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Stabilization of atherosclerotic plaques: an update

Authors :: Yla-Herttuala, S.
Bentzon, J.F.
Daemen, M.
Falk, E.
Garcia-Garcia, H.M.
Herrmann, J.
Hoefer, I.
Jauhiainen, S.
Jukema, J.W.
Krams, R.
Kwak, B.R.
Marx, N.
Naruszewicz, M.
Newby, A.
Pasterkamp, G.
Serruys, P.W.J.C.
Waltenberger, J.
Weber, C.
Tokgozoglu, L.
ESC Working Grp Atherosclerosis Va
ACS - Amsterdam Cardiovascular Sciences
Pathology
Cardiology
Kardiyoloji
Source :: European Heart Journal, 34(42), 3251-3258, EUROPEAN HEART JOURNAL, European heart journal, 34(42), 3251-3258. Oxford University Press, Ylä-Herttuala, S, Bentzon, J F, Daemen, M, Falk, E, Garcia-Garcia, H M, Herrmann, J, Hoefer, I, Jauhiainen, S, Jukema, J W, Krams, R, Kwak, B R, Marx, N, Naruszewicz, M, Newby, A, Pasterkamp, G, Serruys, P W J C, Waltenberger, J, Weber, C, Tokgözoglu, L & ESC Working Group of Atherosclerosis and Vascular Biology 2013, ' Stabilization of atherosclerotic plaques : an update ', European Heart Journal . https://doi.org/10.1093/eurheartj/eht301, European Heart Journal, 34(42), 3251-3258. Oxford University Press, Europe PubMed Central, European heart journal
Publication Year :: 2013
Abstract: The majority of coronary thrombi (∼75%) is caused by plaque rupture .1,2 Prototype of the rupture-prone plaque contains a large, soft, lipid-rich necrotic core with a thin and inflamed fibrous cap, so-called thin-cap fibroatheroma (TCFA) ( Figure 1 ).3,4 Other common features include expansive remodelling, large plaque size, plaque haemorrhage, neovascularization, adventitial inflammation, and ‘spotty’ calcifications.4 Thin-cap fibroatheroma caps are usually