Ca2+ overload- and ROS-associated mitochondrial dysfunction contributes to δ-tocotrienol-mediated paraptosis in melanoma cells

MLA

Nico Mitro, et al. “Ca2+ Overload- and ROS-Associated Mitochondrial Dysfunction Contributes to δ-Tocotrienol-Mediated Paraptosis in Melanoma Cells.” Apoptosis, vol. 26, Apr. 2021, pp. 277–92. EBSCOhost, https://doi.org/10.1007/s10495-021-01668-y.

APA

Nico Mitro, Monica Marzagalli, Michela Raimondi, Patrizia Limonta, Giangiacomo Beretta, Patrizia Procacci, Matteo Audano, Fabrizio Fontana, & Patrizia Sartori. (2021). Ca2+ overload- and ROS-associated mitochondrial dysfunction contributes to δ-tocotrienol-mediated paraptosis in melanoma cells. Apoptosis, 26, 277–292. https://doi.org/10.1007/s10495-021-01668-y

Chicago

Nico Mitro, Monica Marzagalli, Michela Raimondi, Patrizia Limonta, Giangiacomo Beretta, Patrizia Procacci, Matteo Audano, Fabrizio Fontana, and Patrizia Sartori. 2021. “Ca2+ Overload- and ROS-Associated Mitochondrial Dysfunction Contributes to δ-Tocotrienol-Mediated Paraptosis in Melanoma Cells.” Apoptosis 26 (April): 277–92. doi:10.1007/s10495-021-01668-y.