796 results on '"Oh, Goo Taeg"'
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2. A resource of targeted mutant mouse lines for 5,061 genes
3. Complexation of drug and hapten-conjugated aptamer with universal hapten antibody for pancreatic cancer treatment
4. miR-125a-5p attenuates macrophage-mediated vascular dysfunction by targeting Ninjurin1
5. Single-cell transcriptomics reveal cellular diversity of aortic valve and the immunomodulation by PPARγ during hyperlipidemia
6. 2′–5′ oligoadenylate synthetase‑like 1 (OASL1) protects against atherosclerosis by maintaining endothelial nitric oxide synthase mRNA stability
7. ER-associated CTRP1 regulates mitochondrial fission via interaction with DRP1
8. Mitochondrial H2O2 Is a Central Mediator of Diclofenac-Induced Hepatocellular Injury
9. Deficiency of peroxiredoxin 2 exacerbates angiotensin II-induced abdominal aortic aneurysm
10. Oxidized LDL induces vimentin secretion by macrophages and contributes to atherosclerotic inflammation
11. The adipokine Retnla deficiency increases responsiveness to cardiac repair through adiponectin-rich bone marrow cells
12. Current pharmacotherapies for atherosclerotic cardiovascular diseases
13. Ninjurin1 positively regulates osteoclast development by enhancing the survival of prefusion osteoclasts
14. Intragenic CpG islands play important roles in bivalent chromatin assembly of developmental genes
15. Anti-Inflammatory Actions of Soluble Ninjurin-1 Ameliorate Atherosclerosis
16. Mitochondrial H 2 O 2 Is a Central Mediator of Diclofenac-Induced Hepatocellular Injury.
17. N-α-acetyltransferase 10 (NAA10) in development: the role of NAA10
18. Progerinin, an Inhibitor of Progerin, Alleviates Cardiac Abnormalities in a Model Mouse of Hutchinson–Gilford Progeria Syndrome
19. Supplementary Methods and Materials, Tables 1-4, Figure Legends 1-3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
20. Supplementary Figure 3 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
21. Supplementary Figure 1 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
22. Supplementary Figure 5 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
23. Supplementary Figure 2 from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
24. Supplementary Materials and Methods, Figure Legends 1-7, Table 1 Legend from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
25. Data from Functional and Clinical Evidence for NDRG2 as a Candidate Suppressor of Liver Cancer Metastasis
26. Data from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
27. Supplementary Figure 3 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
28. Supplementary Figure 4 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
29. Supplementary Figure 6 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
30. Supplementary Figure 2 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
31. Supplementary Figure 7 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
32. Supplementary Figure 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
33. Supplementary Table 1 from Arrest Defective 1 Autoacetylation Is a Critical Step in Its Ability to Stimulate Cancer Cell Proliferation
34. Indoleamine 2,3-Dioxygenase-Expressing Aortic Plasmacytoid Dendritic Cells Protect against Atherosclerosis by Induction of Regulatory T Cells
35. C1q/TNF-α–Related Protein 1 (CTRP1) Maintains Blood Pressure Under Dehydration Conditions
36. Pancreatic β-cell mitophagy as an adaptive response to metabolic stress and the underlying mechanism that involves lysosomal Ca2+ release.
37. Unbalanced Redox With Autophagy in Cardiovascular Disease
38. Endothelial PTP4A1 mitigates vascular inflammation via USF1/A20 axis-mediated NF-κB inactivation
39. Peroxisomal Fitness: A Potential Protective Mechanism of Fenofibrate against High Fat Diet-Induced Non-Alcoholic Fatty Liver Disease in Mice
40. Negligible effect of eNOS palmitoylation on fatty acid regulation of contraction in ventricular myocytes from healthy and hypertensive rats
41. Disruption of Ninjurin1 Leads to Repetitive and Anxiety-Like Behaviors in Mice
42. Impaired TFEB activation and mitophagy as a cause of PPP3/calcineurin inhibitor-induced pancreatic β-cell dysfunction
43. The antioxidant enzyme Peroxiredoxin-1 controls stroke-associated microglia against acute ischemic stroke
44. Transcriptome Analysis Reveals Nonfoamy Rather Than Foamy Plaque Macrophages Are Proinflammatory in Atherosclerotic Murine Models
45. Inhibition of Ninjurin 1 restores erectile function through dual angiogenic and neurotrophic effects in the diabetic mouse
46. Impaired TFEB activation and mitophagy as a cause of PPP3/calcineurin inhibitor-induced pancreatic β-cell dysfunction.
47. Extract of Rhus verniciflua stokes protects the diet-induced hyperlipidemia in mice
48. Plasma Membrane Localization of CD36 Requires Vimentin Phosphorylation; A Mechanism by Which Macrophage Vimentin Promotes Atherosclerosis
49. Peroxiredoxin 3 deficiency induces cardiac hypertrophy and dysfunction by impaired mitochondrial quality control
50. Identification and characterization of CW108F, a Novel β-carboline compound that promotes cardiomyogenesis of stem cells
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