43 results on '"Wang, J. C."'
Search Results
2. Comprehensive analysis of the PD-L1 and immune infiltrates of N6-methyladenosine related long non-coding RNAs in bladder cancer.
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Xue, M. Q., Wang, Y. L., Wang, J. C., Wang, X. D., Wang, X. J., and Zhang, Y. Q.
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PROGRAMMED cell death 1 receptors ,LINCRNA ,PROGRAMMED death-ligand 1 ,TUMOR-infiltrating immune cells ,BLADDER cancer ,ADENOSINES ,NEUTROPHILS - Abstract
Bladder cancer (BLCA) is one of the most frequent genitourinary cancers, with a high rate of morbidity and mortality. The connection of m6A-related lncRNAs with PD-L1 and tumor immune microenvironment (TIME) in BLCA prognosis was extensively investigated in this study, which could suggest novel therapeutic targets for further investigation. 30 m6A-associated lncRNAs with predictive values from the TCGA data set were identified with co-expression analysis. Cluster2 was correlated with a poor prognosis, upregulated PD-L1 expression, and higher immune ratings. Cluster2 had larger amounts of resting CD4 memory-activated T cells, M2 macrophages, neutrophils, and NK cells infiltration. "CHEMOKINE SIGNALING PATHWAY" was the most significantly enriched signaling pathway according to GSEA, which may play an important role in the different immune cell infiltrates between cluster1/2. The risk model for m6A-related lncRNAs could be employed in a prognostic model to predict BLCA prognosis, regardless of other clinical features. Collectively, m6A-related lncRNAs were linked to PD-L1 and TIME, which would dynamically affect the number of tumor-infiltrating immune cells. m6A-related lncRNAs may be key mediators of PD-L1 expression and immune cells infiltration and may strongly affect the TIME of BLCA. [ABSTRACT FROM AUTHOR]
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- 2022
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3. Geometry and Filling Features of Hydraulic Fractures in Coalbed Methane Reservoirs Based on Subsurface Observations.
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Li, R., Wang, S. W., Lyu, S. F., Lu, W., Li, G. F., and Wang, J. C.
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HYDRAULIC fracturing ,COALBED methane ,GAS condensate reservoirs ,RESERVOIRS ,ANTHRACITE coal - Abstract
Coalbed methane (CBM), generated and stored in coalbeds, is a form of greenhouse gas. Three categories of the major hydraulic fractures were observed in the coal mines, consisting of vertical fractures, horizontal fractures, and "T"-type fractures. It is possible that the fracture monitored by surface detection techniques also has a non-zero flow conductivity, but the flow conductivity of this type fracture is more similar to that of a natural fracture than that of a propped fracture. [Extracted from the article]
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- 2020
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4. Creep Deformation Behavior of a Novel Precipitate-Hardened Ni-Fe-Base Superalloy at 750 °C.
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Zhang, P., Yuan, Y., Gu, Y. F., Dang, Y. Y., Yan, J. B., Lu, J. T., and Wang, J. C.
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HEAT resistant alloys ,HEAT treatment ,POWER plants ,CREEP (Materials) - Abstract
The creep deformation behavior of a novel precipitate-hardened Ni-Fe-base superalloy in solutionized and aged states is investigated at 750 °C/80 to 120 MPa. We found that as creep deformation proceeds, creep curves of specimens in the two states either overlap or are parallel to each other. This study provides a new strategy to design the heat treatment scheme to achieve a good compromise between the strength and fabricability of superalloys for 700 °C-class advanced ultra-supercritical power plants. [ABSTRACT FROM AUTHOR]
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- 2020
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5. Microstructure and Mechanical Properties of Welded Additively Manufactured Stainless Steels SS316L.
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Pasang, T., Kirchner, A., Jehring, U., Aziziderouei, M., Tao, Y., Jiang, C. -P., Wang, J. C., and Aisyah, I. S.
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Microstructure and mechanical properties of additively manufactured SS316L has been investigated. The samples produced by selective electron beam melting machine were then subjected to gas tungsten arc welding. Various examinations were performed including metallography and microscopy, hardness testing, and tensile testing coupled with digital image correlation software. Strain distribution was clearly evident on the samples during tensile testing with necking taking place at the heat affected zone on both sides of the weldments. From tensile testing, it was clear that the ductility and strengths of the samples were equal to those of conventionally produced samples such as rolled sheet. Hardness testing indicated the uniform distribution across the base metal and the weldments. Scanning electron microscopy identified the presence of Cr and Mo-rich precipitates on the grain boundaries, while the fracture surface was entirely covered with dimples (microvoid coalescence) indicating a ductile fracture mode. [ABSTRACT FROM AUTHOR]
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- 2019
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6. Synthesis, Characterization, and Crystal Structures of Oxidovanadium(V) Complexes Derived from 2-Chloro-N'-(3,5-dichloro-2-hydroxybenzylidene)benzohydrazide with Antimicrobial Activity.
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Wu, S. M., Qiu, X. Y., Wang, J. C., Liu, S. J., and He, L. Y.
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CRYSTAL structure ,UNIT cell ,STAPHYLOCOCCUS aureus ,ASPERGILLUS niger ,ESCHERICHIA coli ,SPACE groups - Abstract
Two new oxodovanadium(V) complexes, [VO(L)(OEt)(MeOH)] (I) and [VO(L)(Bha)] · EtOH (II), where L is the anion of 2-chloro-N'-(3,5-dichloro-2-hydroxybenzylidene)benzohydrazide (H
2 L), Bha is the anion of 2-hydroxybenzohydroxamic acid (HBha), were prepared and characterized by IR, UV-Vis and single crystal X-ray determination (CIF files CCDC nos. 1840661 (I) and 1840662 (II)). Complex I crystallizes as the monoclinic space group P21 /c with unit cell dimensions a = 8.272(1), b = 21.326(2), c = 12.979(1) Å, β = 107.173(2)°, V = 2187.5(4) Å3 , Z = 4, R1 = 0.0811, wR2 = 0.2152, GOOF = 1.048. Complex II crystallizes as the triclinic space group P with unit cell dimensions a = 7.407(2), b = 14.195(2), c = 14.330(2) Å, α = 117.262(2)°, β = 92.947(2)°, γ = 95.771(2)°, V = 1324.4(4) Å3 , Z = 2, R1 = 0.0919, wR2 = 0.1539, GOOF = 0.986. X-ray analysis indicates that the complexes are mononuclear vanadium(V) species, with the V atoms in octahedral coordination. The complexes were evaluated for their antibacterial (Bacillus subtilis, Staphylococcus aureus, Escherichia coli, and Pseudomonas fluorescence) and antifungal (Candida albicans and Aspergillus niger) activities by MTT (3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl tetrazolium bromide) method. The two complexes have from medium to strong activities against B. subtilis, S. aureus, and E. coli. [ABSTRACT FROM AUTHOR]- Published
- 2019
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7. Molecular-Based Modeling and Simulation Studies of Water-Water and Water-Macromolecule Interactions in Food and Their Effects on Food Dehydration.
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Wang, J.-C. and Liapis, A. I.
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- 2013
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8. A genome-wide association study of alcohol-dependence symptom counts in extended pedigrees identifies C15orf53.
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Wang, J-C, Foroud, T, Hinrichs, A L, Le, N X H, Bertelsen, S, Budde, J P, Harari, O, Koller, D L, Wetherill, L, Agrawal, A, Almasy, L, Brooks, A I, Bucholz, K, Dick, D, Hesselbrock, V, Johnson, E O, Kang, S, Kapoor, M, Kramer, J, and Kuperman, S
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GENEALOGY , *ALCOHOL-induced disorders , *SINGLE nucleotide polymorphisms , *ALCOHOLISM , *PHENOTYPES , *GENETICS - Abstract
Several studies have identified genes associated with alcohol-use disorders (AUDs), but the variation in each of these genes explains only a small portion of the genetic vulnerability. The goal of the present study was to perform a genome-wide association study (GWAS) in extended families from the Collaborative Study on the Genetics of Alcoholism to identify novel genes affecting risk for alcohol dependence (AD). To maximize the power of the extended family design, we used a quantitative endophenotype, measured in all individuals: number of alcohol-dependence symptoms endorsed (symptom count (SC)). Secondary analyses were performed to determine if the single nucleotide polymorphisms (SNPs) associated with SC were also associated with the dichotomous phenotype, DSM-IV AD. This family-based GWAS identified SNPs in C15orf53 that are strongly associated with DSM-IV alcohol-dependence symptom counts (P=4.5 × 10−8, inflation-corrected P=9.4 × 10−7). Results with DSM-IV AD in the regions of interest support our findings with SC, although the associations were less significant. Attempted replications of the most promising association results were conducted in two independent samples: nonoverlapping subjects from the Study of Addiction: Genes and Environment (SAGE) and the Australian Twin Family Study of AUDs (OZALC). Nominal association of C15orf53 with SC was observed in SAGE. The variant that showed strongest association with SC, rs12912251 and its highly correlated variants (D′=1, r2 0.95), have previously been associated with risk for bipolar disorder. [ABSTRACT FROM AUTHOR]
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- 2013
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9. Engineered human Tmpk fused with truncated cell-surface markers: versatile cell-fate control safety cassettes.
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Scaife, M, Pacienza, N, Au, B C Y, Wang, J C M, Devine, S, Scheid, E, Lee, C-J, Lopez-Perez, O, Neschadim, A, Fowler, D H, Foley, R, and Medin, J A
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GENETIC engineering ,HUMAN genetics ,GENETIC markers ,CELL transplantation ,PRODRUGS ,NERVE growth factor receptors - Abstract
Cell-fate control gene therapy (CFCGT)-based strategies can augment existing gene therapy and cell transplantation approaches by providing a safety element in the event of deleterious outcomes. Previously, we described a novel enzyme/prodrug combination for CFCGT. Here, we present results employing novel lentiviral constructs harboring sequences for truncated surface molecules (CD19 or low-affinity nerve growth factor receptor) directly fused to that CFCGT cDNA (TmpkF105Y). This confers an enforced one-to-one correlation between cell marking and eradication functions. In-vitro analysis demonstrated the full functionality of the fusion product. Next, low-dose 3′-azido-3′-deoxythymidine (AZT) administration to non-obese diabetic/severe combined immunodeficiency (NOD/SCID) mice injected with transduced clonal K562 cells suppressed tumor growth; furthermore, one integrated vector on average was sufficient to mediate cytotoxicity. Further, in a murine xenogeneic leukemia-lymphoma model we also demonstrated in-vivo control over transduced Raji cells. Finally, in a proof-of-principle study to examine the utility of this cassette in combination with a therapeutic cDNA, we integrated this novel CFCGT fusion construct into a lentivector designed for treatment of Fabry disease. Transduction with this vector restored enzyme activity in Fabry cells and retained AZT sensitivity. In addition, human Fabry patient CD34
+ cells showed high transduction efficiencies and retained normal colony-generating capacity when compared with the non-transduced controls. These collective results demonstrated that this novel and broadly applicable fusion system may enhance general safety in gene- and cell-based therapies. [ABSTRACT FROM AUTHOR]- Published
- 2013
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10. Bystander killing of malignant cells via the delivery of engineered thymidine-active deoxycytidine kinase for suicide gene therapy of cancer.
