SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates

MLA

Massachusetts Institute of Technology. Department of Biological Engineering, et al. “SETDB1 Acts as a Topological Accessory to Cohesin via an H3K9me3-Independent, Genomic Shunt for Regulating Cell Fates.” Nucleic Acids Research, 2023. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsoai&AN=edsoai.on1370255906&authtype=sso&custid=ns315887.

APA

Massachusetts Institute of Technology. Department of Biological Engineering, Warrier, T., El Farran, C., Zeng, Y., Ho, B. S. Q., Bao, Q., Zheng, Z. H., Bi, X., Ng, H. H., Ong, D. S. T., Chu, J. J. H., Sanyal, A., Fullwood, M. J., Collins, J. J., Li, H., Xu, J., & Loh, Y.-H. (2023). SETDB1 acts as a topological accessory to Cohesin via an H3K9me3-independent, genomic shunt for regulating cell fates. Nucleic Acids Research.

Chicago

Massachusetts Institute of Technology. Department of Biological Engineering, Tushar Warrier, Chadi El Farran, Yingying Zeng, Benedict Shao Quan Ho, Qiuye Bao, Zi Hao Zheng, et al. 2023. “SETDB1 Acts as a Topological Accessory to Cohesin via an H3K9me3-Independent, Genomic Shunt for Regulating Cell Fates.” Nucleic Acids Research. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsoai&AN=edsoai.on1370255906&authtype=sso&custid=ns315887.