Genetic inactivation of prohormone convertase (PC1) causes a reduction in cholecystokinin (CCK) levels in the hippocampus, amygdala, pons and medulla in mouse brain that correlates with the degree of colocalization of PC1 and CCK mRNA in these structures in rat brain

MLA

James E. Marchand, et al. “Genetic Inactivation of Prohormone Convertase (PC1) Causes a Reduction in Cholecystokinin (CCK) Levels in the Hippocampus, Amygdala, Pons and Medulla in Mouse Brain That Correlates with the Degree of Colocalization of PC1 and CCK MRNA in These Structures in Rat Brain.” Journal of Neurochemistry, vol. 89, no. 2, Apr. 2004. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....f44110b5d5cc57f50b42f3a42272844b&authtype=sso&custid=ns315887.

APA

James E. Marchand, Daesety Vishnuvardhan, Brian M. Cain, Kelly Connolly, Margery C. Beinfeld, Xiaorong Zhu, Donald F. Steiner, & Alissa Blum. (2004). Genetic inactivation of prohormone convertase (PC1) causes a reduction in cholecystokinin (CCK) levels in the hippocampus, amygdala, pons and medulla in mouse brain that correlates with the degree of colocalization of PC1 and CCK mRNA in these structures in rat brain. Journal of Neurochemistry, 89(2).

Chicago

James E. Marchand, Daesety Vishnuvardhan, Brian M. Cain, Kelly Connolly, Margery C. Beinfeld, Xiaorong Zhu, Donald F. Steiner, and Alissa Blum. 2004. “Genetic Inactivation of Prohormone Convertase (PC1) Causes a Reduction in Cholecystokinin (CCK) Levels in the Hippocampus, Amygdala, Pons and Medulla in Mouse Brain That Correlates with the Degree of Colocalization of PC1 and CCK MRNA in These Structures in Rat Brain.” Journal of Neurochemistry 89 (2). http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....f44110b5d5cc57f50b42f3a42272844b&authtype=sso&custid=ns315887.