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Neschadim, A, Wang, J C M, Lavie, A, and Medin, J A
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GENE therapy , *CANCER treatment , *DEOXYCYTIDINE , *THYMIDINE , *GANCICLOVIR , *PRODRUGS , *HERPES simplex - Abstract
Activity and specificity of chemotherapeutic agents against solid tumors can be augmented via the targeted or localized delivery of 'suicide' genes. Selective activation of specific prodrugs in cells expressing the 'suicide' gene drives their elimination by apoptosis, while also enabling the killing of adjacent bystander cells. Strong bystander effects can compensate for poor 'suicide' gene delivery, and depend on the prodrugs used and mechanisms for the acquisition of activated drug by the bystander population, such as the presence of gap junctional intercellular communications. Although a number of 'suicide' gene therapies for cancer have been developed and characterized, such as herpes simplex virus-derived thymidine kinase (HSV-tk)-based activation of ganciclovir, their limited success highlights the need for the development of more robust approaches. Limiting activation kinetics and evolution of chemoresistance are major obstacles. Here we describe 'suicide' gene therapy of cancer based on the lentivirus-mediated delivery of a thymidine-active human deoxycytidine kinase variant. This enzyme possesses substrate plasticity that enables it to activate a multitude of prodrugs, some with distinct mechanisms of action. We evaluated the magnitude and mechanisms of bystander effects induced by different prodrugs, and show that when used in combination, they can synergistically enhance the bystander effect while avoiding off-target toxicity. [ABSTRACT FROM AUTHOR]
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- 2012
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11. ADH1B is associated with alcohol dependence and alcohol consumption in populations of European and African ancestry.
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Bierut, L J, Goate, A M, Breslau, N, Johnson, E O, Bertelsen, S, Fox, L, Agrawal, A, Bucholz, K K, Grucza, R, Hesselbrock, V, Kramer, J, Kuperman, S, Nurnberger, J, Porjesz, B, Saccone, N L, Schuckit, M, Tischfield, J, Wang, J C, Foroud, T, and Rice, J P
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ALCOHOL dehydrogenase ,ALCOHOL drinking ,DRUG abuse ,MENTAL illness ,AFRICAN Americans - Abstract
A coding variant in alcohol dehydrogenase 1B (ADH1B) (rs1229984) that leads to the replacement of Arg48 with His48 is common in Asian populations and reduces their risk for alcoholism, but because of very low allele frequencies the effects in European or African populations have been difficult to detect. We genotyped and analyzed this variant in three large European and African-American case-control studies in which alcohol dependence was defined by the Diagnostic and Statistical Manual of Mental Disorders, Fourth Edition (DSM-IV) criteria, and demonstrated a strong protective effect of the His48 variant (odds ratio (OR) 0.34, 95% confidence interval (CI) 0.24, 0.48) on alcohol dependence, with genome-wide significance (6.6 × 10
-10 ). The hypothesized mechanism of action involves an increased aversive reaction to alcohol; in keeping with this hypothesis, the same allele is strongly associated with a lower maximum number of drinks in a 24-hour period (lifetime), with P=3 × 10-13 . We also tested the effects of this allele on the development of alcoholism in adolescents and young adults, and demonstrated a significantly protective effect. This variant has the strongest effect on risk for alcohol dependence compared with any other tested variant in European populations. [ABSTRACT FROM AUTHOR]- Published
- 2012
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12. Differential immune responses mediated by adenovirus- and lentivirus-transduced DCs in a HER-2/neu overexpressing tumor model.
- Author
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Felizardo, T C, Wang, J C M, McGray, R A J, Evelegh, C, Spaner, D E, Fowler, D H, Bramson, J L, and Medin, J A
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IMMUNE response , *ADENOVIRUSES , *LENTIVIRUSES , *EPIDERMAL growth factor , *GENE expression , *CANCER immunotherapy , *DENDRITIC cells - Abstract
Recent investigations have demonstrated that adenoviral and lentiviral vectors encoding HER-2 can be utilized in cancer immunotherapy. However, it is not known whether both viral systems elicit a similar immune response. Here, we compare the immune response in mice induced by dendritic cells (DCs) infected with either recombinant adenovirus or lentivirus encoding rat HER-2 (rHER-2). Both vaccine types yielded similar control of tumor growth, but we found clear differences in their immune responses 10 days after DC immunization. Adenovirus rHER-2-transduced DCs elicited locally and systemically high frequencies of CD4+ and CD8+ T cells, while lentivirus rHER-2-transduced DCs predominantly led to CD4+ T-cell infiltration at the tumor site. Splenocytes from mice immunized with lentivirus rHER-2-transduced DCs secreted higher levels of interferon (IFN)-γ, mainly by CD4+ T cells, following stimulation by RM-1-mHER-2 tumors. In contrast, the adenovirus vaccinated group exhibited CD4+ and CD8+ T cells that both contributed to IFN-γ production. Besides an established cellular immune response, the rHER-2/DC vaccine elicited a significant humoral response that was highest in the adenovirus group. DC subsets and regulatory T cells in the spleen were also differentially modulated in the two vaccine systems. Finally, adoptive transfer of splenocytes from both groups of immunized mice strongly inhibited in vivo tumor growth. Our results suggest that not only the target antigen but also the virus system may determine the nature and magnitude of antitumor immunity by DC vaccination. [ABSTRACT FROM AUTHOR]
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- 2011
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13. A NOVEL FORMULA FOR EFFECTIVE THERMAL CONDUCTIVITY OF VAPOR CHAMBER.
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Wang, J.-C. and Wang, R.-T.
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THERMAL conductivity , *THERMOELECTRICITY , *VAPORS , *WORKING fluids , *EXPERIMENTS - Abstract
The article presents a study which evaluates the thermal performance of the vapor chamber-based thermal module in the thermal module industry. The study uses analytic model to investigate thermal conductivity, in which the copper meshes are bonded to the lower, upper, and around the internal surfaces to insure circulation of the working fluid. Conclusions show that thermal performance of the vapor-chamber-based thermal module is known within several seconds based on the deduced results.
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- 2011
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14. THE EFFECT OF VAPOR CHAMBER IN AN INJECTION MOLDING PROCESS ON PART TENSILE STRENGTH.
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Tsai, Y.-P., Wang, J.-C., and Hsu, R.-Q.
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INJECTION molding of plastics , *MOLDING of plastics , *EXPERIMENTAL design , *VAPOR pressure , *TEMPERATURE effect , *BLOCK designs - Abstract
The article discusses the effect of vapor chamber in an injection molding process on part tensile strength. An experimental setup was conducted on tensile test parts and multiholed plates which were test-molded utilizing the vapor chamber-based rapid heating and cooling system that was manufactured by Taiwan Microloops Corp. The study uses two different mold designs and a mold with one gate. Results show that the system can minimize the depth of the V-notch as much as 24 times.
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- 2011
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15. Genetic variation in the CHRNA5 gene affects mRNA levels and is associated with risk for alcohol dependence.
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Wang, J. C., Grucza, R., Cruchaga, C., Hinrichs, A. L., Bertelsen, S., Budde, J. P., Fox, L., Goldstein, E., Reyes, O., Saccone, N., Saccone, S., Xuei, X., Bucholz, K., Kuperman, S., Nurnberger, J., Rice, J. P., Schuckit, M., Tischfield, J., Hesselbrock, V., and Porjesz, B.
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SMOKING , *CIGARETTE smokers , *ALKALOIDS , *PEOPLE with alcoholism , *TOBACCO - Abstract
Alcohol dependence frequently co-occurs with cigarette smoking, another common addictive behavior. Evidence from genetic studies demonstrates that alcohol dependence and smoking cluster in families and have shared genetic vulnerability. Recently a candidate gene study in nicotine dependent cases and nondependent smoking controls reported strong associations between a missense mutation (rs16969968) in exon 5 of the CHRNA5 gene and a variant in the 3′-UTR of the CHRNA3 gene and nicotine dependence. In this study we performed a comprehensive association analysis of the CHRNA5–CHRNA3–CHRNB4 gene cluster in the Collaborative Study on the Genetics of Alcoholism (COGA) families to investigate the role of genetic variants in risk for alcohol dependence. Using the family-based association test, we observed that a different group of polymorphisms, spanning CHRNA5-CHRNA3, demonstrate association with alcohol dependence defined by Diagnostic and Statistical Manual of Mental Disorders, 4th edn (DSM-IV) criteria. Using logistic regression we replicated this finding in an independent case-control series from the family study of cocaine dependence. These variants show low linkage disequilibrium with the SNPs previously reported to be associated with nicotine dependence and therefore represent an independent observation. Functional studies in human brain reveal that the variants associated with alcohol dependence are also associated with altered steady-state levels of CHRNA5 mRNA.Molecular Psychiatry (2009) 14, 501–510; doi:10.1038/mp.2008.42; published online 15 April 2008 [ABSTRACT FROM AUTHOR]
- Published
- 2009
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16. A photonic-crystal polarizer integrated with the functions of narrow bandpass and narrow transmission-angle filtering.
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Mao, D., Ouyang, Z., Wang, J. C., Liu, C. P., and Wu, C. J.
