Disruption of Francisella tularensis Schu S4 iglI , iglJ , and pdpC Genes Results in Attenuation for Growth in Human Macrophages and In Vivo Virulence in Mice and Reveals a Unique Phenotype for pdpC

MLA

Lee-Ann H. Allen, et al. “Disruption of Francisella Tularensis Schu S4 IglI , IglJ , and PdpC Genes Results in Attenuation for Growth in Human Macrophages and In Vivo Virulence in Mice and Reveals a Unique Phenotype for PdpC.” Infection and Immunity, vol. 81, Mar. 2013, pp. 850–61. EBSCOhost, https://doi.org/10.1128/iai.00822-12.

APA

Lee-Ann H. Allen, Bradley D. Jones, Jed A. Rasmussen, Matthew E. Long, & Stephen R. Lindemann. (2013). Disruption of Francisella tularensis Schu S4 iglI , iglJ , and pdpC Genes Results in Attenuation for Growth in Human Macrophages and In Vivo Virulence in Mice and Reveals a Unique Phenotype for pdpC. Infection and Immunity, 81, 850–861. https://doi.org/10.1128/iai.00822-12

Chicago

Lee-Ann H. Allen, Bradley D. Jones, Jed A. Rasmussen, Matthew E. Long, and Stephen R. Lindemann. 2013. “Disruption of Francisella Tularensis Schu S4 IglI , IglJ , and PdpC Genes Results in Attenuation for Growth in Human Macrophages and In Vivo Virulence in Mice and Reveals a Unique Phenotype for PdpC.” Infection and Immunity 81 (March): 850–61. doi:10.1128/iai.00822-12.