Targeted Atp1a2 knockdown by antisense oligonucleotides leads to reduced SOD1 aggregation and accelerated disease progression in the SOD1*G93A mouse model of ALS

Authors :: Abhirami Kannan Iyer
Kathleen M. Schoch
Anna Oldenborg
Hao Chen
Sarah E Smith
Timothy M Miller
Celeste M. Karch
Azad Bonni
Source :: Alzheimer's & Dementia. 17
Publication Year :: 2021
Publisher :: Wiley, 2021.

Subjects :: Psychiatry and Mental health
Cellular and Molecular Neuroscience
Developmental Neuroscience
Epidemiology
Health Policy
Neurology (clinical)
Geriatrics and Gerontology

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