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Screening a Molecular Fragment Library to Modulate the PED/PEA15-Phospholipase D1 Interaction in Cellular Lysate Environments

Authors :
Carla Isernia
Emilia Pedone
Marica Sassano
Luigi Russo
Annarita Del Gatto
Gianluca D'Abrosca
Sonia Di Gaetano
Maria Della Valle
Laura Zaccaro
Gaetano Malgieri
Biancamaria Farina
Luciano Pirone
Roberto Fattorusso
Farina, B.
Pirone, L.
D'Abrosca, G.
Della Valle, M.
Russo, L.
Isernia, C.
Sassano, M.
Del Gatto, A.
Di Gaetano, S.
Zaccaro, L.
Malgieri, G.
Pedone, E. M.
Fattorusso, R.
Source :
ACS Chemical Biology. 16:2798-2807
Publication Year :
2021
Publisher :
American Chemical Society (ACS), 2021.

Abstract

The overexpression of PED/PEA15, the phosphoprotein enriched in diabetes/phosphoprotein enriched in the astrocytes 15 protein (here referred simply to as PED), observed in some forms of type II diabetes, reduces the transport of insulin-stimulated glucose by binding to the phospholipase D1 (PLD1). The inhibition of the PED/PLD1 interaction was shown to restore basal glucose transport, indicating PED as a pharmacological target for the development of drugs capable of improving insulin sensitivity and glucose tolerance. We here report the identification and selection of PED ligands by means of NMR screening of a library of small organic molecules, NMR characterization of the PED/PLD1 interaction in lysates of cells expressing PLD1, and modulation of such interactions using BPH03, the best selected ligand. Overall, we complement the available literature data by providing detailed information on the structural determinants of the PED/PLD1 interaction in a cellular lysate environment and indicate BPH03 as a precious scaffold for the development of novel compounds that are able to modulate such interactions with possible therapeutic applications in type II diabetes.

Details

ISSN :
15548937 and 15548929
Volume :
16
Database :
OpenAIRE
Journal :
ACS Chemical Biology
Accession number :
edsair.doi.dedup.....b12c2044643931530c901d9ae58321f3
Full Text :
https://doi.org/10.1021/acschembio.1c00688