Nitric oxide (NO) at the crossroad of mitochondrial dysfunction and nitrosative damage in traumatic brain injury (TBI)

Authors :: Ücal, M
Kraitsy, K
Weidinger, A
Paier-Pourani, J
Tezer, I
Patz, S
Kraitsy, U
Fink, B
Kozlov, A
Schäfer, U
Source :: 65. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC); 20140511-20140514; Dresden; DOCDI.17.09 /20140513/
Publication Year :: 2014
Publisher :: German Medical Science GMS Publishing House, 2014.
Abstract: Objective: NO has been related to both protective and detrimental processes in CNS. Our aim is to elucidate the role of NO in TBI pathophysiology, to reveal protective and detrimental levels. Method: TBI was induced by FPI on Sprague Dawley rats (mTBI<br />65. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC)

Subjects :: ddc: 610
nitric oxide
traumatic brain injury
mitochondrial dysfunction
610 Medical sciences
Medicine
humanities
nervous system diseases

Language :: English
Database :: OpenAIRE
Journal :: 65. Jahrestagung der Deutschen Gesellschaft für Neurochirurgie (DGNC); 20140511-20140514; Dresden; DOCDI.17.09 /20140513/
Accession number :: edsair.doi.dedup.....6fccc658d2806adc823148a71c06fcc9
Full Text :: https://doi.org/10.3205/14dgnc238

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