

Published
2003

2. Italian pediatric nutrition survey

Author

Michelangelo Barbaglia, Luigi Marmetucci, Nicoletta Cimadore, Alessandro Monaci, P. Fiore, Sergio Amarri, Elena Brunori, Maddalena Cioni, Carla Russo, Monica Barrani, P. Gandullia, Giovanna Zuin, Giuseppe Parisi, Rita Bellomo Anna, Michele Pinon, Nunzia Miglietti, Francesca Lizzoli, Elisa Mazzoni, Giulia Bardasi, Marisa Zoppo, Giacomo Cagnoli, S. Borodani, L. Forchielli, Monica Tulli, Fina Belli, Michele Salata, Giovanna Verlato, Vittoria Opinto, Roberto Bonaudo, Luisella Angelotti, Giulia Bruni, Elena Uga, Costantino De Giacomo, Antonietta Antonini Monica, Riccardo Guanà, Flavia Urbano, Rosaria Abate, Barbara Santangelo, Chiara Pettinari, Giovanna Fontanella, Patrizia Fusco, L. Lacitignola, Adalberto Brach Del Prever, Gina Ancora, S. Amarri, Laura Lacitignola, Paola Sparano, Marcello Lanari, Stefano Gatti, Francesca Nesi, Valentina De Cosmi, Alessia Frimaire, A Lezo, Francesca Penagini, Carmen Di Scala, Giuseppina Migliore, Roberta Annibali, Grazia Di Leo, Paola Peverelli, Mara Salmaso, Antonella Lezo, Paola Melli, M. Pastore, E. Brunori, Claudia Banzato, M.I. Spagnuolo, Antonella Diamanti, G. Verlato, Angelo Campanozzi, Mariella Pace, Martina Biagioni, Graziano Memmini, Laura Mistura, Sergio Del Vecchio, Annalisa Famiani, Enrico Felici, Germana Casaccia, Graziana Galvagno, Mario Castello, R. Panceri, Paola Accorsi, Martina Fomasi, Francesca Cortinovis, Michela Perrone, Teresa Capriati, Andrea Chiaro, Silvio Ferraris, Nicola Cecchi, Maria Immacolata Spagnuolo, Patrizia Petitti, Cristina Malaventura, Maria Sangerardi, Enrico Gasparrini, Francesco Savino, Luigi Besenzon, Anna Meneghini, Azzurra Guerra, Alessandra Sala, Maria Magistã Anna, Enrico Aidala, Donata Scatã, Gianluigi Palamone, Tiziano Basso, Giuseppe Maggiore, A. Diamanti, Alessandra Mazzocchi, Alessia Morganti, Andreina Stamati Filomena, Paolo Siani, Roberto Panceri, Maria Pastore, Paolo Gandullia, Lezo, A., Diamanti, A., Capriati, T., Gandullia, P., Fiore, P., Lacitignola, L., Gatti, S., Spagnuolo, M. I., Cecchi, N., Verlato, G., Borodani, S., Forchielli, L., Panceri, R., Brunori, E., Pastore, M., Amarri, S., Abate, R., Accorsi, P., Aidala, E., Ancora, G., Angelotti, L., Annibali, R., Antonini Monica, A., Banzato, C., Barbaglia, M., Bardasi, G., Barrani, M., Basso, T., Brach del Prever, A., Belli, F., Bellomo Anna, R., Besenzon, L., Biagioni, M., Bonaudo, R., Bruni, G., Cagnoli, G., Campanozzi, A., Casaccia, G., Castello, M., Chiaro, A., Cimadore, N., Cioni, M., Cortinovis, F., De