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POLARIZERS (Light) ,PHOTONS ,CRYSTALS ,LIGHT filters ,ANISOTROPY ,OPTICS ,PHYSICS research - Abstract
Through the transfer matrix method, we demonstrated a special polarizer integrated with the functions of narrow bandpass (NBP) and narrow transmission-angle (NTA) filtering at normal incidence in a stack of two one-dimensional defective photonic crystals (PCs). One of the PCs is made of an isotropic dielectric media, while the other PC is made of anisotropic media. The key point in designing the stack is to set the central frequencies of the defect modes of the two sub-PCs to be the same for one polarization at normal incidence, but different for oblique incidence or for other polarizations, so that the stack structure could have the functions of three elements: a polarizer, an NBP filter, and an NTA filter. [ABSTRACT FROM AUTHOR]
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- 2008
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17. Colocalisation of CD9 and mortalin in CD9-induced mitotic catastrophe in human prostate cancer cells.
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Zvereff, V., Wang, J.-C., Shun, K., Lacoste, J., and Chevrette, M.
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PROSTATE cancer , *CANCER cells , *CELL cycle , *CELL communication , *MEDICAL research - Abstract
CD9, a member of the tetraspanin family of proteins, is involved in a variety of cellular interactions with many other proteins and molecules. Although CD9 has been implicated in cell fusion, migration and cancer progression, the detailed function of this protein is not completely understood and likely depends on interactions with different protein partners, which are not yet all known. Using co-immunoprecipitation and mass-spectrometric protein sequencing, we have identified in prostate cancer cells, a novel CD9 partner, the 75-kDa protein HSPA9B, also known as mortalin. We further show that introduction and overexpression of wild-type CD9 into human PC-3 prostate cancer cells induces mitotic catastrophe. We also demonstrate, by immunocolocalisation studies, the interaction of CD9 and mortalin in PC-3 cells undergoing mitotic catastrophe. Our results not only identified mortalin as a new CD9 partner, but also clarify the mechanisms by which CD9 may control prostate cancer progression.British Journal of Cancer (2007) 97, 941–948. doi:10.1038/sj.bjc.6603964 www.bjcancer.com Published online 11 September 2007 [ABSTRACT FROM AUTHOR]
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- 2007
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18. A strategy on constructing core collections by least distance stepwise sampling.
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Wang, J. C., Hu, J., Xu, H. M., and Zhang, S.
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MATHEMATICAL models , *PRINCIPAL components analysis , *GENOTYPE-environment interaction , *PLANT fibers , *GENETIC markers , *AGRICULTURAL statistics - Abstract
A strategy was proposed for constructing core collections by least distance stepwise sampling (LDSS) based on genotypic values. In each procedure of cluster, the sampling is performed in the subgroup with the least distance in the dendrogram during constructing a core collection. Mean difference percentage (MD), variance difference percentage (VD), coincidence rate of range (CR) and variable rate of coefficient of variation (VR) were used to evaluate the representativeness of core collections constructed by this strategy. A cotton germplasm collection of 1,547 accessions with 18 quantitative traits was used to construct core collections. Genotypic values of all quantitative traits of the cotton collection were unbiasedly predicted based on mixed linear model approach. By three sampling percentages (10, 20 and 30%), four genetic distances (city block distance, Euclidean distance, standardized Euclidean distance and Mahalanobis distance) combining four hierarchical cluster methods (nearest distance method, furthest distance method, unweighted pair-group average method and Ward’s method) were adopted to evaluate the property of this strategy. Simulations were conducted in order to draw consistent, stable and reproducible results. The principal components analysis was performed to validate this strategy. The results showed that core collections constructed by LDSS strategy had a good representativeness of the initial collection. As compared to the control strategy (stepwise clusters with random sampling strategy), LDSS strategy could construct more representative core collections. For LDSS strategy, cluster methods did not need to be considered because all hierarchical cluster methods could give same results completely. The results also suggested that standardized Euclidean distance was an appropriate genetic distance for constructing core collections in this strategy. [ABSTRACT FROM AUTHOR]
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- 2007
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19. Influence of initial casting temperature and dosage of fly ash on hydration heat evolution of concrete under adiabatic condition.
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Wang, J.-C. and Yan, P.-Y.
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FLY ash , *SLAG , *SILICA fume , *ADIABATIC invariants , *HEAT of hydration , *PORTLAND cement - Abstract
The calorimetric data of binders containing pure Portland cement, 20% fly ash, 20% slag and 10% silica fume respectively are determined at different initial casting temperatures using an adiabatic calorimeter to measure the adiabatic temperature rising of concrete. The calorimetric data of binders with different dosages of fly ash at two water binder ratios ( w/ b) are determined, too. Elevation of initial casting temperature decreases the heat evolution of binder, enhances the heat evolution rate of binder and increases the heat evolution rate of binder at early age. The dosage of fly ash in concrete has different effects on the heat evolution of binder with different w/ b. At high w/ b ratio the heat evolution of binder decreases when dosage of fly ash increases. At low w/ b ratio the heat evolution of binders increases when dosage of fly ash increases from 0 to 40% of total binder quantity. The heat evolution of binder decreases after the dosage of fly ash over 40%. An appropriate dosage of fly ash in binder benefits the performance of concrete at low w/ b ratio. [ABSTRACT FROM AUTHOR]
- Published
- 2006
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20. Direct evidence for cooperating genetic events in the leukemic transformation of normal human hematopoietic cells.
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Warner, J. K., Wang, J. C. Y., Takenaka, K., Doulatov, S., McKenzie, J. L., Harrington, L., and Dick, J. E.
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LEUKEMIA , *BLOOD diseases , *GENETIC disorders , *CANCER genetics , *HEMATOPOIESIS , *HEMATOPOIETIC agents , *TELOMERASE - Abstract
Although genetic abnormalities associated with hematological malignancies are readily identified, the natural history of human leukemia cannot be observed because initiating and subsequent transforming events occur before clinical presentation. Furthermore, it has not been possible to study leukemogenesis in vitro as normal human cells do not spontaneously transform. Thus, the nature and sequence of genetic changes required to convert human hematopoietic cells into leukemia cells have never been directly examined. We have developed a system where the first step in the leukemogenic process is an engineered disruption of differentiation and self-renewal due to expression of the TLS-ERG oncogene, followed in some cases by overexpression of hTERT. In two of 13 experiments, transduced cells underwent step-wise transformation and immortalization through spontaneous acquisition of additional changes. The acquired karyotypic abnormalities and alterations including upregulation of Bmi-1 and telomerase all occur in acute myeloid leukemia (AML), establishing the relevance of this system. One resultant cell line studied in depth exhibits cellular properties characteristic of AML, notably a hierarchical organization initiated by leukemic stem cells that differentiate abnormally. These findings provide direct evidence for multiple cooperating events in human leukemogenesis, and provide a foundation for studying the genetic changes that occur during leukemic initiation and progression. [ABSTRACT FROM AUTHOR]
- Published
- 2005
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21. Interocular asymmetry of visual field defects in primary open angle glaucoma and primary angle-closure glaucoma.
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Wang, J-C, Gazzard, G, Foster, P J, Devereux, J G, Oen, F T S, Chew, P T K, Khaw, P T, and Seah, S K L
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EYE examination , *VISUAL fields , *EYE diseases , *FLUOROURACIL , *CLINICAL trials ,GLAUCOMA surgery - Abstract
Aim To compare the interocular asymmetry in visual field loss of patients with primary open-angle (POAG) and primary angle-closure glaucoma (PACG).Methods Subjects entering a prospective, randomised, controlled trial of intraoperative 5-fluorouracil in glaucoma surgery in Singapore were included. Preoperative visual field testing was performed using automated white-on-white perimetry (24-2 test pattern, threshold program, Mk II, Model 750, Zeiss-Humphrey, San Leandro, CA, USA). A minimum of two tests were required with mean deviation within 2?dB on two tests, fixation losses <20%, false positives <33%, and false negatives <33%. The second field was scored using AGIS II criteria and the ‘mean asymmetry score’ defined as the mean difference between eyes for both AGIS scores and global indices.Results In 230 subjects assessed (128 POAG, 102 PACG), mean interocular asymmetry of visual field loss was greater for the PACG group. The mean AGIS asymmetry scores for total (PACG=9.21±6.87 vs POAG=6.48±5.58, P=0.001), superior (PACG=4.31±3.39 vs POAG=3.35±3.13, P=0.035), and inferior (PACG=4.43±3.31 vs POAG=2.64±2.77, P<0.0001) areas and mean deviation (MD) asymmetry scores (PACG=6.89±13.22 vs POAG=1.66±16.97, P=0.012) were all significantly different. Interocular correlation of visual field loss for POAG was significant; total AGIS, r=0.27 (P=0.003), superior field AGIS, r=0.24 (P=0.008), inferior field AGIS, r=0.34 (P=0.0001), and MD, r=0.27 (P=0.003). In PACG, there was no significant correlation between eyes; total AGIS, r=-0.02 (P=0.85), superior field AGIS, r=-0.02 (P=0.82), inferior field AGIS, r=-0.17 (P=0.87), and MD, r=0.015 (P=0.89).Conclusion There was a greater asymmetry of visual field loss between eyes, as measured by AGIS scores and MD, in PACG than that in POAG.Eye (2004) 18, 365-368. doi:10.1038/sj.eye.6700664 [ABSTRACT FROM AUTHOR]
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- 2004
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22. Reinvestigation of the beta-decay of 110Mo.
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Wang, J. C., Dendooven, P., Hankonen, S., Huikari, J., Jokinen, A., Kolhinen, V. S., Lhersonneau, G., Nieminen, A., Peräjärvi, K., Rinta-Antila, S., and Äystö, J.
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NUCLEAR fission , *NUCLEAR reactions , *PARTICLES (Nuclear physics) , *NUCLEAR physics , *RADIOACTIVE substances , *COINCIDENCE - Abstract
The beta-decay of the neutron-rich nucleus 110Mo, separated by the IGISOL on-line mass separator from other fission products, has been investigated by using beta-gamma and gamma-gamma coincidence techniques. The decay scheme of 110Mo has been revised, including 3 new excited states and 7 new γ transitions in 110Tc. The β-feedings were measured and log ft values and B(GT) values were deduced based on a Qβ-value from systematics. Three excited 1+ states in 110Tc fed by spin-flip allowed-unhindered beta transitions were identified. The deduced beta-decay strengths are compared with the Gamow-Teller strength distribution obtained from a macroscopic-microscopic calculation. The role of the asymptotic quantum numbers in the context of the allowed beta-decay is discussed. [ABSTRACT FROM AUTHOR]
- Published
- 2004
- Full Text
- View/download PDF
23. Induced recovery of defective membrane expression of a CC chemokine receptor 5 mutant by phytohemagglutinin.
- Author
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Qi, Q. -Y., Wang, F., Zhang, H. -T., Wang, J. -C., Xiao, H. -P, Wang, M. -H., Han, Y. -F., Zhang, R. -M., Tao, S. -H., and Luo, Z. W.
