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20 results on '"Oehme, Ina"'

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1. Selective degradation of mutant FMS-like tyrosine kinase-3 requires BIM-dependent depletion of heat shock proteins

2. ERBB and P‐glycoprotein inhibitors break resistance in relapsed neuroblastoma models through P‐glycoprotein.

3. Aza-SAHA Derivatives Are Selective Histone Deacetylase 10 Chemical Probes That Inhibit Polyamine Deacetylation and Phenocopy HDAC10 Knockout

4. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

6. Structure-Based Design and Biological Characterization of Selective Histone Deacetylase 8 (HDAC8) Inhibitors with Anti-Neuroblastoma Activity

8. Selective Inhibition of Histone Deacetylase 10: Hydrogen Bonding to the Gatekeeper Residue is Implicated

9. A kinome-wide RNAi screen identifies ALK as a target to sensitize neuroblastoma cells for HDAC8-inhibitor treatment

10. Reactivation of endogenous retroviral elements via treatment with DNMT- and HDAC-inhibitors

12. DNMT and HDAC inhibitors induce cryptic transcription start sites encoded in long terminal repeats

13. Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

14. Downregulation of the activating NKp30 ligand B7-H6 by HDAC inhibitors impairs tumor cell recognition by NK cells

15. Downregulation of the activating NKp30 ligand B7-H6 by HDAC inhibitors impairs tumor cell recognition by NK cells

16. Targeting of HDAC8 and investigational inhibitors in neuroblastoma

17. Histone deacetylase 10-promoted autophagy as a druggable point of interference to improve the treatment response of advanced neuroblastomas

18. Erratum: DNMT and HDAC inhibitors induce cryptic transcription start sites encoded in long terminal repeats

19. Epigenetic Drug Treatment Globally Induces Cryptic Transcription Start Sites Encoded in Long Terminal Repeats

20. Epigenetic Drug Treatment Globally Induces Cryptic Transcription Start Sites Encoded in Long Terminal Repeats

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