Sup. Figure 5: Entrectinib suppressed phosphorylation of TRK-A with G667C mutation slightly and inhibited phosphorylation of ERK transiently. from Foretinib Overcomes Entrectinib Resistance Associated with the NTRK1 G667C Mutation in NTRK1 Fusion–Positive Tumor Cells in a Brain Metastasis Model

Tumor cells were treated with 10nM of drugs for indicated time, and harvested lysates were assessed by western blotting.