- Subjects
CHEMOKINES ,CYTOKINES ,IMMUNODEFICIENCY ,PHYTOHEMAGGLUTININS ,MUCOPROTEINS - Abstract
CC chemokine receptor 5 (CCR5) is a member of the G-protein-coupled receptor superfamily. It plays an important role in macrophage tropic human immunodeficiency virus-1 entry and in some inflammatory reactions. CCR5-893(–) is a single-nucleotide deletion that results in complete truncation of the C tail of the gene product. We detected CCR5-893(–) in a sample of patients infected with non-tuberculosis mycobacteria and found that it was maintained heterozygously with a frequency of 2%. There is no association between this mutation and any immunodeficiency. Membrane expression of CCR5-893(–) was substantially reduced compared to the wild type, but this defective surface presentation recovered greatly recovered in the presence of 2 mg l-1 phytohemagglutinin (PHA). However, PHA inducement did not affect the total intracellular expression of CCR5-893(–) or wild-type CCR5. Thus we suggest there exist some PHA-induced factor(s) that could mediate the presentation of truncated CCR5. [ABSTRACT FROM AUTHOR]
- Published
- 2003
- Full Text
- View/download PDF
24. Choriocarcinoma of the gallbladder: treated with cisplatin-based chemotherapy.
- Author
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Wang, J C, Angeles, S, Chak, P, Platt, A B, and Nimmagadda, N
- Abstract
A case of non-gestational choriocarcinoma, primary in the gallbladder, is described. Pathology showed a mixture of adenocarcinoma and choriocarcinoma. A Medline search for the last 10 years has shown no similar cases reported. The use of cisplatin-based chemotherapy to treat non-gestational choriocarcinoma has also not been described. This patient showed a rapidly progressive disease as described in other reported cases of non-gestational choriocarcinoma. However, cisplatin-based chemotherapy, in this patient, produced a partial response defined as significantly decreased beta-HCG titers (more than 50%), and with stable appearance or slightly improvement of hepatic metastasis and survival for 1 yr. [ABSTRACT FROM AUTHOR]
- Published
- 2001
25. Atypical adenomatous hyperplasia of the lung: correlation between high-resolution CT findings and histopathologic features.
- Author
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Kawakami, S., Sone, S., Takashima, S., Li, F., Yang, Z.-G., Maruyama, Y., Honda, T., Hasegawa, M., and Wang, J.-C.
- Subjects
HYPERPLASIA ,CELLULAR pathology ,GINGIVAL hyperplasia ,BENIGN prostatic hyperplasia ,HISTOPATHOLOGY ,CELL proliferation - Abstract
We describe herein the CT features of atypical adenomatous hyperplasia (AAH) of the lung and its histopathological characteristics. Among 17,919 individuals screened for lung cancer by CT scanning, ten AAH nodules were detected in nine asymptomatic subjects. On high-resolution CT, the lesions measured from 6 x 6 mm to 15 x 17 mm and their CT number ranged from -500 to -760 HU. The AAHs appeared as round nodules with smooth and distinct borders and showed a ground-glass opacity. Plain chest radiographs failed to identify all lesions. Histopathologically, AAH lesions showed atypical epithelial cell proliferation along slightly thickened alveolar septa. Whereas it is often easy to differentiate these nodules from inflammatory and benign lung lesions, histopathological examination remains at present the only method to differentiate AAH from lung cancers. [ABSTRACT FROM AUTHOR]
- Published
- 2001
- Full Text
- View/download PDF
26. On Line Model Accuracy Inspection of Model Maker Rapid Prototyping Using Vision Technology.
- Author
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Jeng, J.-Y., Wang, J.-C., Lin, T. T., and Yang, W. P.
- Published
- 2001
- Full Text
- View/download PDF
27. The Application of the Skipped Layer Interior Method and Higher Depostition Height to Accelerate Fabrication Speed of the Model Maker Rapid Prototyping System.
- Author
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Jeng, J.-Y., Wang, J.-C., and Lin, T.T.
- Published
- 2001
- Full Text
- View/download PDF
28. Effects of JTV-519, a novel anti-ischaemic drug, on the delayed rectifier K+ current in guinea-pig ventricular myocytes.
- Author
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Kiriyama, K., Kiyosue, T., Wang, J.-C., Dohi, K., and Arita, M.
- Subjects
ISCHEMIA ,ANTIHYPERTENSIVE agents ,CALCIUM antagonists ,GUINEA pigs as laboratory animals ,NITRENDIPINE ,MUSCLE cells - Abstract
We studied the effects of a newly synthesized anti-ischaemic agent, 4-[3-(4-benzylpiperidin-1-yl) propionyl]-7-methoxy-2, 3, 4, 5-tetrahydro-1, 4-benzothiazepine monohydrochloride (JTV-519) on the delayed rectifier potassium current (I
K ), using guinea-pig ventricular myocytes and whole-cell voltage-clamp techniques, under blockade of the L-type calcium current (ICa,L ) by D600 (1 µM) or nitrendipine (5 µM). The IK in guinea-pig ventricular cells consists of two different components; the rapidly activating, E4031-sensitive component (IKr ) and the slowly activating E4031-resistant component (IKs ). Under steady-state conditions, JTV-519 (1 and 5 µM) did not change the amplitude of IKs remaining after blockade of IKr with 5 µM E4031. The effect of JTV-519 on IKr was assessed using short (50 ms) pulses which evoked a tail current that was sensitive to E4031 but not to chromanol 293B, a specific blocker of IKs . JTV-519 suppressed the IKr with a half-maximal inhibitory concentration of 1.2 µM. Selective inhibition of IKr by this agent was confirmed by using the "envelope of tails" test. These results suggest that the blockade of IKr may underlie the prolongation of action potential duration in ventricular muscle and QT-intervals alleged to occur in animal as well as human hearts. [ABSTRACT FROM AUTHOR]- Published
- 2000
- Full Text
- View/download PDF
29. Cytokine treatment or accessory cells are required to initiate engraftment of purified primitive human hematopoietic cells transplanted at limiting doses into NOD/SCID mice.
- Author
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Bonnet, D, Bhatia, M, Wang, J C Y, Kapp, U, and Dick, J E
- Subjects
ANTIGEN presenting cells ,HEMATOPOIETIC stem cell transplantation ,CYTOKINES - Abstract
Little is known about the cell types or mechanisms that underlie the engraftment process. Here, we have examined parameters affecting the engraftment of purified human Lin
- CD34+ CD38- normal and AML cells transplanted at limiting doses into NOD/SCID recipients. Mice transplanted with 500 to 1000 Lin- CD34+ CD38- cord blood (CB) or AML cells required the co-transplantation of accessory cells (ACs) or short-term in vivo cytokine treatment for engraftment, whereas transplantation of higher doses (>5000 Lin- CD34+ CD38- cells) did not show these requirements suggesting that ACs are effective for both normal and leukemic stem cell engraftment in this model. Mature Lin+ CD34- and primitive Lin- CD34+ CD38+ cells were capable of acting as ACs even though no repopulating cells are present. Cytokine treatment of NOD/SCID mice could partially replace the requirement for co-transplantation of AC. Furthermore, no difference was seen between the percentage of engrafted mice treated with cytokines for only the first 10 days after transplant compared to those receiving cytokines for the entire time of repopulation. Surprisingly, no engraftment was detected in mice when cytokine treatment was delayed until 10 days post- transplant. Together, these studies suggest that the engraftment process requires pluripotent stem cells plus accessory cells or cytokine treatment which act early after transplantation. The NOD/SCID xenotransplant system provides the means to further clarify the processes underlying human stem cell engraftment. [ABSTRACT FROM AUTHOR]- Published
- 1999
- Full Text
- View/download PDF
30. Cloning, sequence analysis, and expression in Escherichia coli of the gene encoding phenylacetaldehyde reductase from styrene-assimilating Corynebacterium sp. strain ST-10.
- Author
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Wang, J.-C., Sakakibara, M., Liu, J.-Q., Dairi, T., and Itoh, N.
- Abstract
The gene encoding phenylacetaldehyde reductase (PAR), a useful biocatalyst for producing chiral alcohols, was cloned from the genomic DNA of the styrene-assimilating Corynebacterium sp. strain ST-10. The gene contained an opening reading frame consisting of 1,158 nucleotides corresponding to 385 amino acid residues. The subunit molecular weight was calculated to be 40,299, which was in agreement with that determined by polyacrylamide gel electrophoresis. The enzyme was sufficiently expressed in recombinant Escherichia coli cells for practical use and purified to homogeneity by three-column chromatography steps. The predicted amino acid sequence displayed only 20–29% identity with zinc-containing, NAD
+ -dependent, long-chain alcohol dehydrogenases. Nevertheless, the probable NAD+ - and zinc-binding sites are conserved although one of the three catalytic zinc-binding residues of the zinc-containing, long-chain alcohol dehydrogenases was substituted by Asp in PAR. The protein contains 7.6 mol zinc/mol tetramer. Therefore, the enzyme was considered as a new member of zinc-containing, long-chain alcohol dehydrogenases with a particular and broad substrate specificity. [ABSTRACT FROM AUTHOR]- Published
- 1999
- Full Text
- View/download PDF
31. Analysis of TSG101 tumour susceptibility gene transcripts in cervical and endometrial cancers.
- Author
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Chang, J-G, Su, T-H, Wei, H-J, Wang, J-C, Chen, Y-J, Chang, C-P, and Jeng, C-J
- Subjects
HEAD & neck cancer ,PELVIS cancer ,GENES ,TUMOR susceptibility gene 101 - Abstract
Carcinoma of the uterine cervix is a common malignancy among women that has been found to show loss of heterozygosity in the chromosome 11p. Recent studies have localized theTSG101 gene in this region, and also demonstrated a high frequency of abnormalities of this gene in human breast cancer. To determine the role of theTSG101 gene in the carcinogenesis of cervical and uterine carcinoma, 19 cases of cervical carcinoma and five cases of endometrial carcinoma, as well as nearby non-cancerous tissue from the same patients, and 16 blood samples from healthy persons as normal control were analysed by Southern blot analysis of genomic DNA, reverse transcription of the TSG101 mRNA followed by PCR amplification and sequencing of the products. We found that abnormal transcripts of theTSG101 gene were common both in cancerous or non-cancerous tissues of the uterus and cervix and in normal peripheral mononuclear cells. There was no genomic deletion or rearrangement in spite of the presence of abnormal transcripts, and no definite relationship between the abnormal transcripts and HPV infection was found. Although the frequency of abnormal transcripts was higher in cancerous than in non-cancerous tissue, normal peripheral mononuclear cells also had abnormal transcripts. Given these findings, the role of theTSG101 gene as a tumour-suppressor gene should be re-evaluated. Because some aberrant transcripts could be found at the first PCR reaction, we suggest that the aberrant transcripts might be the result of imperfect minor splicesome products. [ABSTRACT FROM AUTHOR]
