Additional file 1: Table S1. of Whole transcriptome analysis of human erythropoietic cells during ontogenesis suggests a role of VEGFA gene as modulator of fetal hemoglobin and pharmacogenomic biomarker of treatment response to hydroxyurea in β-type hemoglobinopathy patients

Upregulated genes when (i) peripheral blood is compared to umbilical cord blood and fetal liver (PB vs CB+FL) and (ii) peripheral blood is compared to fetal liver (PB vs FL). Table S2. Downregulated genes when (i) peripheral blood is compared to umbilical cord blood and fetal liver (PB vs CB+FL) and (ii) peripheral blood is compared to fetal liver (PB vs FL). Table S3. Statistical and Hardy-Weinberg analyses on the genotyping data of β-type hemoglobinopathy patients of Hellenic origin and healthy (non-thalassemic) individuals. Table S4. Pairwise linkage disequilibrium (LD) calculations for the tagSNPs of interest across the VEGFA gene (CEU). Figure S1. Differential gene expression when adult peripheral blood is compared to cord blood. A total of 135 genes were over- (FC > 2) or downregulated (FC 2) or downregulated (FC