Cosmi, V., De Giacomo, C., Del Vecchio, S., Di Leo, G., Di Scala, C., Famiani, A., Felici, E., Ferraris, S., Fomasi, M., Fontanella, G., Frimaire, A., Fusco, P., Galvagno, G., Gasparrini, E., Guana, R., Guerra, A., Lanari, M., Lizzoli, F., Maggiore, G., Magista Anna, M., Malaventura, C., Marmetucci, L., Mazzocchi, A., Mazzoni, E., Melli, P., Memmini, G., Meneghini, A., Miglietti, N., Migliore, G., Mistura, L., Monaci, A., Morganti, A., Nesi, F., Opinto, V., Pace, M., Palamone, G., Parisi, G., Penagini, F., Perrone, M., Petitti, P., Pettinari, C., Peverelli, P., Pinon, M., Russo, C., Sala, A., Salata, M., Salmaso, M., Sangerardi, M., Santangelo, B., Savino, F., Scata, D., Siani, P., Sparano, P., Stamati Filomena, A., Tulli, M., Uga, E., Urbano, F., Zoppo, M., Zuin, G., Abate, Rosaria, Accorsi, Paola, Aidala, Enrico, Amarri, Sergio, Ancora, Gina, Angelotti, Luisella, Annibali, Roberta, Antonini Monica, Antonietta, Banzato, Claudia, Barbaglia, Michelangelo, Bardasi, Giulia, Barrani, Monica, Basso, Tiziano, Brach Del Prever, Adalberto, Belli, Fina, Bellomo Anna, Rita, Besenzon, Luigi, Biagioni, Martina, Bonaudo, Roberto, Bruni, Giulia, Brunori, Elena, Cagnoli, Giacomo, Campanozzi, Angelo, Casaccia, Germana, Castello, Mario, Chiaro, Andrea, Cimadore, Nicoletta, Cioni, Maddalena, Cortinovis, Francesca, De Cosmi, Valentina, De Giacomo, Costantino, Del Vecchio, Sergio, Diamanti, Antonella, Di Leo, Grazia, Di Scala, Carmen, Famiani, Annalisa, Felici, Enrico, Ferraris, Silvio, Fomasi, Martina, Fontanella, Giovanna, Frimaire, Alessia, Fusco, Patrizia, Galvagno, Graziana, Gandullia, Paolo, Gasparrini, Enrico, Guanã , Riccardo, Guerra, Azzurra, Lanari, Marcello, Lacitignola, Laura, Lezo, Antonella, Lizzoli, Francesca, Maggiore, Giuseppe, Magistã Anna, Maria, Malaventura, Cristina, Marmetucci, Luigi, Mazzocchi, Alessandra, Mazzoni, Elisa, Melli, Paola, Memmini, Graziano, Meneghini, Anna, Miglietti, Nunzia, Migliore, Giuseppina, Mistura, Laura, Monaci, Alessandro, Morganti, Alessia, Nesi, Francesca, Opinto, Vittoria, Pace, Mariella, Palamone, Gianluigi, Panceri, Roberto, Parisi, Giuseppe, Pastore, Maria, Penagini, Francesca, Perrone, Michela, Petitti, Patrizia, Pettinari, Chiara, Peverelli, Paola, Pinon, Michele, Russo, Carla, Sala, Alessandra, Salata, Michele, Salmaso, Mara, Sangerardi, Maria, Santangelo, Barbara, Savino, Francesco, Scatã , Donata, Siani, Paolo, Spagnuolo, Maria Immacolata, Sparano, Paola, Stamati Filomena, Andreina, Tulli, Monica, Uga, Elena, Urbano, Flavia, Verlato, Giovanna, Zoppo, Marisa, and Zuin, Giovanna