- Published
- 1999
- Full Text
- View/download PDF
32. Blood thrombopoietin, IL-6 and IL-11 levels in patients with agnogenic myeloid metaplasia.
- Author
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Wang, J C, Chen, C, Lou, L-H, and Mora, M
- Subjects
- *
MYELOID metaplasia , *BONE marrow diseases , *MEGAKARYOCYTES , *HYPERPLASIA - Abstract
Agnogenic myeloid metaplasia (AMM) is a disease characterized by bone marrow megakaryocyte hyperplasia and clusters of megakaryocytes, in which many of the megakaryocytes are atypical. In order to elucidate the mechanisms of megakaryocytosis, ELISA assays of blood levels of thrombopoietin (TPO), interleukin-6 (IL-6) and interleukin-11 (IL-11) were done in 45 patients with AMM and compared with normal volunteer controls. Higher blood TPO levels were found in AMM than in controls (P < 0.0001), and blood TPO levels were correlated with the degree of marrow fibrosis (P = 0.0078). Blood levels of IL-6 were also significantly higher in AMM, when compared with controls (P < 0.0001). However, no correlation was found between blood IL-6 levels and degree of marrow fibrosis. No correlation was found between either TPO or IL-6 and the number of blood platelet counts, the number of marrow megakaryocytes, WBC counts, or the degree of splenomegaly. Blood IL-11 levels were undetectable in most patients and no significant difference was found in AMM as compared to controls. The present study demonstrated that, while in idiopathic thrombocytopenic purpura (ITP) or aplastic anemia, blood TPO levels are relatively correlated with the numbers of platelet and/or megakaryocyte mass, blood TPO levels do not correlate with blood platelet counts, or marrow megakaryocyte mass in AMM. Therefore, in AMM, other mechanisms such as the number of TPO receptors on platelets or megakaryocytes, c-MPL receptor abnormalities, abnormal production of TPO mRNA and so on, will have to be studied. Furthermore, TPO may play a significant role in the pathogenesis of marrow fibrosis; IL-6 may be a factor in the development of marrow megakaryocytosis but its elevated blood levels may represent a secondary immune phenomenon; and IL-11 probably does not play a significant role in causing marrow megakaryocytosis in this disease. [ABSTRACT FROM AUTHOR]
- Published
- 1997
- Full Text
- View/download PDF
33. Utilization and dosing of angiotensin-converting enzyme inhibitors for heart failure. Effect of physician specialty and patient characteristics.
- Author
-
Chin, Marshall, Wang, John, Žhang, James, Lang, Roberto, Chin, M H, Wang, J C, Zhang, J X, and Lang, R M
- Abstract
To determine if physician specialty is associated with underutilization and underdosing of angiotensin-converting enzyme inhibitors among patients with heart failure, we reviewed the charts of 214 outpatients with decreased systolic function at an urban academic medical center. Regardless of whether patients were cared for by cardiologists, generalist physicians, or a combination of the two specialities, approximately 75% of the patients were taking an angiotensin-converting enzyme inhibitor. However, only approximately 60% of these patients were taking dosages proved to be efficacious in trials. Emphasis on adequate dosing is needed among all specialty groups. [ABSTRACT FROM AUTHOR]
- Published
- 1997
- Full Text
- View/download PDF
34. High levels of loss at the 17p telomere suggest the close proximity of a tumour suppressor.
- Author
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White, GRM, Stack, M, Santibáñez-Koref, M, Liscia, DS, Venesio, T, Wang, J-C, Helms, C, Donis-Keller, H, Betticher, DC, Altermatt, HJ, Hoban, PR, Heighway, J, White, G R, Santibáñez-Koref, M, Liscia, D S, Betticher, D C, Altermatt, H J, and Hoban, P R
- Published
- 1996
- Full Text
- View/download PDF
35. Measurement of τ polarization in W→τν decays with the ATLAS detector in pp collisions at $\sqrt{s} =7~\mathrm{TeV}$
- Author
-
Aad, G., Abbott, B., Abdallah, J., Abdel Khalek, S., Abdelalim, A. A., Abdesselam, A., Abdinov, O., Abi, B., Abolins, M., AbouZeid, O. S., Abramowicz, H., Abreu, H., Acerbi, E., Acharya, B. S., Adamczyk, L., Adams, D. L., Addy, T. N., Adelman, J., Aderholz, M., Adomeit, S., Adragna, P., Adye, T., Aefsky, S., Aguilar-Saavedra, J. A., Aharrouche, M., Ahlen, S. P., Ahles, F., Ahmad, A., Ahsan, M., Aielli, G., Akdogan, T., Åkesson, T. P. A., Akimoto, G., Akimov, A. V., Akiyama, A., Alam, M. S., Alam, M. A., Albert, J., Albrand, S., Aleksa, M., Aleksandrov, I. N., Alessandria, F., Alexa, C., Alexander, G., Alexandre, G., Alexopoulos, T., Alhroob, M., Aliev, M., Alimonti, G., Alison, J., Aliyev, M., Allbrooke, B. M. M., Allport, P. P., Allwood-Spiers, S. E., Almond, J., Aloisio, A., Alon, R., Alonso, A., Alvarez Gonzalez, B., Alviggi, M. G., Amako, K., Amaral, P., Amelung, C., Ammosov, V. V., Amorim, A., Amorós, G., Amram, N., Anastopoulos, C., Ancu, L. S., Andari, N., Andeen, T., Anders, C. F., Anders, G., Anderson, K. J., Andreazza, A., Andrei, V., Andrieux, M-L., Anduaga, X. S., Angerami, A., Anghinolfi, F., Anisenkov, A., Anjos, N., Annovi, A., Antonaki, A., Antonelli, M., Antonov, A., Antos, J., Anulli, F., Aoun, S., Aperio Bella, L., Apolle, R., Arabidze, G., Aracena, I., Arai, Y., Arce, A. T. H., Arfaoui, S., Arguin, J-F., Arik, E., Arik, M., Armbruster, A. J., Arnaez, O., Arnal, V., Arnault, C., Artamonov, A., Artoni, G., Arutinov, D., Asai, S., Asfandiyarov, R., Ask, S., Åsman, B., Asquith, L., Assamagan, K., Astbury, A., Aubert, B., Auge, E., Augsten, K., Aurousseau, M., Avolio, G., Avramidou, R., Axen, D., Ay, C., Azuelos, G., Azuma, Y., Baak, M. A., Baccaglioni, G., Bacci, C., Bach, A. M., Bachacou, H., Bachas, K., Backes, M., Backhaus, M., Badescu, E., Bagnaia, P., Bahinipati, S., Bai, Y., Bailey, D. C., Bain, T., Baines, J. T., Baker, O. K., Baker, M. D., Baker, S., Banas, E., Banerjee, P., Banerjee, Sw., Banfi, D., Bangert, A., Bansal, V., Bansil, H. S., Barak, L., Baranov, S. P., Barashkou, A., Barbaro Galtieri, A., Barber, T., Barberio, E. L., Barberis, D., Barbero, M., Bardin, D. Y., Barillari, T., Barisonzi, M., Barklow, T., Barlow, N., Barnett, B. M., Barnett, R. M., Baroncelli, A., Barone, G., Barr, A. J., Barreiro, F., Barreiro Guimarães da Costa, J., Barrillon, P., Bartoldus, R., Barton, A. E., Bartsch, V., Bates, R. L., Batkova, L., Batley, J. R., Battaglia, A., Battistin, M., Bauer, F., Bawa, H. S., Beale, S., Beau, T., Beauchemin, P. H., Beccherle, R., Bechtle, P., Beck, H. P., Becker, S., Beckingham, M., Becks, K. H., Beddall, A. J., Beddall, A., Bedikian, S., Bednyakov, V. A., Bee, C. P., Begel, M., Behar Harpaz, S., Behera, P. K., Beimforde, M., Belanger-Champagne, C., Bell, P. J., Bell, W. H., Bella, G., Bellagamba, L., Bellina, F., Bellomo, M., Belloni, A., Beloborodova, O., Belotskiy, K., Beltramello, O., Benary, O., Benchekroun, D., Bendel, M., Bendtz, K., Benekos, N., Benhammou, Y., Benhar Noccioli, E., Benitez Garcia, J. A., Benjamin, D. P., Benoit, M., Bensinger, J. R., Benslama, K., Bentvelsen, S., Berge, D., Bergeaas Kuutmann, E., Berger, N., Berghaus, F., Berglund, E., Beringer, J., Bernat, P., Bernhard, R., Bernius, C., Berry, T., Bertella, C., Bertin, A., Bertinelli, F., Bertolucci, F., Besana, M. I., Besson, N., Bethke, S., Bhimji, W., Bianchi, R. M., Bianco, M., Biebel, O., Bieniek, S. P., Bierwagen, K., Biesiada, J., Biglietti, M., Bilokon, H., Bindi, M., Binet, S., Bingul, A., Bini, C., Biscarat, C., Bitenc, U., Black, K. M., Blair, R. E., Blanchard, J.