116 results on '"Papazian, Natalie"'
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2. Inhibition of MICA and MICB shedding enhances memory-like NK-cell–mediated cytotoxicity against multiple myeloma
3. The multiple myeloma microenvironment is defined by an inflammatory stromal cell landscape
4. An IL-1β driven neutrophil-stromal cell axis fosters a BAFF-rich microenvironment in multiple myeloma
5. Immune-mediated tumor control in the 5TGM1 transfer model of multiple myeloma
6. Tertiary Lymphoid Structures in Rheumatoid Arthritis: NF-κB–Inducing Kinase–Positive Endothelial Cells as Central Players
7. MAF Translocations Are Enriched in NDMM Patients with Elevated Levels of Circulating Tumor Cells Suggesting a Genetic Basis for Aggressive Disease Course
8. Inhibition of MICA and MICB Shedding Enhances Cytokine-Induced Memory-like NK Cell-Mediated Activity Against Multiple Myeloma
9. Stromal Cell-Activated Bone Marrow Neutrophils Provide BAFF in Newly Diagnosed and Treated Multiple Myeloma
10. Single-Cell Transcriptomic Analysis Reveals Reduction of Cytotoxic NK Cells in a Subset of Newly Diagnosed Multiple Myeloma Patients Impacting Outcome after Daratumumab Therapy
11. P-021: 7C6 is a novel monoclonal antibody that induces enhanced anti-myeloma activity of cytokine induced memory-like (CIML) NK cells by blocking MICA/B shedding and antibodydependent cell cytotoxicity
12. P-062: MAF translocations are enriched in high-risk NDMM patients with elevated levels of circulating tumor cells suggesting a genetic basis for the aggressive disease course
13. OAB-029: Single-cell transcriptomic analysis reveals reduction of cytotoxic NK cells in a subset of newly diagnosed multiple myeloma patients impacting outcome after daratumumab therapy
14. P-082: Stromal cell - neutrophil interactions are driving a pro tumor environment in multiple myeloma
15. Single-Cell Transcriptomic Analysis Reveals Loss of Activated Bone Marrow NK Cells in Multiple Myeloma Patients Which Associates with Disease Progression in Mice
16. High Levels of Circulating Tumor Cells Are Associated with Increased Bone Marrow Proliferation in Newly Diagnosed Multiple Myeloma Patients
17. Inflammasome-Primed Myeloid Cells Maintain a Pro-Tumor Microenvironment in Multiple Myeloma
18. P-080: Single-cell transcriptomic analysis of bone marrow NK cells reveals loss of activated cytotoxic NK cells in Multiple Myeloma
19. P-072: Progression and dissemination of experimental multiple myeloma is associated with loss of innate and adaptive immune-mediated tumor control
20. OAB-014: Newly diagnosed Multiple Myeloma patients with high levels of circulating tumor cells are distinguished by increased bone marrow plasma cell proliferation
21. P-069: Inflammasome-primed neutrophils maintain a pro-tumor microenvironment in Multiple Myeloma
22. Intestinal-derived ILCs migrating in lymph increase IFNγ production in response to Salmonella Typhimurium infection
23. Fibroblast-derived IL-33 is dispensable for lymph node homeostasis but critical for CD8 T-cell responses to acute and chronic viral infection
24. P-388 An IL-1β driven neutrophil-stromal cell axis fosters a BAFF-rich tumor-supportive bone marrow environment in multiple myeloma patients
25. Yap1-Driven Intestinal Repair Is Controlled by Group 3 Innate Lymphoid Cells
26. Fibroblast‐derived IL‐33 is dispensable for lymph node homeostasis but critical for CD8 T‐cell responses to acute and chronic viral infection
27. Yap1-Driven Intestinal Repair Is Controlled by Group 3 Innate Lymphoid Cells
28. Single Cell Transcriptomic Analysis of the Multiple Myeloma Bone Marrow Identifies a Unique Inflammatory Stromal Cell Population Associated with TNF Signaling
29. Expression of Plet1 controls interstitial migration of murine small intestinal dendritic cells
30. Yap1-driven intestinal repair is controlled by group 3 innate lymphoid cells
31. Expression of Plet1 controls interstitial migration of murine small intestinal dendritic cells
32. IL-7-dependent maintenance of ILC3s is required for normal entry of lymphocytes into lymph nodes
33. Fibroblast‐derived IL‐33 is dispensable for lymph node homeostasis but critical for CD8 T‐cell responses to acute and chronic viral infection.
34. Intestinal-derived ILCs migrating in lymph increase IFNγ production in response to SalmonellaTyphimurium infection
35. IL-7–dependent maintenance of ILC3s is required for normal entry of lymphocytes into lymph nodes
36. Cross-Tissue Transcriptomic Analysis of Human Secondary Lymphoid Organ-Residing ILC3s Reveals a Quiescent State in the Absence of Inflammation
37. Progressive maturation toward hematopoietic stem cells in the mouse embryo aorta
38. Cross-Tissue Transcriptomic Analysis of Human Secondary Lymphoid Organ-Residing ILC3s Reveals a Quiescent State in the Absence of Inflammation
39. Tertiary Lymphoid Structures in Rheumatoid Arthritis NF-kappa B-Inducing Kinase-Positive Endothelial Cells as Central Players
40. Expression of Plet1 controls interstitial migration of murine small intestinal dendritic cells.
41. Loss of IL-22 inhibits autoantibody formation in collagen-induced arthritis in mice
42. Progressive maturation toward hematopoietic stem cells in the mouse embryo aorta
43. Type 3 innate lymphoid cells maintain intestinal epithelial stem cells after tissue damage
44. A Stromal Cell Niche for Human and Mouse Type 3 Innate Lymphoid Cells
45. Type 3 innate lymphoid cells maintain intestinal epithelial stem cells after tissue damage
46. Functional differences between human NKp44- and NKp44+ RORC+ innate lymphoid cells
47. IL-22 receptor-expressing stromal cells are located in the germinal center dark zone (P1451)
48. Functional Differences between Human NKp44− and NKp44+ RORC+ Innate Lymphoid Cells
49. NK cells can generate from precursors in the adult human liver
50. Human Placenta Is a Potent Hematopoietic Niche Containing Hematopoietic Stem and Progenitor Cells throughout Development
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