Subjects
0301 basic medicine, Male, Pediatrics, Hospitalized patients, Endocrinology, Diabetes and Metabolism, Pediatric nutrition, 0302 clinical medicine, Child Development, Endocrinology, Prevalence, 030212 general & internal medicine, Growth Charts, Child, Nutritional support, Wasting, Growth Disorders, Pediatric, Stunting, Nutrition and Dietetics, Nutritional status, Nutrition Surveys, Diabetes and Metabolism, Italy, Malnutrition, Child, Preschool, Female, medicine.symptom, medicine.medical_specialty, Adolescent, Nutritional Status, Socio-culturale, Malnutrition in children, 03 medical and health sciences, Young Adult, medicine, Humans, 030109 nutrition & dietetics, business.industry, Infant, Anthropometry, medicine.disease, Parenteral nutrition, Chronic Disease, business, Child, Hospitalized
Abstract

Introduction the prevalence of malnutrition in children and its impact on clinical outcomes is underrecognized by clinicians in Italy as well as worldwide. A novel definition of pediatric malnutrition has been recently proposed by a working group of the Academy of Nutrition and Dietetics and American Society for Parenteral and Enteral Nutrition (A.S.P.E.N.), based on the correlation between illness and the use of zscores of anthropometric measurements. Aim to investigate the prevalence of malnutrition and related nutritional support among hospitalized children in Italy, in a nationwide survey performed in a single day (16/4/2015). Methods an open access website (http://nday.biomedia.net) was used to collected data from 73 hospitals and 101 wards in 14 Italian regions (1994 patients). Anonymous information was collected on hospitals' characteristics, patient's anthropometry, admission diagnosis, presence of chronic diseases and use of nutritional support: oral nutritional supplements (ONS), enteral nutrition (EN) or parenteral nutrition (PN). Z-scores of anthropometric measurements, calculated with Epi Info 7.1.5, defined nutritional status: wasting was identified by BMI or Weight-for-Length z-score (

Published
2017

3. Autosomal-dominant Alport syndrome: Natural history of a disease due to COL4A3 or COL4A4 gene

Author

Alessandra Renieri, Elena Bresin, Francesca Mari, Chiara Pescucci, Marco Seri, Rosanna Gusmano, Nunzia Miglietti, Cataldo Abaterusso, Paraskevi Vogiatzi, Elisa Scala, Rossella Caselli, Ilaria Longo, Pescucci C, Mari F, Longo I, Vogiatzi P, Caselli R, Scala E, Abaterusso C, Gusmano R, Seri M, Miglietti N, Bresin E, and Renieri A

Subjects
Adult, Collagen Type IV, Male, Genetic counseling, COL4A3, Nephritis, Hereditary, ALPORT SYNDROME, Kidney, urologic and male genital diseases, Asymptomatic, Autoantigens, medicine, Humans, COL4A4, Alport syndrome, Microhematuria, collagen IV genes, Child, COL4A3 gene, Aged, Genes, Dominant, Genetics, Aged, 80 and over, incomplete penetrance, Genetic heterogeneity, business.industry, COL4A4 gene, Glomerulonephritis, Middle Aged, medicine.disease, Penetrance, medicine.icd_9_cm_classification, Pedigree, Phenotype, inherited nephropathy, Nephrology, Mutation (genetic algorithm), Mutation, phenotypic variability, Female, autosomal-dominant Alport syndrome, medicine.symptom, business
Abstract

Autosomal-dominant Alport syndrome: Natural history of a disease due to COL4A3 or COL4A4 gene. Background Alport syndrome is a clinically and genetically heterogeneous nephropathy. The majority of cases are transmitted as an X-linked semidominant condition due to COL4A5 mutations. In this form males are more severely affected than females. Less than 10% of cases are autosomal recessive due to mutation in either COL4A3 or COL4A4 . In this rarer form, both males and females are severely affected. Only two cases of autosomal-dominant Alport syndrome have been reported, one due to a COL4A3 mutation and the other due to a COL4A4 mutation. Because of the paucity of the reported families, the natural history of autosomal-dominant Alport syndrome is mostly unknown. Methods Four families with likely autosomal-dominant Alport syndrome were investigated. COL4A3 and COL4A4 genes were analyzed by denaturing high-performance liquid chromatography (HPLC). Automated sequencing was performed to identify the underlying mutation. Results Two families had a mutation in the COL4A4 gene and two in the COL4A3 . Accurate clinical evaluation of family members showed interesting results. Affected individuals (22 persons) had a wide range of phenotypes from end-stage renal disease (ESRD) in the fifth decade to a nonprogressive isolated microhematuria. Finally, three heterozygous individuals (90, 22 and 11years old, respectively) were completely asymptomatic. Conclusion This paper demonstrated that patients affected by autosomal-dominant Alport syndrome have a high clinical variability. Moreover, a reduced penetrance of about 90% (3 of 25) may be considered for the assessment of recurrence risk during genetic counseling of these families.