-B., Blanchot, G., Blazek, T., Blocker, C., Blocki, J., Blondel, A., Blum, W., Blumenschein, U., Bobbink, G. J., Bobrovnikov, V. B., Bocchetta, S. S., Bocci, A., Boddy, C. R., Boehler, M., Boek, J., Boelaert, N., Bogaerts, J. A., Bogdanchikov, A., Bogouch, A., Bohm, C., Bohm, J., Boisvert, V., Bold, T., Boldea, V., Bolnet, N. M., Bomben, M., Bona, M., Bondarenko, V. G., Bondioli, M., Boonekamp, M., Booth, C. N., Bordoni, S., Borer, C., Borisov, A., Borissov, G., Borjanovic, I., Borri, M., Borroni, S., Bortolotto, V., Bos, K., Boscherini, D., Bosman, M., Boterenbrood, H., Botterill, D., Bouchami, J., Boudreau, J., Bouhova-Thacker, E. V., Boumediene, D., Bourdarios, C., Bousson, N., Boveia, A., Boyd, J., Boyko, I. R., Bozhko, N. I., Bozovic-Jelisavcic, I., Bracinik, J., Braem, A., Branchini, P., Brandenburg, G. W., Brandt, A., Brandt, G., Brandt, O., Bratzler, U., Brau, B., Brau, J. E., Braun, H. M., Brelier, B., Bremer, J., Brendlinger, K., Brenner, R., Bressler, S., Britton, D., Brochu, F. M., Brock, I., Brock, R., Brodbeck, T. J., Brodet, E., Broggi, F., Bromberg, C., Bronner, J., Brooijmans, G., Brooks, W. K., Brown, G., Brown, H., Bruckman de Renstrom, P. A., Bruncko, D., Bruneliere, R., Brunet, S., Bruni, A., Bruni, G., Bruschi, M., Buanes, T., Buat, Q., Bucci, F., Buchanan, J., Buchholz, P., Buckingham, R. M., Buckley, A. G., Buda, S. I., Budagov, I. A., Budick, B., Büscher, V., Bugge, L., Bulekov, O., Bundock, A. C., Bunse, M., Buran, T., Burckhart, H., Burdin, S., Burgess, T., Burke, S., Busato, E., Bussey, P., Buszello, C. P., Butin, F., Butler, B., Butler, J. M., Buttar, C. M., Butterworth, J. M., Buttinger, W., Cabrera Urbán, S., Caforio, D., Cakir, O., Calafiura, P., Calderini, G., Calfayan, P., Calkins, R., Caloba, L. P., Caloi, R., Calvet, D., Calvet, S., Camacho Toro, R., Camarri, P., Cambiaghi, M., Cameron, D., Caminada, L. M., Campana, S., Campanelli, M., Canale, V., Canelli, F., Canepa, A., Cantero, J., Capasso, L., Capeans Garrido, M. D. M., Caprini, I., Caprini, M., Capriotti, D., Capua, M., Caputo, R., Cardarelli, R., Carli, T., Carlino, G., Carminati, L., Caron, B., Caron, S., Carquin, E., Carrillo Montoya, G. D., Carter, A. A., Carter, J. R., Carvalho, J., Casadei, D., Casado, M. P., Cascella, M., Caso, C., Castaneda Hernandez, A. M., Castaneda-Miranda, E., Castillo Gimenez, V., Castro, N. F., Cataldi, G., Catastini, P., Catinaccio, A., Catmore, J. R., Cattai, A., Cattani, G., Caughron, S., Cauz, D., Cavalleri, P., Cavalli, D., Cavalli-Sforza, M., Cavasinni, V., Ceradini, F., Cerqueira, A. S., Cerri, A., Cerrito, L., Cerutti, F., Cetin, S. A., Cevenini, F., Chafaq, A., Chakraborty, D., Chalupkova, I., Chan, K., Chapleau, B., Chapman, J. D., Chapman, J. W., Chareyre, E., Charlton, D. G., Chavda, V., Chavez Barajas, C. A., Cheatham, S., Chekanov, S., Chekulaev, S. V., Chelkov, G. A., Chelstowska, M. A., Chen, C., Chen, H., Chen, S., Chen, T., Chen, X., Cheng, S., Cheplakov, A., Chepurnov, V. F., Cherkaoui El Moursli, R., Chernyatin, V., Cheu, E., Cheung, S. L., Chevalier, L., Chiefari, G., Chikovani, L., Childers, J. T., Chilingarov, A., Chiodini, G., Chisholm, A. S., Chislett, R. T., Chizhov, M. V., Choudalakis, G., Chouridou, S., Christidi, I. A., Christov, A., Chromek-Burckhart, D., Chu, M. L., Chudoba, J., Ciapetti, G., Ciftci, A. K., Ciftci, R., Cinca, D., Cindro, V., Ciocca, C., Ciocio, A., Cirilli, M., Citterio, M., Ciubancan, M., Clark, A., Clark, P. J., Cleland, W., Clemens, J. C., Clement, B., Clement, C., Coadou, Y., Cobal, M., Coccaro, A., Cochran, J., Coe, P., Cogan, J. G., Coggeshall, J., Cogneras, E., Colas, J., Colijn, A. P., Collins, N. J., Collins-Tooth, C., Collot, J., Colon, G., Conde Muiño, P., Coniavitis, E., Conidi, M. C., Consonni, M., Consonni, S. M., Consorti, V., Constantinescu, S., Conta, C., Conti, G., Conventi, F., Cook, J., Cooke, M., Cooper, B. D., Cooper-Sarkar, A. M., Copic, K., Cornelissen, T., Corradi, M., Corriveau, F., Cortes-Gonzalez, A., Cortiana, G., Costa, G., Costa, M. J., Costanzo, D., Costin, T., Côté, D., Courneyea, L., Cowan, G., Cowden, C., Cox, B. E., Cranmer, K., Crescioli, F., Cristinziani, M., Crosetti, G., Crupi, R., Crépé-Renaudin, S., Cuciuc, C.-M., Cuenca Almenar, C., Cuhadar Donszelmann, T., Cummings, J., Curatolo, M., Curtis, C. J., Cuthbert, C., Cwetanski, P., Czirr, H., Czodrowski, P., Czyczula, Z., D’Auria, S., D’Onofrio, M., D’Orazio, A., Da Silva, P. V. M., Da Via, C., Dabrowski, W., Dafinca, A., Dai, T., Dallapiccola, C., Dam, M., Dameri, M., Damiani, D. S., Danielsson, H. O., Dannheim, D., Dao, V., Darbo, G., Darlea, G. L., Davey, W., Davidek, T., Davidson, N., Davidson, R., Davies, E., Davies, M., Davison, A. R., Davygora, Y., Dawe, E., Dawson, I., Dawson, J. W., Daya-Ishmukhametova, R. K., De, K., de Asmundis, R., De Castro, S., De Castro Faria Salgado, P. E., De Cecco, S., de Graat, J., De Groot, N., de Jong, P., De La Taille, C., De la Torre, H., De Lorenzi, F., De Lotto, B., de Mora, L., De Nooij, L., De Pedis, D., De Salvo, A., De Sanctis, U., De Santo, A., De Vivie De Regie, J. B., De Zorzi, G., Dean, S., Dearnaley, W. J., Debbe, R., Debenedetti, C., Dechenaux, B., Dedovich, D. V., Degenhardt, J., Del Papa, C., Del Peso, J., Del Prete, T., Delemontex, T., Deliyergiyev, M., Dell’Acqua, A., Dell’Asta, L., Della Pietra, M., della Volpe, D., Delmastro, M., Delruelle, N., Delsart, P. A., Deluca, C., Demers, S., Demichev, M., Demirkoz, B., Deng, J., Denisov, S. P., Derendarz, D., Derkaoui, J. E., Derue, F., Dervan, P., Desch, K., Devetak, E., Deviveiros, P. O., Dewhurst, A., DeWilde, B., Dhaliwal, S., Dhullipudi, R., Di Ciaccio, A., Di Ciaccio, L., Di Girolamo, A., Di Girolamo, B., Di Luise, S., Di Mattia, A., Di Micco, B., Di Nardo, R., Di Simone, A., Di Sipio, R., Diaz, M. A., Diblen, F., Diehl, E. B., Dietrich, J., Dietzsch, T. 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G., Zhu, H., Zhu, J., Zhu, Y., Zhuang, X., Zhuravlov, V., Zieminska, D., Zimmermann, R., Zimmermann, S., Ziolkowski, M., Zitoun, R., Živković, L., Zmouchko, V. V., Zobernig, G., Zoccoli, A., zur Nedden, M., Zutshi, V., and Zwalinski, L.
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Physics ,Particle physics ,Large Hadron Collider ,Physics and Astronomy (miscellaneous) ,010308 nuclear & particles physics ,Atlas detector ,Hadron ,ATLAS experiment ,Astrophysics::Cosmology and Extragalactic Astrophysics ,Polarization (waves) ,01 natural sciences ,Charged particle ,Nuclear physics ,Pair production ,0103 physical sciences ,High Energy Physics::Experiment ,010306 general physics ,Nuclear Experiment ,Engineering (miscellaneous) - Abstract
In this paper, a measurement of tau polarization in W->taunu decays is presented. It is measured from the energies of the decay products in hadronic tau decays with a single final state charged particle. The data, corresponding to an integrated luminosity of 24 pb^-1, were collected by the ATLAS experiment at the Large Hadron Collider in 2010. The measured value of the tau polarization is Ptau = -1.06 +/- 0.04 (stat) + 0.05 (syst) - 0.07 (syst), in agreement with the Standard Model prediction, and is consistent with a physically allowed 95% CL interval [-1,-0.91]. Measurements of tau polarization have not previously been made at hadron colliders.
