241 results on '"Horai, Reiko"'
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2. The Cytokine IL-17A Limits Th17 Pathogenicity via a Negative Feedback Loop Driven by Autocrine Induction of IL-24
3. TLR2 Supports γδ T cell IL-17A Response to ocular surface commensals by Metabolic Reprogramming
4. AS101 ameliorates experimental autoimmune uveitis by regulating Th1 and Th17 responses and inducing Treg cells
5. Too Much of a Good Thing: Extended Duration of Gut Microbiota Depletion Reverses Protection From Experimental Autoimmune Uveitis
6. IL-17 Production from Activated T Cells Is Required for the Spontaneous Development of Destructive Arthritis in Mice Deficient in IL-1 Receptor Antagonist
7. T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis
8. Exposure of autoimmune uveitis-prone germ-free mice to commensal microbiota promotes development of ocular inflammation
9. Human gut commensals support development of ocular autoimmunity in retina-specific T cell receptor transgenic mice
10. Microbiota-Dependent Activation of an Autoreactive T Cell Receptor Provokes Autoimmunity in an Immunologically Privileged Site
11. IL-27p28 inhibits central nervous system autoimmunity by concurrently antagonizing Th1 and Th17 responses
12. Breakdown of immune privilege and spontaneous autoimmunity in mice expressing a transgenic T cell receptor specific for a retinal autoantigen
13. The Role of IL-1 in the Immune System
14. Development of promyelocytic leukemia zinc finger-expressing innate CD4 T cells requires stronger T-cell receptor signals than conventional CD4 T cells
15. Cbl Enforces an SLP76-dependent Signaling Pathway for T Cell Differentiation
16. Kid-Mediated Chromosome Compaction Ensures Proper Nuclear Envelope Formation
17. Faculty Opinions recommendation of Skin and gut imprinted helper T cell subsets exhibit distinct functional phenotypes in central nervous system autoimmunity.
18. NR2B tyrosine phosphorylation modulates fear learning as well as amygdaloid synaptic plasticity
19. Impaired in vitro regulatory T cell function associated with Wiskott–Aldrich syndrome
20. Requirements for Selection of Conventional and Innate T Lymphocyte Lineages
21. Alteration of behavioural phenotype in mice by targeted disruption of the progranulin gene
22. Microbiota as Drivers and as Therapeutic Targets in Ocular and Tissue Specific Autoimmunity
23. Reciprocal interaction between NK and DC regulates the autopathogenic Th17 response by controlling the innate IFN-γ/IL-27 axis: 46
24. Altered Development of CD8 + T Cell Lineages in Mice Deficient for the Tec Kinases Itk and Rlk
25. Retinal Inflammation: Uveitis/Uveoretinitis
26. Regulated Tristetraprolin Overexpression Dampens the Development and Pathogenesis of Experimental Autoimmune Uveitis
27. Association of gut microbiota with spontaneous autoimmune uveitis studied by human flora reconstitution of germ-free mice
28. Toll-like Receptor 2 is crucial for commensal-specific immune responses on the ocular surface
29. A novel self-regulatory mechanism of Th17 cells controls autoimmune uveitis through interleukin-24
30. GM-CSF mediates development of eosinophil-dominant autoimmune uveitis in the absence of both IFN-γ and IL-17A
31. The gut as a potential licensing site for central nervous system (CNS)-specific autoimmune lymphocytes
32. A novel role for lipoxin A4 in driving a lymph node–eye axis that controls autoimmunity to the neuroretina
33. Author response: A novel role for lipoxin A4 in driving a lymph node–eye axis that controls autoimmunity to the neuroretina
34. Tec kinases regulate T-lymphocyte development and function: new insights into the roles of Itk and Rlk/Txk
35. Deficiency of Interleukin-1 Receptor Antagonist Deteriorates Fatty Liver and Cholesterol Metabolism in Hypercholesterolemic Mice
36. Involvement of protein-tyrosine phosphatase PTPMEG in motor learning and cerebellar long-term depression
37. Ovol2/Movo, a homologue of Drosophila ovo, is required for angiogenesis, heart formation and placental development in mice
38. Altered Development of CD8+ T Cell Lineages in Mice Deficient for the Tec Kinases Itk and Rlk
39. CD28-Dependent Differentiation Into the Effector/Memory Phenotype Is Essential for Induction of Arthritis in Interleukin-1 Receptor Antagonist–Deficient Mice
40. Abnormal T cell activation caused by the imbalance of the IL-1/IL-1R antagonist system is responsible for the development of experimental autoimmune encephalomyelitis
41. Arthritis in Mice That Are Deficient in Interleukin-1 Receptor Antagonist Is Dependent on Genetic Background
42. TNF-α is crucial for the development of autoimmune arthritis in IL-1 receptor antagonist-deficient mice
43. IL-1 is required for allergen-specific Th2 cell activation and the development of airway hypersensitivity response
44. IL-1-induced tumor necrosis factor-α elicits inflammatory cell infiltration in the skin by inducing IFN-γ-inducible protein 10 in the elicitation phase of the contact hypersensitivity response
45. Suppression of Autoimmune Arthritis in Interleukin-1–Deficient Mice in Which T Cell Activation is Impaired Due to Low Levels of CD40 Ligand and OX40 Expression on T Cells
46. IL-1α, but not IL-1β, is required for contact-allergen-specific T cell activation during the sensitization phase in contact hypersensitivity
47. Interleukin-1β, but not interleukin-1α, is required for T-cell-dependent antibody production
48. Suppression of oxidative neuronal damage after transient middle cerebral artery occlusion in mice lacking interleukin-1
49. Lipopolysaccharide-induced HIV-1 expression in transgenic mice is mediated by tumor necrosis factor-α and interleukin-1, but not by interferon-γ nor interleukin-6
50. Development of Chronic Inflammatory Arthropathy Resembling Rheumatoid Arthritis in Interleukin 1 Receptor Antagonist-deficient Mice
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