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Automated identification and characterization of clustered weld defects, which comprise a plural of closely distributed individual member defects, is challenging. In this paper, time, Cartesian and Polar domain features in phased array ultrasonic sectorial scanning data are studied to segment clustered weld defect echoes by density based and K-means clustering. Through multiple domain segmentation, all individual member defect echoes are successfully identified. Then size and depth estimation is performed on each individual member defect to describe its patterns and to locate its depth. The developed technique is applied to experimental data collected from scanning on a tubular T weld test sample and is shown to discriminate, locate, and measure all the individual defects inside clustered defects. Based on the geometrical features of each individual member defect, they are classified as either point, linear or planar defects.

1. Myeloblasts can be discriminated in the supravital technique by the great numbers of tiny mitochondria in the cytoplasm and the absence of any other vitally stainable substance. 2. There are three stages in the maturation of myelocytes. 3. These three phases can be correlated with three types of the oxidase reaction. 4. One case of myeloblastic leucemia showed such an amount of an abnormal type of amitosis as to suggest the disordered cell division of neoplasms. 5. In this case transfusions were correlated with a maturation of myeloblasts into myelocytes, with an increase of the oxidase reaction, and with an increase in amitosis.

The exhibition of purpura as a clinical sign of disease was first ascribed solely to the effects of pestilential fevers. With the demonstration, however, by Denys 1 in 1887 that the purpura of Werlhof is characterized by a lowered level of circulating blood platelets, the clinical purpuric states were definitely shown to consist of thrombocytopenic and nonthrombocytopenic groups. Further differentiation within the thrombocytopenic group has been accomplished slowly since Denys's observations and has depended largely on cumulative experience with known etiologic states in which both purpura and marked thrombocytopenia have been observed to occur. There has thus arisen the practical and important subclassification of thrombocytopenic purpura into two groups—the idiopathic and the symptomatic. This is justified because the successful management of the former often requires life-saving surgery, whereas the latter should be managed strictly by medical means. The reversal of these therapeutic principles may promote disaster to the patient. The

Progress in the solution of the problems of disease in man must depend to a considerable degree on fundamental progress in the recognition and interpretation of alterations in disturbed physiologic equilibriums. It may be true that physiology "has its own problems, great in number and enormous in complexity, and those of disease are not among them."1Nevertheless, experience has established that the origin and explanation of many abnormal states may be traced to deranged physiologic processes, and more particularly to disturbed physiologic equilibriums. Granted that this is beyond the scope of pure physiologic research, the recognition of states of physiologic imbalance and of pathologic alterations produced in the tissues by this imbalance then becomes an important concern of the clinical investigator. That this "borderline" territory between physiology and medicine has proved a very fruitful field for clinical investigation, in one direction at least, is quite apparent when one reviews