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2. Prospective study of oral microbiome and gastric cancer risk among Asian, African American and European American populations

Author

Yang, Yaohua, primary, Long, Jirong, additional, Wang, Cong, additional, Blot, William J., additional, Pei, Zhiheng, additional, Shu, Xiang, additional, Wu, Fen, additional, Rothman, Nathaniel, additional, Wu, Jie, additional, Lan, Qing, additional, Cai, Qiuyin, additional, Zheng, Wei, additional, Chen, Yu, additional, and Shu, Xiao‐Ou, additional

Published
2021
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3. Red meat consumption, cooking mutagens,NAT1 /2genotypes and pancreatic cancer risk in two ethnically diverse prospective cohorts

Author

Huang, Brian Z., primary, Wang, Songren, additional, Bogumil, David, additional, Wilkens, Lynne R., additional, Wu, Lang, additional, Blot, William J., additional, Zheng, Wei, additional, Shu, Xiao‐Ou, additional, Pandol, Stephen J., additional, Le Marchand, Loïc, additional, and Setiawan, Veronica Wendy, additional

Published
2021
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5. Associations of coffee and tea consumption with lung cancer risk

Author

Zhu, Jingjing, primary, Smith‐Warner, Stephanie A., additional, Yu, Danxia, additional, Zhang, Xuehong, additional, Blot, William J., additional, Xiang, Yong‐Bing, additional, Sinha, Rashmi, additional, Park, Yikyung, additional, Tsugane, Shoichiro, additional, White, Emily, additional, Koh, Woon‐Puay, additional, Park, Sue K., additional, Sawada, Norie, additional, Kanemura, Seiki, additional, Sugawara, Yumi, additional, Tsuji, Ichiro, additional, Robien, Kim, additional, Tomata, Yasutake, additional, Yoo, Keun‐Young, additional, Kim, Jeongseon, additional, Yuan, Jian‐Min, additional, Gao, Yu‐Tang, additional, Rothman, Nathaniel, additional, Lazovich, DeAnn, additional, Abe, Sarah K., additional, Rahman, Md Shafiur, additional, Loftfield, Erikka, additional, Takata, Yumie, additional, Li, Xin, additional, Lee, Jung Eun, additional, Saito, Eiko, additional, Freedman, Neal D., additional, Inoue, Manami, additional, Lan, Qing, additional, Willett, Walter C., additional, Zheng, Wei, additional, and Shu, Xiao‐Ou, additional

Published
2020
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7. Circulating markers of cellular immune activation in prediagnostic blood sample and lung cancer risk in the Lung Cancer Cohort Consortium (LC3)

Author

Huang, Joyce Y., primary, Larose, Tricia L., additional, Luu, Hung N., additional, Wang, Renwei, additional, Fanidi, Anouar, additional, Alcala, Karine, additional, Stevens, Victoria L., additional, Weinstein, Stephanie J., additional, Albanes, Demetrius, additional, Caporaso, Neil E., additional, Purdue, Mark P., additional, Ziegler, Regina G., additional, Freedman, Neal D., additional, Lan, Qing, additional, Prentice, Ross L., additional, Pettinger, Mary, additional, Thomson, Cynthia A., additional, Cai, Qiuyin, additional, Wu, Jie, additional, Blot, William J., additional, Shu, Xiao‐Ou, additional, Zheng, Wei, additional, Arslan, Alan A., additional, Zeleniuch‐Jacquotte, Anne, additional, Le Marchand, Loïc, additional, Wilkens, Lynn R., additional, Haiman, Christopher A., additional, Zhang, Xuehong, additional, Stampfer, Meir J., additional, Han, Jiali, additional, Giles, Graham G., additional, Hodge, Allison M., additional, Severi, Gianluca, additional, Johansson, Mikael, additional, Grankvist, Kjell, additional, Langhammer, Arnulf, additional, Hveem, Kristian, additional, Xiang, Yong‐Bing, additional, Li, Hong‐Lan, additional, Gao, Yu‐Tang, additional, Visvanathan, Kala, additional, Ueland, Per M., additional, Midttun, Øivind, additional, Ulvi, Arve, additional, Buring, Julie E., additional, Lee, I‐Min, additional, Sesso, Howard D., additional, Gaziano, J. Michael, additional, Manjer, Jonas, additional, Relton, Caroline, additional, Koh, Woon‐Puay, additional, Brennan, Paul, additional, Johansson, Mattias, additional, and Yuan, Jian‐Min, additional