Published
2004
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4. Risk factors for poor renal prognosis in children with hemolytic uremic syndrome

Author

Angela Caringella, Elio Salvaggio, Laura De Petris, Leopoldo Peratoner, Maurizio Gaido, Lucilla Ravà, Alberto Edefonti, Alfredo Caprioli, Rosanna Coppo, Alfonso Ferretti, Giovanni Montini, Giovambattista Capasso, Gianfranco Rizzoni, Rosa Penza, Salvatore Li Volti, Alessandra Gianviti, Carmen Setzu, Gianluigi Ardissino, Nunzia Miglietti, Salvatore Maffei, Carmine Pecoraro, Alberto Eugenio Tozzi, Graziella Zacchello, Ilse Ratsche, Alberto Bettinelli, Silvio Maringhini, Marco Pennesi, Francesco Perfumo, Ivana Pela, Giuliana Lama, Tommaso De Palo, Gianviti, A, Tozzi, Ae, DE PETRIS, L, Caprioli, A, Rava, L, Edefonti, A, Ardissino, G, Montini, G, Zacchello, G, Ferretti, A, Pecoraro, C, DE PALO, T, Caringella, A, Gaido, M, Coppo, R, Perfumo, F, Miglietti, N, Ratsche, I, Penza, R, Capasso, Giovambattista, Maringhini, S, LI VOLTI, S, Setzu, C, Pennesi, M, Bettinelli, A, Peratoner, L, Pela, I, Salvaggio, E, Lama, G, Maffei, S, and Rizzoni, G.

Subjects
Nephrology, Diarrhea, Male, medicine.medical_specialty, Atypical hemolytic uremic syndrome, Adolescent, medicine.medical_treatment, Gastroenterology, Shiga Toxin, Cohort Studies, Leukocyte Count, fluids and secretions, Shiga toxin-producing Escherichia coli, Central Nervous System Diseases, Risk Factors, Internal medicine, medicine, Humans, Risk factor, Age of Onset, Child, Escherichia coli Infections, Proportional Hazards Models, Prognostic factor, business.industry, Proportional hazards model, Infant, Classification, medicine.disease, Prognosis, Long-term outcome, Survival Analysis, Hemolytic urenic syndrome, Treatment Outcome, Italy, Child, Preschool, Pediatrics, Perinatology and Child Health, Immunology, Hemolytic-Uremic Syndrome, Plasmapheresis, Female, medicine.symptom, Age of onset, business, Kidney disease
Abstract

Many factors have been proposed as predictors of poor renal prognosis in children with hemolytic uremic syndrome (HUS), but their role is still controversial. Our aim was to detect the most reliable early predictors of poor renal prognosis to promptly identify children at major risk of bad outcome who could eventually benefit from early specific treatments, such as plasmapheresis. Prognostic factors identifiable at onset of HUS were evaluated by survival analysis and a proportional hazard model. These included age at onset, prodromal diarrhea (D), leukocyte count, central nervous system (CNS) involvement, and evidence of Shiga toxin-producing Escherichia coli (STEC) infection. Three hundred and eighty-seven HUS cases were reported; 276 were investigated for STEC infection and 189 (68%) proved positive. Age at onset, leukocyte count, and CNS involvement were not associated with the time to recovery. Absence of prodromal D and lack of evidence of STEC infection were independently associated with a poor renal prognosis; only 34% of patients D(-)STEC(- )recovered normal renal function compared with 65%-76% of D(+)STEC(+), D(+)STEC(-) and D(-)STEC(+ )patients. In conclusion, absence of both D and evidence of STEC infection are needed to identify patients with HUS and worst prognosis, while D(-) but STEC(+) patients have a significantly better prognosis.

Published
2003

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