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36. Measurement of the top quark mass with the template method in the $t\bar{t} \to\mathrm{lepton}+\mathrm{jets}$ channel using ATLAS data
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Aad, G., Abbott, B., Abdallah, J., Abdelalim, A. A., Abdesselam, A., Abdinov, O., Abi, B., Abolins, M., AbouZeid, O. S., Abramowicz, H., Abreu, H., Acerbi, E., Acharya, B. S., Adamczyk, L., Adams, D. L., Addy, T. N., Adelman, J., Aderholz, M., Adomeit, S., Adragna, P., Adye, T., Aefsky, S., Aguilar-Saavedra, J. A., Aharrouche, M., Ahlen, S. P., Ahles, F., Ahmad, A., Ahsan, M., Aielli, G., Akdogan, T., Åkesson, T. P. A., Akimoto, G., Akimov, A. V., Akiyama, A., Alam, M. S., Alam, M. A., Albert, J., Albrand, S., Aleksa, M., Aleksandrov, I. N., Alessandria, F., Alexa, C., Alexander, G., Alexandre, G., Alexopoulos, T., Alhroob, M., Aliev, M., Alimonti, G., Alison, J., Aliyev, M., Allbrooke, B. M. M., Allport, P. P., Allwood-Spiers, S. E., Almond, J., Aloisio, A., Alon, R., Alonso, A., Alvarez Gonzalez, B., Alviggi, M. G., Amako, K., Amaral, P., Amelung, C., Ammosov, V. V., Amorim, A., Amorós, G., Amram, N., Anastopoulos, C., Ancu, L. S., Andari, N., Andeen, T., Anders, C. 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G., Sarangi, T., Sarkisyan-Grinbaum, E., Sarri, F., Sartisohn, G., Sasaki, O., Sasao, N., Satsounkevitch, I., Sauvage, G., Sauvan, E., Sauvan, J. B., Savard, P., Savinov, V., Savu, D. O., Sawyer, L., Saxon, D. H., Says, L. P., Sbarra, C., Sbrizzi, A., Scallon, O., Scannicchio, D. A., Scarcella, M., Schaarschmidt, J., Schacht, P., Schäfer, U., Schaepe, S., Schaetzel, S., Schaffer, A. C., Schaile, D., Schamberger, R. D., Schamov, A. G., Scharf, V., Schegelsky, V. A., Scheirich, D., Schernau, M., Scherzer, M. I., Schiavi, C., Schieck, J., Schioppa, M., Schlenker, S., Schlereth, J. L., Schmidt, E., Schmieden, K., Schmitt, C., Schmitt, S., Schmitz, M., Schöning, A., Schott, M., Schouten, D., Schovancova, J., Schram, M., Schroeder, C., Schroer, N., Schuh, S., Schuler, G., Schultens, M. J., Schultes, J., Schultz-Coulon, H.-C., Schulz, H., Schumacher, J. W., Schumacher, M., Schumm, B. A., Schune, Ph., Schwanenberger, C., Schwartzman, A., Schwemling, Ph., Schwienhorst, R., Schwierz, R., Schwindling, J., Schwindt, T., Schwoerer, M., Scott, W. G., Searcy, J., Sedov, G., Sedykh, E., Segura, E., Seidel, S. C., Seiden, A., Seifert, F., Seixas, J. M., Sekhniaidze, G., Selbach, K. E., Seliverstov, D. M., Sellden, B., Sellers, G., Seman, M., Semprini-Cesari, N., Serfon, C., Serin, L., Serkin, L., Seuster, R., Severini, H., Sevior, M. E., Sfyrla, A., Shabalina, E., Shamim, M., Shan, L. Y., Shank, J. T., Shao, Q. T., Shapiro, M., Shatalov, P. B., Shaver, L., Shaw, K., Sherman, D., Sherwood, P., Shibata, A., Shichi, H., Shimizu, S., Shimojima, M., Shin, T., Shiyakova, M., Shmeleva, A., Shochet, M. J., Short, D., Shrestha, S., Shulga, E., Shupe, M. A., Sicho, P., Sidoti, A., Siegert, F., Sijacki, Dj., Silbert, O., Silva, J., Silver, Y., Silverstein, D., Silverstein, S. B., Simak, V., Simard, O., Simic, Lj., Simion, S., Simmons, B., Simonyan, M., Sinervo, P., Sinev, N. B., Sipica, V., Siragusa, G., Sircar, A., Sisakyan, A. N., Sivoklokov, S. Yu., Sjölin, J., Sjursen, T. B., Skinnari, L. A., Skottowe, H. P., Skovpen, K., Skubic, P., Skvorodnev, N., Slater, M., Slavicek, T., Sliwa, K., Sloper, J., Smakhtin, V., Smirnov, S. Yu., Smirnov, Y., Smirnova, L. N., Smirnova, O., Smith, B. C., Smith, D., Smith, K. M., Smizanska, M., Smolek, K., Snesarev, A. A., Snow, S. W., Snow, J., Snuverink, J., Snyder, S., Soares, M., Sobie, R., Sodomka, J., Soffer, A., Solans, C. A., Solar, M., Solc, J., Soldatov, E., Soldevila, U., Solfaroli Camillocci, E., Solodkov, A. A., Solovyanov, O. V., Soni, N., Sopko, V., Sopko, B., Sosebee, M., Soualah, R., Soukharev, A., Spagnolo, S., Spanò, F., Spighi, R., Spigo, G., Spila, F., Spiwoks, R., Spousta, M., Spreitzer, T., Spurlock, B., St. Denis, R. D., Stahlman, J., Stamen, R., Stanecka, E., Stanek, R. W., Stanescu, C., Stapnes, S., Starchenko, E. A., Stark, J., Staroba, P., Starovoitov, P., Staude, A., Stavina, P., Stavropoulos, G., Steele, G., Steinbach, P., Steinberg, P., Stekl, I., Stelzer, B., Stelzer, H. J., Stelzer-Chilton, O., Stenzel, H., Stern, S., Stevenson, K., Stewart, G. A., Stillings, J. A., Stockton, M. C., Stoerig, K., Stoicea, G., Stonjek, S., Strachota, P., Stradling, A. R., Straessner, A., Strandberg, J., Strandberg, S., Strandlie, A., Strang, M., Strauss, E., Strauss, M., Strizenec, P., Ströhmer, R., Strom, D. M., Strong, J. A., Stroynowski, R., Strube, J., Stugu, B., Stumer, I., Stupak, J., Sturm, P., Styles, N. A., Soh, D. A., Su, D., Subramania, HS., Succurro, A., Sugaya, Y., Sugimoto, T., Suhr, C., Suita, K., Suk, M., Sulin, V. V., Sultansoy, S., Sumida, T., Sun, X., Sundermann, J. E., Suruliz, K., Sushkov, S., Susinno, G., Sutton, M. R., Suzuki, Y., Svatos, M., Sviridov, Yu. M., Swedish, S., Sykora, I., Sykora, T., Szeless, B., Sánchez, J., Ta, D., Tackmann, K., Taffard, A., Tafirout, R., Taiblum, N., Takahashi, Y., Takai, H., Takashima, R., Takeda, H., Takeshita, T., Takubo, Y., Talby, M., Talyshev, A., Tamsett, M. C., Tanaka, J., Tanaka, R., Tanaka, S., Tanaka, Y., Tanasijczuk, A. J., Tani, K., Tannoury, N., Tappern, G. P., Tapprogge, S., Tardif, D., Tarem, S., Tarrade, F., Tartarelli, G. F., Tas, P., Tasevsky, M., Tassi, E., Tatarkhanov, M., Tayalati, Y., Taylor, C., Taylor, F. E., Taylor, G. N., Taylor, W., Teinturier, M., Teixeira Dias Castanheira, M., Teixeira-Dias, P., Temming, K. K., Ten Kate, H., Teng, P. K., Terada, S., Terashi, K., Terron, J., Testa, M., Teuscher, R. J., Thadome, J., Therhaag, J., Theveneaux-Pelzer, T., Thioye, M., Thoma, S., Thomas, J. P., Thompson, E. N., Thompson, P. D., Thompson, A. S., Thomson, E., Thomson, M., Thun, R. P., Tian, F., Tibbetts, M. J., Tic, T., Tikhomirov, V. O., Tikhonov, Y. A., Timoshenko, S., Tipton, P., Tique Aires Viegas, F. J., Tisserant, S., Toczek, B., Todorov, T., Todorova-Nova, S., Toggerson, B., Tojo, J., Tokár, S., Tokunaga, K., Tokushuku, K., Tollefson, K., Tomoto, M., Tompkins, L., Toms, K., Tong, G., Tonoyan, A., Topfel, C., Topilin, N. D., Torchiani, I., Torrence, E., Torres, H., Torró Pastor, E., Toth, J., Touchard, F., Tovey, D. R., Trefzger, T., Tremblet, L., Tricoli, A., Trigger, I. M., Trincaz-Duvoid, S., Trinh, T. N., Tripiana, M. F., Trischuk, W., Trivedi, A., Trocmé, B., Troncon, C., Trottier-McDonald, M., Trzebinski, M., Trzupek, A., Tsarouchas, C., Tseng, J. C-L., Tsiakiris, M., Tsiareshka, P. V., Tsionou, D., Tsipolitis, G., Tsiskaridze, V., Tskhadadze, E. G., Tsukerman, I. I., Tsulaia, V., Tsung, J.-W., Tsuno, S., Tsybychev, D., Tua, A., Tudorache, A., Tudorache, V., Tuggle, J. M., Turala, M., Turecek, D., Turk Cakir, I., Turlay, E., Turra, R., Tuts, P. M., Tykhonov, A., Tylmad, M., Tyndel, M., Tzanakos, G., Uchida, K., Ueda, I., Ueno, R., Ugland, M., Uhlenbrock, M., Uhrmacher, M., Ukegawa, F., Unal, G., Underwood, D. G., Undrus, A., Unel, G., Unno, Y., Urbaniec, D., Usai, G., Uslenghi, M., Vacavant, L., Vacek, V., Vachon, B., Vahsen, S., Valenta, J., Valente, P., Valentinetti, S., Valkar, S., Valladolid Gallego, E., Vallecorsa, S., Valls Ferrer, J. A., van der Graaf, H., van der Kraaij, E., Van Der Leeuw, R., van der Poel, E., van der Ster, D., van Eldik, N., van Gemmeren, P., van Kesteren, Z., van Vulpen, I., Vanadia, M., Vandelli, W., Vandoni, G., Vaniachine, A., Vankov, P., Vannucci, F., Varela Rodriguez, F., Vari, R., Varnes, E. W., Varouchas, D., Vartapetian, A., Varvell, K. E., Vassilakopoulos, V. I., Vazeille, F., Vegni, G., Veillet, J. J., Vellidis, C., Veloso, F., Veness, R., Veneziano, S., Ventura, A., Ventura, D., Venturi, M., Venturi, N., Vercesi, V., Verducci, M., Verkerke, W., Vermeulen, J. C., Vest, A., Vetterli, M. C., Vichou, I., Vickey, T., Vickey Boeriu, O. E., Viehhauser, G. H. A., Viel, S., Villa, M., Villaplana Perez, M., Vilucchi, E., Vincter, M. G., Vinek, E., Vinogradov, V. B., Virchaux, M., Virzi, J., Vitells, O., Viti, M., Vivarelli, I., Vives Vaque, F., Vlachos, S., Vladoiu, D., Vlasak, M., Vlasov, N., Vogel, A., Vokac, P., Volpi, G., Volpi, M., Volpini, G., von der Schmitt, H., von Loeben, J., von Radziewski, H., von Toerne, E., Vorobel, V., Vorobiev, A. P., Vorwerk, V., Vos, M., Voss, R., Voss, T. T., Vossebeld, J. H., Vranjes, N., Vranjes Milosavljevic, M., Vrba, V., Vreeswijk, M., Vu