Published
2019
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9. Alcohol consumption and prostate cancer incidence and progression: A Mendelian randomisation study

Author

Brunner, Clair, Davies, Neil M., Martin, Richard M., Eeles, Rosalind, Easton, Doug, Kote‐Jarai, Zsofia, Al Olama, Ali Amin, Benlloch, Sara, Muir, Kenneth, Giles, Graham, Wiklund, Fredrik, Gronberg, Henrik, Haiman, Christopher A., Schleutker, Johanna, Nordestgaard, Børge G., Travis, Ruth C., Neal, David, Donovan, Jenny, Hamdy, Freddie C., Pashayan, Nora, Khaw, Kay‐Tee, Stanford, Janet L., Blot, William J., Thibodeau, Stephen, Maier, Christiane, Kibel, Adam S., Cybulski, Cezary, Cannon‐Albright, Lisa, Brenner, Hermann, Park, Jong, Kaneva, Radka, Batra, Jyotsna, Teixeira, Manuel R., Pandha, Hardev, and Zuccolo, Luisa

Subjects
Aged, 80 and over, Male, Alcohol Drinking, alcohol, Aldehyde Dehydrogenase, Mitochondrial, Incidence, Prostatic Neoplasms, Retinal Dehydrogenase, Aldehyde Dehydrogenase, Middle Aged, prostate cancer, Polymorphism, Single Nucleotide, Survival Analysis, Aldehyde Dehydrogenase 1 Family, Linkage Disequilibrium, Case-Control Studies, Disease Progression, Humans, Regression Analysis, alcohol metabolising genes, Neoplasm Grading, Mendelian randomisation, Cancer Epidemiology, Aged
Abstract

Prostate cancer is the most common cancer in men in developed countries, and is a target for risk reduction strategies. The effects of alcohol consumption on prostate cancer incidence and survival remain unclear, potentially due to methodological limitations of observational studies. In this study, we investigated the associations of genetic variants in alcohol‐metabolising genes with prostate cancer incidence and survival. We analysed data from 23,868 men with prostate cancer and 23,051 controls from 25 studies within the international PRACTICAL Consortium. Study‐specific associations of 68 single nucleotide polymorphisms (SNPs) in 8 alcohol‐metabolising genes (Alcohol Dehydrogenases (ADHs) and Aldehyde Dehydrogenases (ALDHs)) with prostate cancer diagnosis and prostate cancer‐specific mortality, by grade, were assessed using logistic and Cox regression models, respectively. The data across the 25 studies were meta‐analysed using fixed‐effect and random‐effects models. We found little evidence that variants in alcohol metabolising genes were associated with prostate cancer diagnosis. Four variants in two genes exceeded the multiple testing threshold for associations with prostate cancer mortality in fixed‐effect meta‐analyses. SNPs within ALDH1A2 associated with prostate cancer mortality were rs1441817 (fixed effects hazard ratio, HRfixed = 0.78; 95% confidence interval (95%CI):0.66,0.91; p values = 0.002); rs12910509, HRfixed = 0.76; 95%CI:0.64,0.91; p values = 0.003); and rs8041922 (HRfixed = 0.76; 95%CI:0.64,0.91; p values = 0.002). These SNPs were in linkage disequilibrium with each other. In ALDH1B1, rs10973794 (HRfixed = 1.43; 95%CI:1.14,1.79; p values = 0.002) was associated with prostate cancer mortality in men with low‐grade prostate cancer. These results suggest that alcohol consumption is unlikely to affect prostate cancer incidence, but it may influence disease progression., What's new? Alcohol may spur prostate cancer progression, though it does not appear to affect incidence, according to new analysis. Variation in genes involved in alcohol metabolism affect how much the body is exposed to carcinogenic metabolites. These authors examined 68 genetic variants in alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH) genes, seeking a link with prostate cancer risk. While they found no evidence that these variants alter prostate cancer incidence, they did show that SNPs in the ALDH1A2 gene affect prostate cancer mortality. From a public health standpoint, these results suggest reducing alcohol consumption could slow prostate cancer disease progression.

