Mendelian randomization supports bidirectional causality between telomere length and clonal hematopoiesis of indeterminate potential

MLA

Tetsushi, Nakao, et al. “Mendelian Randomization Supports Bidirectional Causality between Telomere Length and Clonal Hematopoiesis of Indeterminate Potential.” Science Advances, vol. 8, Apr. 2022. EBSCOhost, https://doi.org/10.1126/sciadv.abl6579.

APA

Tetsushi, N., Alexander G, B., Margaret A, T., Seyedeh M, Z., Md M, U., Abhishek, N., Cara L, C., John, L., Michael C, H., Joshua S, W., Akhil, P., Christopher J, G., Gabriel K, G., Shoa L, C., Romit, B., Themistocles L, A., Leslie S, E., Adrienne M, S., Quenna, W., … Pradeep, N. (2022). Mendelian randomization supports bidirectional causality between telomere length and clonal hematopoiesis of indeterminate potential. Science Advances, 8. https://doi.org/10.1126/sciadv.abl6579

Chicago

Tetsushi, Nakao, Bick Alexander G, Taub Margaret A, Zekavat Seyedeh M, Uddin Md M, Niroula Abhishek, Carty Cara L, et al. 2022. “Mendelian Randomization Supports Bidirectional Causality between Telomere Length and Clonal Hematopoiesis of Indeterminate Potential.” Science Advances 8 (April). doi:10.1126/sciadv.abl6579.