Serum hepcidin following autologous hematopoietic cell transplantation: an illustration of the interplay of iron status, erythropoiesis and inflammation

Hepcidin, the key hormone in iron homeostasis,[1][1] is regulated by erythropoietic activity and hypoxia (negative feedback),[2][2] as well as iron stores (hepcidin induction by iron) and inflammation (upregulation).[1][1],[3][3] Only a few publications have focused on hepcidin in the context of