42 results on '"Lee, Chunsik"'
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2. Critical role of mitogen-inducible gene 6 in restraining endothelial cell permeability to maintain vascular homeostasis
3. Correction to: Critical role of mitogen-inducible gene 6 in restraining endothelial cell permeability to maintain vascular homeostasis
4. Targeting of histone methyltransferase DOT1L plays a dual role in chemosensitization of retinoblastoma cells and enhances the efficacy of chemotherapy
5. Critical role of caveolin-1 in ocular neovascularization and multitargeted antiangiogenic effects of cavtratin via JNK
6. Correction to: Critical role of mitogen-inducible gene 6 in restraining endothelial cell permeability to maintain vascular homeostasis
7. Critical role of mitogen-inducible gene 6 in restraining endothelial cell permeability to maintain vascular homeostasis
8. Vasoprotective effect of PDGF-CC mediated by HMOX1 rescues retinal degeneration
9. UHRF1 depletion sensitizes retinoblastoma cells to chemotherapeutic drugs via downregulation of XRCC4
10. Genome maintenance in retinoblastoma: Implications for therapeutic vulnerabilities (Review)
11. PDGF-CC blockade inhibits pathological angiogenesis by acting on multiple cellular and molecular targets
12. VEGF-B Is Dispensable for Blood Vessel Growth but Critical for Their Survival, and VEGF-B Targeting Inhibits Pathological Angiogenesis
13. Corrigendum: Platelet-Derived Growth Factor-D Activates Complement System to Propagate Macrophage Polarization and Neovascularization
14. Platelet-Derived Growth Factor-D Activates Complement System to Propagate Macrophage Polarization and Neovascularization
15. Mitogen-Inducible Gene 6 Inhibits Angiogenesis by Binding to SHC1 and Suppressing Its Phosphorylation
16. UHRF1 downmodulation enhances antitumor effects of histone deacetylase inhibitors in retinoblastoma by augmenting oxidative stress‐mediated apoptosis
17. VEGF-B: A survival, or an angiogenic factor?
18. UHRF1 downmodulation enhances antitumor effects of histone deacetylase inhibitors in retinoblastoma by augmenting oxidative stress‐mediated apoptosis.
19. Inhibitory effect of caveolin-1 in vascular endothelial cells, pericytes and smooth muscle cells
20. PDGF-CC underlies resistance to VEGF-A inhibition and combinatorial targeting of both suppresses pathological angiogenesis more efficiently
21. VEGF-B inhibits hyperglycemia- and Macugen-induced retinal apoptosis
22. Elevated expression of NEU1 sialidase in idiopathic pulmonary fibrosis provokes pulmonary collagen deposition, lymphocytosis, and fibrosis
23. Molecular Mechanisms of Action of Histidine-rich Glycoprotein in Angiogenesis Inhibition
24. NEU1 Sialidase Regulates the Sialylation State of CD31 and Disrupts CD31-driven Capillary-like Tube Formation in Human Lung Microvascular Endothelia
25. Proliferative and Survival Effects of PUMA Promote Angiogenesis
26. Survival effect of PDGF-CC rescues neurons from apoptosis in both brain and retina by regulating GSK3β phosphorylation
27. Type 3 cystatins : fetuins, kininogen and histidine-rich glycoprotein
28. Minimal active domain and mechanism of action of the angiogenesis inhibitor histidine-rich glycoprotein
29. Signal transduction in endothelial cells by the angiogenesis inhibitor histidine-rich glycoprotein targets focal adhesions
30. Proliferative and Survival Effects of PUMA Promote Angiogenesis
31. A fragment of histidine-rich glycoprotein is a potent inhibitor of tumor vascularization.
32. VEGF-independent angiogenic pathways induced by PDGF-C
33. Platelet-derived Growth Factor-DD Targeting Arrests Pathological Angiogenesis by Modulating Glycogen Synthase Kinase-3β Phosphorylation
34. Survival effect of PDGF-CC rescues neurons from apoptosis in both brain and retina by regulating GSK3β phosphorylation
35. VEGF-B
36. Minimal Active Domain and Mechanism of Action of the Angiogenesis Inhibitor Histidine-Rich Glycoprotein
37. Transgenic Expression of Dominant Negative Tuberin through a Strong Constitutive Promoter Results in a Tissue-specific Tuberous Sclerosis Phenotype in the Skin and Brain
38. A Fragment of Histidine-Rich Glycoprotein Is a Potent Inhibitor of Tumor Vascularization
39. PDGF-CC blockade inhibits pathological angiogenesis by acting on multiple cellular and molecular targets.
40. Regulation of tumor-associated macrophage infiltration, VEGF and MMP production, by the angiogenesis inhibitor histidine-rich glycoprotein.
41. Factors Influencing Tacit Knowledge Sharing in Research Groups in Higher Education Institutions.
42. Patent Application Titled 'Use Of Vegf-B For Treating Diseases Involving Neoangiogenesis' Published Online (USPTO 20190060404)
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