Cite
Acquired resistance to EGFR-TKIs in NSCLC mediates epigenetic downregulation of MUC17 by facilitating NF-κB activity via UHRF1/DNMT1 complex
MLA
Shuye Lin, et al. “Acquired Resistance to EGFR-TKIs in NSCLC Mediates Epigenetic Downregulation of MUC17 by Facilitating NF-ΚB Activity via UHRF1/DNMT1 Complex.” International Journal of Biological Sciences, vol. 19, Jan. 2023, pp. 832–51. EBSCOhost, https://doi.org/10.7150/ijbs.75963.
APA
Shuye Lin, Hongyun Ruan, Lin Qin, Cong Zhao, Meng Gu, Ziyu Wang, Bin Liu, Haichao Wang, & Jinghui Wang. (2023). Acquired resistance to EGFR-TKIs in NSCLC mediates epigenetic downregulation of MUC17 by facilitating NF-κB activity via UHRF1/DNMT1 complex. International Journal of Biological Sciences, 19, 832–851. https://doi.org/10.7150/ijbs.75963
Chicago
Shuye Lin, Hongyun Ruan, Lin Qin, Cong Zhao, Meng Gu, Ziyu Wang, Bin Liu, Haichao Wang, and Jinghui Wang. 2023. “Acquired Resistance to EGFR-TKIs in NSCLC Mediates Epigenetic Downregulation of MUC17 by Facilitating NF-ΚB Activity via UHRF1/DNMT1 Complex.” International Journal of Biological Sciences 19 (January): 832–51. doi:10.7150/ijbs.75963.