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Advances in gene therapy for muscular dystrophies [version 1; referees: 2 approved]

Authors :
Hayder Abdul-Razak
Alberto Malerba
George Dickson
Author Affiliations :
<relatesTo>1</relatesTo>School of Biological Sciences, Royal Holloway, University of London, Egham, Surrey, TW20 0EX, UK
Source :
F1000Research. 5:F1000 Faculty Rev-2030
Publication Year :
2016
Publisher :
London, UK: F1000 Research Limited, 2016.

Abstract

Duchenne muscular dystrophy (DMD) is a recessive lethal inherited muscular dystrophy caused by mutations in the gene encoding dystrophin, a protein required for muscle fibre integrity. So far, many approaches have been tested from the traditional gene addition to newer advanced approaches based on manipulation of the cellular machinery either at the gene transcription, mRNA processing or translation levels. Unfortunately, despite all these efforts, no efficient treatments for DMD are currently available. In this review, we highlight the most advanced therapeutic strategies under investigation as potential DMD treatments.

Details

ISSN :
20461402
Volume :
5
Database :
F1000Research
Journal :
F1000Research
Notes :
Editorial Note on the Review Process F1000 Faculty Reviews are commissioned from members of the prestigious F1000 Faculty and are edited as a service to readers. In order to make these reviews as comprehensive and accessible as possible, the referees provide input before publication and only the final, revised version is published. The referees who approved the final version are listed with their names and affiliations but without their reports on earlier versions (any comments will already have been addressed in the published version). The referees who approved this article are: Shin'ichi Takeda, National Center of Neurology and Psychiatry, Tokyo, Japan No competing interests were disclosed. Giulio Cossu, University of Manchester, Manchester, UK No competing interests were disclosed., , [version 1; referees: 2 approved]
Publication Type :
Academic Journal
Accession number :
edsfor.10.12688.f1000research.8735.1
Document Type :
review
Full Text :
https://doi.org/10.12688/f1000research.8735.1