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Your search keyword '"sun damage"' showing total 5 results
Start Over Descriptor "sun damage" Journal journal of investigative dermatology
5 results on '"sun damage"'

1. 553 Biophoton-revealed ultraweak oxidative stress represents unnoticed chronic sun damage in the human skin

Author

Y. Haryuu, Yoshito Takahashi, S. Nakamura, Nakajima Yoko, Megumi Tobiishi, Daiki Murase, Kikuchi Sho, Yukio Inomata, Takeda Katsuya, Koki Tsuda, and Yu Gabe

Subjects
Pathology, medicine.medical_specialty, business.industry, Human skin, Sun damage, Cell Biology, Dermatology, medicine.disease_cause, Biochemistry, Biophoton, Medicine, business, Molecular Biology, Oxidative stress
Published
2019

2. 830 Gene-UV interactions determining sun damage

Author

Joseph G. Morelli, E. Meyer, Tamara Terzian, C. Little, Lori A. Crane, Neil F. Box, D. Lynn, Robert P. Dellavalle, Marianne Berwick, J. Aalborg, N. Asdigian, and S. Mokrohisky

Subjects
Chemistry, Sun damage, Cell Biology, Dermatology, Molecular Biology, Biochemistry, Gene, Cell biology
Published
2017

4. Absence of PDGFRA Mutations in Primary Melanoma

Author

Daniel Pinkel, Boris C. Bastian, and John A. Curtin

Subjects
Neuroblastoma RAS viral oncogene homolog, Receptor, Platelet-Derived Growth Factor alpha, Skin Neoplasms, medicine.medical_treatment, Viral Oncogene, Dermatology, PDGFRA, Biology, Biochemistry, 03 medical and health sciences, 0302 clinical medicine, Neuroblastoma, medicine, Humans, Melanoma, Molecular Biology, 030304 developmental biology, 0303 health sciences, Growth factor, Sun damage, Cell Biology, medicine.disease, 3. Good health, Gene Expression Regulation, Neoplastic, 030220 oncology & carcinogenesis, Cancer research, Sarcoma
Abstract

Abbreviations: CSD, chronic sun damage; KIT, v-kit Hardy-Zuckerman 4 feline sarcoma viral oncogene homolog; NRAS, neuroblastoma ras viral oncogene homolog; PDGFRA, platelet-derived growth factor α receptor

Published
2008

5. Alterations in Human Dermal Connective Tissue with Age and Chronic Sun Damage

Author

W. Mitchell Sams, J. Graham Smith, Eugene A. Davidson, and Richard D. Clark

Subjects
Pathology, medicine.medical_specialty, Aging, Actinic Damage, Connective tissue, Human skin, Dermatology, Biochemistry, Hydroxyproline, chemistry.chemical_compound, medicine, Humans, Molecular Biology, Skin, Dermal collagen, integumentary system, business.industry, Sun damage, Cell Biology, Actinic elastosis, medicine.disease, Radiation Effects, medicine.anatomical_structure, chemistry, Connective Tissue, Sunlight, business
Abstract

Aged unexposed human skin has decreased hexosamine (22), decreased solubility of dermal collagen (1), and increased hydroxyproline (22), with few dramatic histological alterations (8) as compared with young skin. In contrast, the identity of the histopathologic changes in chronically sun-damaged skin (actinic elastosis) has been debated for many years (5, 6, 9, 10, 23). These changes in exposed skin are caused primarily by prolonged and repeated actinic damage (7,8,11,21,23).

Published
1962
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