Published
2017
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11. Impaired functional vitamin B6 status is associated with increased risk of lung cancer

Author

Theofylaktopoulou, Despoina, primary, Midttun, Øivind, additional, Ueland, Per M., additional, Meyer, Klaus, additional, Fanidi, Anouar, additional, Zheng, Wei, additional, Shu, Xiao‐Ou, additional, Xiang, Yong‐Bing, additional, Prentice, Ross, additional, Pettinger, Mary, additional, Thomson, Cynthia A., additional, Giles, Graham G, additional, Hodge, Allison, additional, Cai, Qiuyin, additional, Blot, William J., additional, Wu, Jie, additional, Johansson, Mikael, additional, Hultdin, Johan, additional, Grankvist, Kjell, additional, Stevens, Victoria L., additional, McCullough, Marjorie M., additional, Weinstein, Stephanie J., additional, Albanes, Demetrius, additional, Ziegler, Regina, additional, Freedman, Neal D., additional, Langhammer, Arnulf, additional, Hveem, Kristian, additional, Næss, Marit, additional, Sesso, Howard D., additional, Gaziano, J. Michael, additional, Buring, Julie E., additional, Lee, I‐Min, additional, Severi, Gianluca, additional, Zhang, Xuehong, additional, Stampfer, Meir J., additional, Han, Jiali, additional, Smith‐Warner, Stephanie A., additional, Zeleniuch‐Jacquotte, Anne, additional, Le Marchand, Loic, additional, Yuan, Jian‐Min, additional, Wang, Renwei, additional, Butler, Lesley M., additional, Koh, Woon‐Puay, additional, Gao, Yu‐Tang, additional, Rothman, Nathaniel, additional, Ericson, Ulrika, additional, Sonestedt, Emily, additional, Visvanathan, Kala, additional, Jones, Miranda R., additional, Relton, Caroline, additional, Brennan, Paul, additional, Johansson, Mattias, additional, and Ulvik, Arve, additional

Published
2018
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12. Assessing the role of insulin-like growth factors and binding proteins in prostate cancer using Mendelian randomization: Genetic variants as instruments for circulating levels

Author

Bonilla, Carolina, Lewis, Sarah J, Rowlands, Mari-Anne, Gaunt, Tom R, Davey Smith, George, Gunnell, David, Palmer, Tom, Donovan, Jenny L, Hamdy, Freddie C, Neal, David E, Eeles, Rosalind, Easton, Doug, Kote-Jarai, Zsofia, Al Olama, Ali Amin, Benlloch, Sara, Muir, Kenneth, Giles, Graham G, Wiklund, Fredrik, Grönberg, Henrik, Haiman, Christopher A, Schleutker, Johanna, Nordestgaard, Børge G, Travis, Ruth C, Pashayan, Nora, Khaw, Kay-Tee, Stanford, Janet L, Blot, William J, Thibodeau, Stephen, Maier, Christiane, Kibel, Adam S, Cybulski, Cezary, Cannon-Albright, Lisa, Brenner, Hermann, Park, Jong, Kaneva, Radka, Batra, Jyotsna, Teixeira, Manuel R, Pandha, Hardev, PRACTICAL consortium, Lathrop, Mark, Martin, Richard M, Holly, Jeff MP, Easton, Douglas [0000-0003-2444-3247], Khaw, Kay-Tee [0000-0002-8802-2903], and Apollo - University of Cambridge Repository

