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EVI1 overexpression reprograms hematopoiesis via upregulation of Spi1 transcription

Authors :
Edward Ayoub
Michael P. Wilson
Kathleen E. McGrath
Allison J. Li
Benjamin J. Frisch
James Palis
Laura M. Calvi
Yi Zhang
Archibald S. Perkins
Source :
Nature Communications, Vol 9, Iss 1, Pp 1-12 (2018)
Publication Year :
2018
Publisher :
Nature Portfolio, 2018.

Abstract

Chr3q26 rearrangements cause overexpression of EVI1 and associate with myeloid neoplasms, but the mechanism behind this association is unclear. Here, using a novel mouse model they show that EVI1 causes premalignant myeloid expansion with suppression of other lineages through upregulation of Spi1/PU.1.

Subjects

Subjects :
Science

Details

Language :
English
ISSN :
20411723
Volume :
9
Issue :
1
Database :
Directory of Open Access Journals
Journal :
Nature Communications
Publication Type :
Academic Journal
Accession number :
edsdoj.7ab778ef571e43deb286e077ef67c640
Document Type :
article
Full Text :
https://doi.org/10.1038/s41467-018-06208-y