Anh, T., Vuillermet, R., Vukotic, I., Wagner, W., Wagner, P., Wahlen, H., Wakabayashi, J., Walbersloh, J., Walch, S., Walder, J., Walker, R., Walkowiak, W., Wall, R., Waller, P., Wang, C., Wang, H., Wang, J., Wang, J. C., Wang, R., Wang, S. M., Warburton, A., Ward, C. P., Warsinsky, M., Watkins, P. M., Watson, A. T., Watson, I. J., Watson, M. F., Watts, G., Watts, S., Waugh, A. T., Waugh, B. M., Weber, M., Weber, M. S., Weber, P., Weidberg, A. R., Weigell, P., Weingarten, J., Weiser, C., Wellenstein, H., Wells, P. S., Wen, M., Wenaus, T., Wendler, S., Weng, Z., Wengler, T., Wenig, S., Wermes, N., Werner, M., Werner, P., Werth, M., Wessels, M., Weydert, C., Whalen, K., Wheeler-Ellis, S. J., Whitaker, S. P., White, A., White, M. J., Whitehead, S. R., Whiteson, D., Whittington, D., Wicek, F., Wicke, D., Wickens, F. J., Wiedenmann, W., Wielers, M., Wienemann, P., Wiglesworth, C., Wiik-Fuchs, L. A. M., Wijeratne, P. A., Wildauer, A., Wildt, M. A., Wilhelm, I., Wilkens, H. G., Will, J. Z., Williams, E., Williams, H. H., Willis, W., Willocq, S., Wilson, J. A., Wilson, M. G., Wilson, A., Wingerter-Seez, I., Winkelmann, S., Winklmeier, F., Wittgen, M., Wolter, M. W., Wolters, H., Wong, W. C., Wooden, G., Wosiek, B. K., Wotschack, J., Woudstra, M. J., Wozniak, K. W., Wraight, K., Wright, C., Wright, M., Wrona, B., Wu, S. L., Wu, X., Wu, Y., Wulf, E., Wunstorf, R., Wynne, B. M., Xella, S., Xiao, M., Xie, S., Xie, Y., Xu, C., Xu, D., Xu, G., Yabsley, B., Yacoob, S., Yamada, M., Yamaguchi, H., Yamamoto, A., Yamamoto, K., Yamamoto, S., Yamamura, T., Yamanaka, T., Yamaoka, J., Yamazaki, T., Yamazaki, Y., Yan, Z., Yang, H., Yang, U. K., Yang, Y., Yang, Z., Yanush, S., Yao, Y., Yasu, Y., Ybeles Smit, G. V., Ye, J., Ye, S., Yilmaz, M., Yoosoofmiya, R., Yorita, K., Yoshida, R., Young, C., Youssef, S., Yu, D., Yu, J., Yuan, L., Yurkewicz, A., Zabinski, B., Zaets, V. G., Zaidan, R., Zaitsev, A. M., Zajacova, Z., Zanello, L., Zarzhitsky, P., Zaytsev, A., Zeitnitz, C., Zeller, M., Zeman, M., Zemla, A., Zendler, C., Zenin, O., Ženiš, T., Zinonos, Z., Zenz, S., Zerwas, D., Zevi della Porta, G., Zhan, Z., Zhang, D., Zhang, H., Zhang, J., Zhang, X., Zhang, Z., Zhao, L., Zhao, T., Zhao, Z., Zhemchugov, A., Zheng, S., Zhong, J., Zhou, B., Zhou, N., Zhou, Y., Zhu, C. G., Zhu, H., Zhu, J., Zhu, Y., Zhuang, X., Zhuravlov, V., Zieminska, D., Zimmermann, R., Zimmermann, S., Ziolkowski, M., Zitoun, R., Živković, L., Zmouchko, V. V., Zobernig, G., Zoccoli, A., Zolnierowski, Y., Zsenei, A., zur Nedden, M., Zutshi, V., and Zwalinski, L.
- Subjects
Physics ,Particle physics ,Top quark ,Large Hadron Collider ,Physics and Astronomy (miscellaneous) ,010308 nuclear & particles physics ,Astrophysics::High Energy Astrophysical Phenomena ,High Energy Physics::Phenomenology ,01 natural sciences ,7. Clean energy ,Luminosity ,Nuclear physics ,medicine.anatomical_structure ,Atlas (anatomy) ,0103 physical sciences ,medicine ,High Energy Physics::Experiment ,Nuclear Experiment ,010306 general physics ,Engineering (miscellaneous) ,Communication channel ,Bar (unit) ,Template method pattern ,Lepton - Abstract
The top quark mass has been measured using the template method in the t (t) over bar -> lepton + jets channel based on data recorded in 2011 with the ATLAS detector at the LHC. The data were taken at a proton-proton centre-of-mass energy of root s = 7 TeV and correspond to an integrated luminosity of 1.04 fb(-1). The analyses in the e + jets and mu + jets decay channels yield consistent results. The top quark mass is measured to be m(top) = 174.5 +/- 0.6(stat) +/- 2.3(syst) GeV.
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- View/download PDF
37. Structure of doubly-even cadmium nuclei studied by β− decay.
- Author
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Gross, Carl J., Nazarewicz, Witold, Rykaczewski, Krzysztof P., Rinta-Antila, S., Wang, Y., Dendooven, P., Huikari, J., Jokinen, A., Kankainen, A., Kolhinen, V. S., Lhersonneau, G., Nieminen, A., Nummela, S., Penttilä, H., Peräjärvi, K., Szerypo, J., Wang, J. C., and Äystö, J.
- Abstract
We have studied the structure of even-even cadmium isotopes via beta decay of ground and excited isomeric states of parent silver isotopes. Measurements of mass A = 116, 118 and 120 cadmium nuclides were carried out at an ion guide isotope separation on-line facility at the University of Jyväskylä. Decay schemes of 116mAg, 118mAg, 120gAg and 120mAg are considerably extended. Obtained data have enabled extension of available systematics of the three-phonon states to more neutron-rich cadmium nuclei. As a continuation we have conducted an experiment at ISOLDE, CERN to study heavier A = 122, 124, and 126 cadmium nuclides, the analysis of the collected data is underway. [ABSTRACT FROM AUTHOR]
- Published
- 2005
- Full Text
- View/download PDF
38. Atomic mass ratios for some stable isotopes of platinum relative to 197Au.
- Author
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Gross, Carl J., Nazarewicz, Witold, Rykaczewski, Krzysztof P., Sharma, K. S., Vaz, J., Barber, R. C., Buchinger, F., Clark, J. A., Crawford, J. E., Fukutani, H., Greene, J. P., Gulick, S., Heinz, A., Lee, J. K. P., Savard, G., Zhou, Z., and Wang, J. C.
- Abstract
The Canadian Penning Trap mass spectrometer was designed to determine precisely the masses of stable and unstable isotopes. To date, such measurements have been carried out on approximately 60 short-lived species. A laser ablation ion source is also available to produce ions of stable isotopes, intended for use in calibrations, checks for systematic effects and for measurements involving stable isotopes. Mass ratios for the isotopes 194,195,196,198Pt relative to 197Au have been determined to a precision of better than 3 × 10−8. These measurements were motivated, in part, by the long-standing discrepancy between earlier mass measurements and the Atomic Mass Evaluations in the mercury region. The results also demonstrate the stability of the measurement system and set limits on the magnitude of systematic effects. No significant deviations from accepted values were found. [ABSTRACT FROM AUTHOR]
- Published
- 2005
- Full Text
- View/download PDF
39. Association of childhood trauma exposure and GABRA2 polymorphisms with risk of posttraumatic stress disorder in adults.
- Author
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Nelson, E. C., Agrawal, A., Pergadia, M. L., Lynskey, M. T., Todorov, A. A., Wang, J. C., Todd, R. .D, Martin, N. G., Heath, A. C., Goate, A. M., Montgomery, G. .W, and Madden, P. A. F.
- Subjects
LETTERS to the editor ,GENETIC polymorphisms - Abstract
A letter to the editor is presented discussing the study on the association of childhood trauma exposure and GABRA2 polymorphisms with risk of posttraumatic stress disorder in adults.
- Published
- 2009
- Full Text
- View/download PDF
40. Atomic mass ratios for some stable isotopes of platinum relative to 197Au.
- Author
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Sharma, K. S., Vaz, J., Barber, R. C., Buchinger, F., Clark, J. A., Crawford, J. E., Fukutani, H., Greene, J. P., Gulick, S., Heinz, A., Lee, J. K. P., Savard, G., Zhou, Z., and Wang, J. C.
- Subjects
PENNING trap mass spectrometry ,ISOTOPES ,LASER ablation ,ION sources ,MASS measurement ,NUCLEAR physics - Abstract
The Canadian Penning Trap mass spectrometer was designed to determine precisely the masses of stable and unstable isotopes. To date, such measurements have been carried out on approximately 60 short-lived species. A laser ablation ion source is also available to produce ions of stable isotopes, intended for use in calibrations, checks for systematic effects and for measurements involving stable isotopes. Mass ratios for the isotopes
194,195,196,198 Pt relative to197 Au have been determined to a precision of better than 3 × 10-8 . These measurements were motivated, in part, by the long-standing discrepancy between earlier mass measurements and the Atomic Mass Evaluations in the mercury region. The results also demonstrate the stability of the measurement system and set limits on the magnitude of systematic effects. No significant deviations from accepted values were found. [ABSTRACT FROM AUTHOR]- Published
- 2005
- Full Text
- View/download PDF
41. Autologous plasma eyedrops prepared in a closed system: a treatment for dry eye.
- Author
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Petznick, A, Tong, L, Chung, R, Wang, J C, Koh, M, Salleh, R, and Waduthantri, S
- Subjects
DRY eye syndromes ,EYE diseases - Abstract
A letter to the editor on treatment for dry eye is presented.
- Published
- 2013
- Full Text
- View/download PDF
42. Records of the freshwater lampreys, Lampetra lamottenii and Okkelbergia aepyptera, from the Delmarva Peninsula, East Coast, United States
- Author
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Rohde, F. C., Arndt, R. G., and Wang, J. C. S.
- Published
- 1975
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- View/download PDF
43. A supplemental sampling method of estuarine ichthyoplankton with emphasis on the Atherinidae
- Author
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Lindsay, J. A., Wang, J. C. S., and Radle, E. R.
- Published
- 1978
- Full Text
- View/download PDF
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