Subjects
Male, Prostatic Neoplasms, ALSPAC, Mendelian Randomization Analysis, Middle Aged, prostate cancer, insulin-like growth factor-binding proteins, Polymorphism, Single Nucleotide, PRACTICAL, United Kingdom, UKHLS, Insulin-Like Growth Factor Binding Proteins, single nucleotide polymorphisms, IGFBP3, Somatomedins, insulin-like growth factors, Case-Control Studies, Mendelian randomization, Humans, ProtecT, Longitudinal Studies, Neoplasm Grading, Aged, Genome-Wide Association Study, Neoplasm Staging
Abstract

Circulating insulin-like growth factors (IGFs) and their binding proteins (IGFBPs) are associated with prostate cancer. Using genetic variants as instruments for IGF peptides, we investigated whether these associations are likely to be causal. We identified from the literature 56 single nucleotide polymorphisms (SNPs) in the IGF axis previously associated with biomarker levels (8 from a genome-wide association study [GWAS] and 48 in reported candidate genes). In ∼700 men without prostate cancer and two replication cohorts (N ∼ 900 and ∼9,000), we examined the properties of these SNPS as instrumental variables (IVs) for IGF-I, IGF-II, IGFBP-2 and IGFBP-3. Those confirmed as strong IVs were tested for association with prostate cancer risk, low (< 7) vs. high (≥ 7) Gleason grade, localised vs. advanced stage, and mortality, in 22,936 controls and 22,992 cases. IV analysis was used in an attempt to estimate the causal effect of circulating IGF peptides on prostate cancer. Published SNPs in the IGFBP1/IGFBP3 gene region, particularly rs11977526, were strong instruments for IGF-II and IGFBP-3, less so for IGF-I. Rs11977526 was associated with high (vs. low) Gleason grade (OR per IGF-II/IGFBP-3 level-raising allele 1.05; 95% CI: 1.00, 1.10). Using rs11977526 as an IV we estimated the causal effect of a one SD increase in IGF-II (∼265 ng/mL) on risk of high vs. low grade disease as 1.14 (95% CI: 1.00, 1.31). Because of the potential for pleiotropy of the genetic instruments, these findings can only causally implicate the IGF pathway in general, not any one specific biomarker.

Published
2016

13. Alcohol consumption and prostate cancer incidence and progression: A Mendelian randomisation study

Author

Brunner, Clair, Davies, Neil M, Martin, Richard M, Eeles, Rosalind, Easton, Doug, Kote-Jarai, Zsofia, Al Olama, Ali Amin, Benlloch, Sara, Muir, Kenneth, Giles, Graham, Wiklund, Fredrik, Gronberg, Henrik, Haiman, Christopher A, Schleutker, Johanna, Nordestgaard, Børge G, Travis, Ruth C, Neal, David, Donovan, Jenny, Hamdy, Freddie C, Pashayan, Nora, Khaw, Kay-Tee, Stanford, Janet L, Blot, William J, Thibodeau, Stephen, Maier, Christiane, Kibel, Adam S, Cybulski, Cezary, Cannon-Albright, Lisa, Brenner, Hermann, Park, Jong, Kaneva, Radka, Batra, Jyotsna, Teixeira, Manuel R, Pandha, Hardev, PRACTICAL Consortium, Zuccolo, Luisa, Easton, Douglas [0000-0003-2444-3247], Khaw, Kay-Tee [0000-0002-8802-2903], and Apollo - University of Cambridge Repository

Subjects
Aged, 80 and over, Male, Alcohol Drinking, alcohol, Aldehyde Dehydrogenase, Mitochondrial, Incidence, Prostatic Neoplasms, Retinal Dehydrogenase, Aldehyde Dehydrogenase, Middle Aged, prostate cancer, Polymorphism, Single Nucleotide, Survival Analysis, Aldehyde Dehydrogenase 1 Family, Linkage Disequilibrium, Case-Control Studies, Disease Progression, Humans, Regression Analysis, alcohol metabolising genes, Neoplasm Grading, Mendelian randomisation, Aged
Abstract

