CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism

MLA

Sonia García-Rodríguez, et al. “CD38 Promotes Pristane-Induced Chronic Inflammation and Increases Susceptibility to Experimental Lupus by an Apoptosis-Driven and TRPM2-Dependent Mechanism.” Scientific Reports, vol. 8, no. 1, Feb. 2018, pp. 1–19. EBSCOhost, https://doi.org/10.1038/s41598-018-21337-6.

APA

Sonia García-Rodríguez, Antonio Rosal-Vela, Davide Botta, Luz M. Cumba Garcia, Esther Zumaquero, Verónica Prados-Maniviesa, Daniela Cerezo-Wallis, Nicola Lo Buono, José-Ángel Robles-Guirado, Salvador Guerrero, Elena González-Paredes, Eduardo Andrés-León, Ángel Corbí, Matthias Mack, Friedrich Koch-Nolte, Ramón Merino, Mercedes Zubiaur, Frances E. Lund, & Jaime Sancho. (2018). CD38 promotes pristane-induced chronic inflammation and increases susceptibility to experimental lupus by an apoptosis-driven and TRPM2-dependent mechanism. Scientific Reports, 8(1), 1–19. https://doi.org/10.1038/s41598-018-21337-6

Chicago

Sonia García-Rodríguez, Antonio Rosal-Vela, Davide Botta, Luz M. Cumba Garcia, Esther Zumaquero, Verónica Prados-Maniviesa, Daniela Cerezo-Wallis, et al. 2018. “CD38 Promotes Pristane-Induced Chronic Inflammation and Increases Susceptibility to Experimental Lupus by an Apoptosis-Driven and TRPM2-Dependent Mechanism.” Scientific Reports 8 (1): 1–19. doi:10.1038/s41598-018-21337-6.