Cite
Genetic impairment of interleukin-1 signaling attenuates neuropathic pain, autotomy, and spontaneous ectopic neuronal activity, following nerve injury in mice
MLA
Michael Tal, et al. “Genetic Impairment of Interleukin-1 Signaling Attenuates Neuropathic Pain, Autotomy, and Spontaneous Ectopic Neuronal Activity, Following Nerve Injury in Mice.” PainReferences, vol. 120, no. 3, July 2005. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....462a6c4edeea98dc4b48b1bce168027c&authtype=sso&custid=ns315887.
APA
Michael Tal, Yehuda Shavit, Eran Gabay, Raz Yirmiya, & Gilly Wolf. (2005). Genetic impairment of interleukin-1 signaling attenuates neuropathic pain, autotomy, and spontaneous ectopic neuronal activity, following nerve injury in mice. PainReferences, 120(3).
Chicago
Michael Tal, Yehuda Shavit, Eran Gabay, Raz Yirmiya, and Gilly Wolf. 2005. “Genetic Impairment of Interleukin-1 Signaling Attenuates Neuropathic Pain, Autotomy, and Spontaneous Ectopic Neuronal Activity, Following Nerve Injury in Mice.” PainReferences 120 (3). http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....462a6c4edeea98dc4b48b1bce168027c&authtype=sso&custid=ns315887.