Prostate cancer is the most common cancer in men in developed countries, and is a target for risk reduction strategies. The effects of alcohol consumption on prostate cancer incidence and survival remain unclear, potentially due to methodological limitations of observational studies. In this study, we investigated the associations of genetic variants in alcohol-metabolising genes with prostate cancer incidence and survival. We analysed data from 23,868 men with prostate cancer and 23,051 controls from 25 studies within the international PRACTICAL Consortium. Study-specific associations of 68 single nucleotide polymorphisms (SNPs) in 8 alcohol-metabolising genes (Alcohol Dehydrogenases (ADHs) and Aldehyde Dehydrogenases (ALDHs)) with prostate cancer diagnosis and prostate cancer-specific mortality, by grade, were assessed using logistic and Cox regression models, respectively. The data across the 25 studies were meta-analysed using fixed-effect and random-effects models. We found little evidence that variants in alcohol metabolising genes were associated with prostate cancer diagnosis. Four variants in two genes exceeded the multiple testing threshold for associations with prostate cancer mortality in fixed-effect meta-analyses. SNPs within ALDH1A2 associated with prostate cancer mortality were rs1441817 (fixed effects hazard ratio, HRfixed = 0.78; 95% confidence interval (95%CI):0.66,0.91; p values = 0.002); rs12910509, HRfixed = 0.76; 95%CI:0.64,0.91; p values = 0.003); and rs8041922 (HRfixed = 0.76; 95%CI:0.64,0.91; p values = 0.002). These SNPs were in linkage disequilibrium with each other. In ALDH1B1, rs10973794 (HRfixed = 1.43; 95%CI:1.14,1.79; p values = 0.002) was associated with prostate cancer mortality in men with low-grade prostate cancer. These results suggest that alcohol consumption is unlikely to affect prostate cancer incidence, but it may influence disease progression.

Published
2016
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14. Blood lipids and prostate cancer: a Mendelian randomization analysis

Author

Bull, Caroline J, Bonilla, Carolina, Holly, Jeff MP, Perks, Claire M, Davies, Neil, Haycock, Philip, Yu, Oriana Hoi Yun, Richards, J Brent, Eeles, Rosalind, Easton, Doug, Kote-Jarai, Zsofia, Amin Al Olama, Ali, Benlloch, Sara, Muir, Kenneth, Giles, Graham G, MacInnis, Robert J, Wiklund, Fredrik, Gronberg, Henrik, Haiman, Christopher A, Schleutker, Johanna, Nordestgaard, Børge G, Travis, Ruth C, Neal, David, Pashayan, Nora, Khaw, Kay-Tee, Stanford, Janet L, Blot, William J, Thibodeau, Stephen, Maier, Christiane, Kibel, Adam S, Cybulski, Cezary, Cannon-Albright, Lisa, Brenner, Hermann, Park, Jong, Kaneva, Radka, Batra, Jyotsna, Teixeira, Manuel R, Micheal, Agnieszka, Pandha, Hardev, Smith, George Davey, Lewis, Sarah J, Martin, Richard M, PRACTICAL Consortium, Easton, Douglas [0000-0003-2444-3247], Amin Al Olama, Ali [0000-0002-7178-3431], Khaw, Kay-Tee [0000-0002-8802-2903], and Apollo - University of Cambridge Repository

Subjects
Male, Genetic Variation, Prostatic Neoplasms, Mendelian Randomization Analysis, prostate cancer, Lipids, Polymorphism, Single Nucleotide, statins, Cholesterol, Quantitative Trait, Heritable, Meta-Analysis as Topic, Case-Control Studies, Population Surveillance, Mendelian randomization, Odds Ratio, Humans, lipids (amino acids, peptides, and proteins), Genetic Predisposition to Disease, Neoplasm Grading, Genetic Association Studies, Genome-Wide Association Study, Neoplasm Staging
Abstract

