Increase in membrane surface expression and phosphorylation of <scp>TRPC3</scp> related to olfactory dysfunction in α‐synuclein transgenic mice

Olfactory impairment is an initial non-motor symptom of Parkinson&#39;s disease that causes the deposition of aggregated α-synuclein (α-syn) in olfactory neurons. Transient receptor potential canonical (TRPC) channels are a diverse group of non-selective Ca