Low-level expression of proapoptotic Bcl-2-interacting mediator in leukemic cells in patients with chronic myeloid leukemia: role of BCR/ABL, characterization of underlying signaling pathways, and reexpression by novel pharmacologic compounds

MLA

Matthias Mayerhofer, et al. “Low-Level Expression of Proapoptotic Bcl-2-Interacting Mediator in Leukemic Cells in Patients with Chronic Myeloid Leukemia: Role of BCR/ABL, Characterization of Underlying Signaling Pathways, and Reexpression by Novel Pharmacologic Compounds.” Cancer Research, vol. 65, no. 20, Oct. 2005. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....e0b2e082a5e94620ef81359723fa0e4b&authtype=sso&custid=ns315887.

APA

Matthias Mayerhofer, Winfried F. Pickl, Anja Vales, Karl J. Aichberger, Christian Sillaber, Peter Valent, Harald Esterbauer, Rudin Kondo, Michael W. Deininger, Brian J. Druker, Edgar Selzer, Maria Theresa Krauth, & Sophia Derdak. (2005). Low-level expression of proapoptotic Bcl-2-interacting mediator in leukemic cells in patients with chronic myeloid leukemia: role of BCR/ABL, characterization of underlying signaling pathways, and reexpression by novel pharmacologic compounds. Cancer Research, 65(20).

Chicago

Matthias Mayerhofer, Winfried F. Pickl, Anja Vales, Karl J. Aichberger, Christian Sillaber, Peter Valent, Harald Esterbauer, et al. 2005. “Low-Level Expression of Proapoptotic Bcl-2-Interacting Mediator in Leukemic Cells in Patients with Chronic Myeloid Leukemia: Role of BCR/ABL, Characterization of Underlying Signaling Pathways, and Reexpression by Novel Pharmacologic Compounds.” Cancer Research 65 (20). http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....e0b2e082a5e94620ef81359723fa0e4b&authtype=sso&custid=ns315887.