Genetic risk scores were used as unconfounded instruments for specific lipid traits (Mendelian randomization) to assess whether circulating lipids causally influence prostate cancer risk. Data from 22,249 prostate cancer cases and 22,133 controls from 22 studies within the international PRACTICAL consortium were analyzed. Allele scores based on single nucleotide polymorphisms (SNPs) previously reported to be uniquely associated with each of low-density lipoprotein (LDL), high-density lipoprotein (HDL), and triglyceride (TG) levels, were first validated in an independent dataset, and then entered into logistic regression models to estimate the presence (and direction) of any causal effect of each lipid trait on prostate cancer risk. There was weak evidence for an association between the LDL genetic score and cancer grade: the odds ratio (OR) per genetically instrumented standard deviation (SD) in LDL, comparing high- (≥7 Gleason score) versus low-grade (

Published
2016
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17. A Small Number of Candidate Gene SNPs Reveal Continental Ancestry in African Americans

Author

Kodaman, Nuri, primary, Aldrich, Melinda C., additional, Smith, Jeffrey R., additional, Signorello, Lisa B., additional, Bradley, Kevin, additional, Breyer, Joan, additional, Cohen, Sarah S., additional, Long, Jirong, additional, Cai, Qiuyin, additional, Giles, Justin, additional, Bush, William S., additional, Blot, William J., additional, Matthews, Charles E., additional, and Williams, Scott M., additional

Published
2012
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18. ADIPOQ, ADIPOR1, andADIPOR2Polymorphisms in Relation to Serum Adiponectin Levels and BMI in Black and White Women

Author

Cohen, Sarah S., primary, Gammon, Marilie D., additional, North, Kari E., additional, Millikan, Robert C., additional, Lange, Ethan M., additional, Williams, Scott M., additional, Zheng, Wei, additional, Cai, Qiuyin, additional, Long, Jirong, additional, Smith, Jeffrey R., additional, Signorello, Lisa B., additional, Blot, William J., additional, and Matthews, Charles E., additional

Published
2011
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20. Food group intake and risk of subtypes of esophageal and gastric cancer

Author

Navarro Silvera, Stephanie A., primary, Mayne, Susan T., additional, Risch, Harvey, additional, Gammon, Marilee D., additional, Vaughan, Thomas L., additional, Chow, Wong‐Ho, additional, Dubrow, Robert, additional, Schoenberg, Janet B., additional, Stanford, Janet L., additional, West, A. Brian, additional, Rotterdam, Heidrun, additional, Blot, William J., additional, and Fraumeni, Joseph F., additional

Published
2008
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25. Use of nonaspirin NSAIDs and risk of lung cancer

Author

Skriver, Mette Vinther, primary, Nørgaard, Mette, additional, Poulsen, Aslak Harbo, additional, Friis, Søren, additional, Harving, Henrik, additional, Fryzek, Jon, additional, McLaughlin, Joseph K., additional, Blot, William J., additional, Olsen, Jørgen H., additional, and Sørensen, Henrik Toft, additional

Published
2005
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28. Occupation and risk of esophageal and gastric cardia adenocarcinoma

Author

Engel, Lawrence S., primary, Vaughan, Thomas L., additional, Gammon, Marilie D., additional, Chow, Wong-Ho, additional, Risch, Harvey A., additional, Dubrow, Robert, additional, Mayne, Susan T., additional, Rotterdam, Heidrun, additional, Schoenberg, Janet B., additional, Stanford, Janet L., additional, West, A. Brian, additional, Blot, William J., additional, and Fraumeni, Joseph F., additional

Published
2002
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34. Family history of cancer and risk of esophageal and gastric cancers in the United States

Author

Dhillon, Preet K., primary, Farrow, Diana C., additional, Vaughan, Thomas L., additional, Chow, Wong-Ho, additional, Risch, Harvey A., additional, Gammon, Marilie D., additional, Mayne, Susan T., additional, Stanford, Janet L., additional, Schoenberg, Janet B., additional, Ahsan, Habibul, additional, Dubrow, Robert, additional, West, A. Brian, additional, Rotterdam, Heidrun, additional, Blot, William J., additional, and Fraumeni, Joseph F., additional

Published
2001
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35. Cancer occurrence after cosmetic breast implantation in Denmark

