Knock out of S1P3 receptor signaling attenuates inflammation and fibrosis in bleomycin-induced lung injury mice model.

MLA

Ken Murakami, et al. “Knock out of S1P3 Receptor Signaling Attenuates Inflammation and Fibrosis in Bleomycin-Induced Lung Injury Mice Model.” PLoS ONE, vol. 9, no. 9, Jan. 2014, p. e106792. EBSCOhost, https://doi.org/10.1371/journal.pone.0106792.

APA

Ken Murakami, Masataka Kohno, Masatoshi Kadoya, Hidetake Nagahara, Wataru Fujii, Takahiro Seno, Aihiro Yamamoto, Ryo Oda, Hiroyoshi Fujiwara, Toshikazu Kubo, Satoshi Morita, Hiroshi Nakada, Timothy Hla, & Yutaka Kawahito. (2014). Knock out of S1P3 receptor signaling attenuates inflammation and fibrosis in bleomycin-induced lung injury mice model. PLoS ONE, 9(9), e106792. https://doi.org/10.1371/journal.pone.0106792

Chicago

Ken Murakami, Masataka Kohno, Masatoshi Kadoya, Hidetake Nagahara, Wataru Fujii, Takahiro Seno, Aihiro Yamamoto, et al. 2014. “Knock out of S1P3 Receptor Signaling Attenuates Inflammation and Fibrosis in Bleomycin-Induced Lung Injury Mice Model.” PLoS ONE 9 (9): e106792. doi:10.1371/journal.pone.0106792.