Author

Mellemkjær, Lene, primary, Kjøller, Kim, additional, Friis, Søren, additional, McLaughlin, Joseph K., additional, Høgsted, Charlotte, additional, Winther, Jeanette F., additional, Breiting, Vibeke, additional, Krag, Christen, additional, Krüger Kjær, Susanne, additional, Blot, William J., additional, and Olsen, Jørgen H., additional

Published
2000
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36. Evolution of precancerous lesions in a rural Chinese population at high risk of gastric cancer

Author

You, Wei-Cheng, primary, Li, Ji-You, additional, Blot, William J., additional, Chang, Yun-Sheng, additional, Jin, Mao-Lin, additional, Gail, Mitchell H., additional, Zhang, Lian, additional, Liu, Wei-Dong, additional, Ma, Jun-Ling, additional, Hu, Yuan-Ren, additional, Mark, Steven D., additional, Correa, Pelayo, additional, Fraumeni, Joseph F., additional, and Xu, Guang-Wei, additional

Published
1999
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37. Risk of stomach cancer in relation to consumption of cigarettes, alcohol, tea and coffee in Warsaw, Poland

Author

Chow, Wong‐Ho, primary, Swanson, Christine A., additional, Lissowska, Jolanta, additional, Groves, Frank D., additional, Sobin, Leslie H., additional, Nasierowska‐Guttmejer, Anna, additional, Radziszewski, Jakub, additional, Regula, Jaroslaw, additional, Hsing, Ann W., additional, Jagannatha, Shyla, additional, Zatonski, Witold, additional, and Blot, William J., additional

Published
1999
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43. Breast implants and cancer risk in Denmark

Author

Friis, Søren, primary, McLaughlin, Joseph K., additional, Mellemkjaer, Lene, additional, Kjøller, Kim H., additional, Blot, William J., additional, Boice, John D., additional, Fraumeni, Joseph F., additional, and Olsen, Jørgen H., additional

Published
1997
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45. Cancer risks among iron and steel workers in Anshan, China, part II: Case‐control studies of lung and stomach cancer

Author

Xu, Zhaoyi, primary, Brown, Linda Morris, additional, Pan, Guo‐Wei, additional, Liu, Tie‐Fu, additional, Gao, Gun‐Shen, additional, Stone, B. J., additional, Cao, Rei‐Ming, additional, Guan, De‐Xian, additional, Sheng, Jia‐Hua, additional, Yan, Zuo‐Shen, additional, Dosemeci, Mustafa, additional, Fraumeni, Joseph F., additional, and Blot, William J., additional

Published
1996
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47. A cohort study of cancer among benzene-exposed workers in China: Overall results

Author

Yin, Song-Nian, primary, Hayes, Richard B., additional, Linet, Martha S., additional, Li, Gui-Lan, additional, Dosemeci, Mustafa, additional, Travis, Lois B., additional, Li, Chin-Yang, additional, Zhang, Zhi-Nan, additional, Li, De-Gao, additional, Chow, Wong-Ho, additional, Wacholder, Sholom, additional, Wang, Yao-Zu, additional, Jiang, Zhong-Lian, additional, Dai, Tan-Rong, additional, Zhang, Wan-You, additional, Chao, Xin-Jie, additional, Ye, Pei-Zheng, additional, Kou, Qing-Rui, additional, Zhang, Xi-Chun, additional, Lin, Xia-Fang, additional, Meng, Juan-Fei, additional, Ding, Cheng-Yu, additional, Zho, Jie-Sheng, additional, and Blot, William J., additional

Published
1996
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48. Human papillomavirus and cervical neoplasia: A case‐control study in Taiwan

Author

Liaw, Kai‐Li, primary, Hsing, Ann W., additional, Chen, Chem‐Jen, additional, Schiffmfman, Mark H., additional, Zhang, Tracy Y., additional, Hsieh, Chang‐Yao, additional, Greer, Catherine E., additional, You, San‐Lin, additional, Huang, Thomas W., additional, Wu, Tzyy‐Choou, additional, O'leary, Timothy J., additional, Seidman, Jeffrey D., additional, Blot, William J., additional, Meinert, Curtis L., additional, and Manos, M. Michele, additional

Published
1995
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